id	author	title	date	pages	extension	mime	words	sentences	flesch	summary	cache	txt
work_cmodtvoqizb7xoa3svqnkdblj4	Christopher C. Giza	The New Neurometabolic Cascade of Concussion	2014	8	.pdf	application/pdf	7413	839	51	Data Synthesis: The primary elements of the pathophysiologic cascade following concussive brain injury include abrupt events following experimental concussive brain injury and resulting energy crisis is a likely mechanism for postconcussive vulnerability, making the brain less able to respond adequately to a second injury and potentially leading to longerlasting deficits. Following the initial period of accelerated glucose utilization, the concussed brain goes into a period of depressed metabolism. With increasing numbers of children and young adults participating in organized sports and sustaining head injuries, understanding the effects of TBI on the immature brain becomes extracellular potassium and the indiscriminate release of glutamate following concussive brain injury. blood flow following concussive brain injury. prolonged depression of cerebral oxidative metabolism following concussive brain injury in the rat: a cytochrome oxidase histochemistry study. Dissociation of cerebral glucose metabolism and level of consciousness during the period of metabolic depression following human traumatic brain injury. magnesium concentration following traumatic brain injury in rats.	./cache/work_cmodtvoqizb7xoa3svqnkdblj4.pdf	./txt/work_cmodtvoqizb7xoa3svqnkdblj4.txt