id	author	title	date	pages	extension	mime	words	sentences	flesch	summary	cache	txt
cord-007637-o2cijp5a	Knobler, R.L.	Host genetic regulation of acute MHV-4 viral encephalomyelitis and acute experimental autoimmune encephalomyelitis in (BALB/cKe × SJL/J) recombinant-inbred mice()	2005-06-03		.txt	text/plain	4138	195	49	In the present report we provide the strain distribution patterns of susceptibility to acute mouse hepatitis virus type-4 (MHV-4) encephalomyelitis, acute experimental allergic encephalomyelitis (EAE) and vasoactive amine sensitivity (VAAS) for 9 (CXJ) recombinant-inbred strains between BALB/cKe (C) and SJL/J (J) mice. Susceptibility to acute experimental autoimmune encephalomyelitis (EAE), an acute perivascular inflammatory demyelinating disease, is controlled by at least two genes, one, H-2 linked [responder haplotype (H-2 S, H-2 q or H-2d)]; the other associated with natural vasoactive amine sensitivity (VAAS) or that induced by Bordetella pertussis (Linthicum and Frelinger 1982) . Nine RI strains between BALB/cKe and SJL/J (CXJ) have been tested to provide the SDP of susceptibility to acute MHV-4 encephalomyelitis, acute EAE and VAAS. Finally, although some (CXJ) RI strains are susceptible to acute EAE, induced VAAS and acute M H V -4 encephalomyelitis EAE-like histopathological disease does not occur in these animals following MHV-4 intracerebral infection.	./cache/cord-007637-o2cijp5a.txt	./txt/cord-007637-o2cijp5a.txt