id	author	title	date	pages	extension	mime	words	sentences	flesch	summary	cache	txt
cord-303384-bgvagdft	Bilinska, Katarzyna	Anosmia in COVID-19: A Bumpy Road to Establishing a Cellular Mechanism	2020-07-16		.txt	text/plain	2173	124	49	Several very recent papers contributed to explaining the key cellular steps occurring in the olfactory epithelium leading to anosmia/hyposmia (collectively known as dysosmia) initiated by SARS-CoV-2 infection. Initial hospital observations and early studies have suggested several possible mechanisms for the development of anosmia in COVID-19, including olfactory cleft syndrome, nasal obstruction and rhinorrhea, cytokine storm, direct damage to olfactory receptor neurons (ORNs), and impairment of the olfactory perception centers in the brain. The current model of olfactory dysfunction in COVID-19 is based on the already proven observation that SUS cells are the primary target of the virus and that SUSs infection initiates a series of events leading to dysosmia. Non-neural expression of SARS-CoV-2 entry genes in the olfactory epithelium suggests mechanisms underlying anosmia in COVID-19 patients.	./cache/cord-303384-bgvagdft.txt	./txt/cord-303384-bgvagdft.txt