id	author	title	date	pages	extension	mime	words	sentences	flesch	summary	cache	txt
cord-337972-otfloo64	Kao, Shang Jyh	Mechanism of Fulminant Pulmonary Edema Caused by Enterovirus 71	2004-06-15		.txt	text/plain	1949	135	53	Destruction of the medial, ventral, and caudal medulla may lead to sympathetic overactivation, causing blood to shift to the lungs. Destruction of the medial, ventral, and caudal medulla may lead to sympathetic overactivation, causing blood to shift to the lungs. Central sympathetic activation resulting in systemic vasoconstriction and the shift of blood volume to the pulmonary region of the circulatory system, as demonstrated in earlier findings from our laboratory, was used to explain centrogenic PE [2, 3] . In the present study, we report CNS lesions in patients associated with PE due to enterovirus infection. The present study also supports the contention that hyperglycemia, leukocytosis, and elevated protein levels in CSF are risk factors for the development of ARDS due to enterovirus infection [1, 2] . In this connection, we found that endogenous and exogenous nitric oxide reduced pulmonary hypertension but increased the capillary filtration coefficient and the extent of injury in an isolated rat's lung that was subjected to ischemia and reperfusion [16] .	./cache/cord-337972-otfloo64.txt	./txt/cord-337972-otfloo64.txt