id	author	title	date	pages	extension	mime	words	sentences	flesch	summary	cache	txt
cord-013280-kczj24se	Yang, Bo	Molecular Mechanisms of Immune Escape for Foot-and-Mouth Disease Virus	2020-09-04		.txt	text/plain	11293	608	46	Viral capsid protein VP1 and leading protein L pro can inhibit the production of interferon (IFN) and innate immune response by interacting with soluble resistance-related calcium-binding protein (sorcin) or host transcription factor ADNP [12, 13] . Viral capsid protein VP1 and leading protein L pro can inhibit the production of interferon (IFN) and innate immune response by interacting with soluble resistance-related calcium-binding protein (sorcin) or host transcription factor ADNP [12, 13] . FMDV VP1 interacts with host ribosomal protein SA (RPSA) to continually activate the MAPK signal pathway and promote virus replication by inhibiting the RPSA-mediated function [59] (Figure 2 , Table 1 ). It interacts with the VISA protein to inhibit the formation of VISA-regulated complex, thereby inhibiting the dimerization and phosphorylation of IRF3, inhibiting the expression of antiviral genes induced by IFN-β, and promoting FMDV replication [60] (Figure 2 , Table 1 ).	./cache/cord-013280-kczj24se.txt	./txt/cord-013280-kczj24se.txt