id	author	title	date	pages	extension	mime	words	sentences	flesch	summary	cache	txt
cord-299277-ana68vpf	Horn, Sarah R.	Identifying causal role of COVID-19 in immunopsychiatry models	2020-05-29		.txt	text/plain	1339	81	44	There are several immunopsychiatry examples in which COVID-19 may be appropriately considered a confounder, including research on inflammation and mortality risk. Like before, we simulated effect sizes leading to and away from COVID-19 as either strong or weak, and set the direct relationship between the predictor and outcome of interest (SES and respiratory distress, respectively) to a moderate effect size. As shown in Figure 2 , models that omit the virus best estimate the total effect of SES on distress (i.e., the total effect). Unless researchers are interested in the effect of all pathways from SES to respiratory distress except COVID-19, they should not include the virus as a covariate in their models, or their estimates will not capture the full pathway (see Figure 3 ). Data were simulated from models in which COVID-19 mediates the causal relationship between socioeconomic status (SES) and respiratory distress.	./cache/cord-299277-ana68vpf.txt	./txt/cord-299277-ana68vpf.txt