id	author	title	date	pages	extension	mime	words	sentences	flesch	summary	cache	txt
cord-267268-50h13gso	Spagnolo, Paolo	Pulmonary fibrosis secondary to COVID-19: a call to arms?	2020-05-15		.txt	text/plain	984	64	51	3 The mechanisms through which severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) causes lung damage are only partly known, but plausible contributors include a cytokine release syndrome triggered by the viral antigen, drug-induced pulmonary toxicity, and high airway pressure and hyperoxiainduced acute lung injury secondary to mechanical ventilation. To date, about 1·2 million people worldwide have recovered from COVID-19, but there remains concern that some organs, including the lungs, might have long-term impairment following infection (figure). In a study of 36 patients who had recovered from MERS, chest x-rays taken a median of 43 (range 32-320) days after hospital discharge showed abnormalities described as lung fibrosis in about a third of the patients. Long-term bone and lung consequences associated with hospital-acquired severe acute respiratory syndrome: a 15-year follow-up from a prospective cohort study Risk factors associated with acute respiratory distress syndrome and death in patients with coronavirus disease 2019 pneumonia in Wuhan, China	./cache/cord-267268-50h13gso.txt	./txt/cord-267268-50h13gso.txt