id	author	title	date	pages	extension	mime	words	sentences	flesch	summary	cache	txt
cord-251986-ajlpb9li	Li, Yan‐Chao	The neuroinvasive potential of SARS‐CoV2 may play a role in the respiratory failure of COVID‐19 patients	2020-03-11		.txt	text/plain	2250	125	47	This virus shares highly homological sequence with SARS‐CoV, and causes acute, highly lethal pneumonia coronavirus disease 2019 (COVID‐19) with clinical symptoms similar to those reported for SARS‐CoV and MERS‐CoV. A growing body of evidence shows that neurotropism is one common feature of CoVs. 1, [9] [10] [11] [12] Therefore, it is urgent to make clear whether SARS-CoV-2 can gain access to the central nervous system (CNS) and induce neuronal injury leading to the acute respiratory distress. Mechanisms of host defense following severe acute respiratory syndrome-coronavirus (SARS-CoV) pulmonary infection of mice Exploring the pathogenesis of severe acute respiratory syndrome (SARS): the tissue distribution of the coronavirus (SARS-CoV) and its putative receptor, angiotensin-converting enzyme 2 (ACE2) Organ distribution of severe acute respiratory syndrome (SARS) associated coronavirus (SARS-CoV) in SARS patients: implications for pathogenesis and virus transmission pathways Severe acute respiratory syndrome coronavirus infection causes neuronal death in the absence of encephalitis in mice transgenic for human ACE2 The neuroinvasive potential of SARS-CoV2 may play a role in the respiratory failure of COVID-19 patients	./cache/cord-251986-ajlpb9li.txt	./txt/cord-251986-ajlpb9li.txt