id	author	title	date	pages	extension	mime	words	sentences	flesch	summary	cache	txt
work_a4mox65c4reh7itnnabvwq54be	Mario Herrera-Marschitz	Targeting Sentinel Proteins and Extrasynaptic Glutamate Receptors: a Therapeutic Strategy for Preventing the Effects Elicited by Perinatal Asphyxia?	2017	13	.pdf	application/pdf	11347	1167	54	Targeting Sentinel Proteins and Extrasynaptic Glutamate Receptors: a Therapeutic Strategy for Preventing the Effects Elicited by Perinatal Asphyxia? impairment, and inactivation of antioxidant enzymes, increasing further the activity of pro-oxidant enzymes, thereby making the surviving neonate vulnerable to recurrent metabolic Upon delivery and during neonatal and early developmental stages, oxidative stress is a permanent risk for the developing individual, enhanced by a sudden increase or decrease of PARP-1 inhibition attenuates nNOS activation and reduces cell death induced by oxidative conditions asphyxia-exposed rat neonates, making it evident that PA produces a permanent energy deficit (increased ADP/ATP ratio) long-term effects produced by perinatal asphyxia: effects of nicotinamide and theophylline on PARP-1 activity in brain and peripheral Andersson K, Herrera-Marschitz M (1997) Effects of perinatal asphyxia on systemic and intracerebral glycolysis metabolism and pH Morales P, Herrera-Marschitz M, Valdés JL (2016) Effect of perinatal asphyxia on tuberomammillary nucleus neuronal density and	./cache/work_a4mox65c4reh7itnnabvwq54be.pdf	./txt/work_a4mox65c4reh7itnnabvwq54be.txt