id	author	title	date	pages	extension	mime	words	sentences	flesch	summary	cache	txt
cord-266450-g9vihgbk	Tran, Michael	SARS-CoV-2 and pulmonary embolism: who stole the platelets?	2020-09-03		.txt	text/plain	1498	93	38	Careful attention to his daily platelet count suggested the possibility of immune mediated heparin-induced thrombocytopenia (HIT) which was confirmed by laboratory testing and resolved when anticoagulation was switched to a direct thrombin inhibitor. CONCLUSIONS: Since excessive platelet activation and in situ thrombosis occur in HIT, this case underscores the need to consider that thrombocytopenia in patients with SARS-CoV-2—most of whom receive heparinoids—may be unrecognized HIT. Emerging reports suggest the possibility of HIT developing in SARS-CoV-2 patients receiving heparin anticoagulation [4, 5] . The patient's platelet count decreased from 487 k/uL to a nadir of 91 k/uL over the following 4 days, raising the concern for heparin induced thrombocytopenia (HIT) with an intermediate pretest probability by the 4Ts score of 4 ( Table 1 ). Platelet count and time points for anticoagulation administration and laboratory testing COVID-19 patients receiving heparin-involved treatment.	./cache/cord-266450-g9vihgbk.txt	./txt/cord-266450-g9vihgbk.txt