id	author	title	date	pages	extension	mime	words	sentences	flesch	summary	cache	txt
cord-329384-uvgwth5n	Milligan, Gregory P.	Recognizing Right Ventricular Dysfunction in COVID-19 Related Respiratory Illness	2020-05-11		.txt	text/plain	537	34	41	COVID-19, coronavirus, Right ventricular failure, Heart failure, Acute cor pulmonale, Shock, ARDS The mechanism of ACP in ARDS is established as refractory hypoxemia, pulmonary edema, and microvascular thrombosis acutely increase RV afterload (2) . Alveolar ventilation must be optimized to limit hypoxic vasoconstriction while considering the hemodynamic consequences of high positive end-expiratory pressure (PEEP) on RV preload and afterload. A pulmonary arterial catheter may limit comorbid cardiogenic edema while also aiding in optimization of RV preload, cardiac output, and pulmonary vascular resistance for which inotropic support and inhaled pulmonary vasodilators should be considered. This requires optimization and support for perhaps the most impacted side of the heart in critical COVID-19 infections, the right ventricle. 1) The presented work has not been previously published and is not currently under consideration for publication at any other journal. Elevated troponin in patients with Coronavirus Disease 2019 (COVID-19): possible mechanisms Pulmonary and Cardiac Pathology in COVID-19	./cache/cord-329384-uvgwth5n.txt	./txt/cord-329384-uvgwth5n.txt