id	author	title	date	pages	extension	mime	words	sentences	flesch	summary	cache	txt
cord-356112-c32icxir	Weiss, R. C.	Increased plasma levels of leukotriene B4 and prostaglandin E2 in cats experimentally inoculated with feline infectious peritonitis virus	1988		.txt	text/plain	4022	220	47	Specific-pathogen-free kittens experimentally infected with feline infectious peritonitis virus (FIPV) subsequently demonstrated increased plasma levels of the arachidonic acid metabolites, leukotriene (LT) B4 and prostaglandin (PG) E2. Loss of fluid and other plasma constituents from inflamed blood vessels, immunologically damaged by complexes of virus, antiviral antibodies and complement (C) proteins (August, 1984; Barlough &Weiss, 1983; Pedersen & Boyle, 1980; Weiss & Scott, 1981~; Jacobse-Geels et al., 1982) or by cytotoxic lymphokines or other enzymatically-active factors released during cellular immune responses (Pedersen, 1985) , may occur in cases of acute FIP. Although vascular lesions, and possibly the pathologic effusions, in FIP are believed to occur via immunological (antibody-mediated) mechanisms (Pedersen, 1985; August, 1984; Weiss & Scott, 1981b; 1981c) , the occurrence of fever and increased plasma levels of LTB4 and PGE2 prior to detectable antibody responses in some FIPV-infected kittens suggest that nonimmunological factors can be involved in some disease manifestations.	./cache/cord-356112-c32icxir.txt	./txt/cord-356112-c32icxir.txt