id	author	title	date	pages	extension	mime	words	sentences	flesch	summary	cache	txt
cord-295191-xu26mvc3	Avirutnan, Panisadee	Complement and its role in protection and pathogenesis of flavivirus infections	2008-12-30		.txt	text/plain	6120	333	32	Complement evasion mechanisms include: (a) use of complement receptors to enhance viral entry or suppress adaptive immune response (e.g., HIV, West Nile virus (WNV), measles virus, adenoviruses, herpesviruses, enteroviruses, hepatitis B and C viruses ); (b) expression of viral proteins that directly inhibit complement (e.g., herpesviruses, coronaviruses, and astroviruses [127] [128] [129] [130] [131] [132] [133] [134] [135] [136] ); (c) modulation of expression of complement regulators on host cells to prevent complement-dependent lysis (e.g., herpesviruses [137] [138] [139] ); (d) incorporation of human regulators on the surface of virions to protect from complement-mediated virolysis (e.g. HIV, HTLV, cytomegalovirus, and vaccinia virus [140] [141] [142] [143] [144] [145] [146] ); (e) recruitment of soluble complement regulatory proteins to the virion or infected cell surface (e.g., WNV and HIV [147] [148] [149] [150] [151] ); (f) expression of viral decoy proteins that structurally or functionally mimic complement regulatory proteins (e.g., poxviruses and herpesviruses [152] [153] [154] [155] [156] [157] [158] [159] .	./cache/cord-295191-xu26mvc3.txt	./txt/cord-295191-xu26mvc3.txt