id	author	title	date	pages	extension	mime	words	sentences	flesch	summary	cache	txt
work_vcz5sf2ko5bfpcfabvre4zg4mu	Gyorgy Csordas	ER-Mitochondrial Ultrastructure in the Liver of Normal and Ethnaol-Fed Rats	2013	1	.pdf	application/pdf	1402	111	40	Calcium-Induced ROS Generation during Ischemia Triggers mPTPDependent Cell Death during Reperfusion Impairment of mitochondrial function is a central event of ischemiareperfusion (I/R) injury leading to tissue damage and cell death. 1, ROS generation with Mito-Sox Red, mPTP opening as mitochondrial calcein ROS with superoxide dismutase mimetic MnTBAP or antioxidant Trolox prevented reperfusion-induced cell death. We conclude that mPTP-dependent cell death during reperfusion is mediated by Ca2þdependent ROS generation during ischemia. These probes, targeted to the ER lumen, ER membrane (cytosolic face), cytosol, mitochondrial matrix and outer membrane (OMM) and the IP3-receptor, of the ER membrane, bulk cytosol and mitochondrial matrix and OMM. It was found that the mitochondrial surface in close association with ER was significantly reduced in the chronic ethanol-fed condition as Calcium-Induced ROS Generation during Ischemia Triggers mPTP-Dependent Cell Death during Reperfusion Dynamic Measurement of Ca2+-Induced Changes in Organelle-Specific Redox Microdomains Mitochondrial Nm23-H4/NDPK-D is Multifunctional: Intermembrane Cardiolipin Transfer Linked to Apoptosis	./cache/work_vcz5sf2ko5bfpcfabvre4zg4mu.pdf	./txt/work_vcz5sf2ko5bfpcfabvre4zg4mu.txt