trigram

This is a table of type trigram and their frequencies. Use it to search & browse the list to learn more about your study carrel.

trigram	frequency
in patients with	466
angiotensin converting enzyme	223
as well as	172
the use of	168
of ang ii	167
acute respiratory syndrome	166
severe acute respiratory	164
the presence of	152
the treatment of	145
patients with covid	139
respiratory syndrome coronavirus	131
of the renin	127
angiotensin ii receptor	113
the role of	112
converting enzyme inhibitors	112
of ace inhibitors	107
of the ace	107
the risk of	101
been shown to	100
patients with hae	98
the effects of	96
angiotensin ii type	95
of angiotensin ii	95
ace inhibitors and	95
of patients with	94
the development of	87
angiotensin receptor blockers	85
is associated with	83
of the ras	81
in the treatment	77
the at receptor	76
ace and ace	75
as compared to	75
of angiotensin converting	73
of ace in	71
activation of the	71
ii type receptor	70
the effect of	70
in addition to	68
it has been	68
for the treatment	67
of the angiotensin	67
patients with hypertension	65
one of the	64
in the brain	64
acute respiratory distress	63
an increase in	62
expression of ace	62
respiratory distress syndrome	61
inhibitors and angiotensin	61
converting enzyme in	61
and angiotensin ii	60
has been shown	60
converting enzyme inhibition	59
in the presence	59
the absence of	58
role in the	57
due to the	57
of blood pressure	56
of ace and	55
risk of covid	55
there is no	55
ii receptor blockers	53
of the sars	52
the active site	50
a role in	50
ace activity in	50
inhibitors and arbs	49
ang ii levels	49
of the c	49
increased risk of	48
ace in the	48
clinical characteristics of	48
role of ace	48
use of ace	48
shown to be	48
of severe acute	48
role of the	48
involved in the	47
enzyme inhibitors and	47
of the n	47
of angiotensin i	46
ang ii and	46
was associated with	45
associated with a	45
of the virus	45
based on the	44
in response to	44
a number of	44
in the kidney	44
vascular smooth muscle	44
the expression of	44
in the heart	43
responsible for the	43
ang ii to	43
patients with severe	43
and angiotensin receptor	43
the united states	43
effects of ace	43
middle east respiratory	42
the novel coronavirus	42
in the absence	42
ace inhibitor therapy	42
effects of the	42
the number of	41
east respiratory syndrome	41
in order to	41
the formation of	41
of the disease	41
with ace inhibitors	41
after myocardial infarction	40
the angiotensin ii	40
renin angiotensin system	40
ace inhibitors or	40
structure of the	40
analysis of the	40
effect of angiotensin	39
the release of	39
of novel coronavirus	39
the regulation of	39
a variety of	39
of ace inhibition	39
the pathogenesis of	39
in db mice	38
aceis and arbs	38
the spike protein	38
converting enzyme is	38
found to be	38
well as the	38
in blood pressure	38
in heart failure	38
smooth muscle cells	38
converting enzyme and	37
the discovery of	37
activity of the	37
blood pressure and	36
c nh gene	36
as a result	36
associated with increased	36
the activity of	36
angiotensin system in	35
part of the	35
of ang i	35
on the other	35
functional receptor for	35
angiotensin i to	34
congestive heart failure	34
components of the	34
is involved in	34
the conversion of	34
for treatment of	34
risk factors for	34
the b receptor	34
world health organization	34
has been reported	34
central nervous system	34
levels of ace	34
the basis of	34
acute lung injury	33
converting enzyme gene	33
associated with the	33
the efficacy of	33
the importance of	33
a result of	33
treatment of covid	33
the management of	33
i to angiotensin	32
calcium channel blockers	32
to ang ii	32
expression in the	32
role of angiotensin	32
spontaneously hypertensive rats	32
of coronavirus disease	32
in the rat	32
on the basis	32
compared to cont	32
the level of	32
with heart failure	31
a systematic review	31
in animal models	31
heart failure and	31
the course of	31
has not been	31
patients infected with	31
the fact that	31
ii to ang	31
some of the	31
be associated with	31
in the ace	30
of ace inhibitor	30
contribute to the	30
risk factor for	30
and ang ii	30
i converting enzyme	30
ang i and	30
the activation of	30
the other hand	30
showed that the	30
in the united	30
of the at	30
which was not	30
the copyright holder	30
levels of ang	29
with coronavirus disease	29
of the two	29
in the s	29
differences in the	29
has also been	29
are associated with	29
to be the	29
effect of ace	29
in contrast to	29
also known as	29
granted medrxiv a	28
because of the	28
a license to	28
european society of	28
of the enzyme	28
preprint in perpetuity	28
inhibition of ace	28
to display the	28
display the preprint	28
critically ill patients	28
holder for this	28
induced lung injury	28
there is a	28
has granted medrxiv	28
converting enzyme inhibitor	28
and characterization of	28
in the lungs	28
who has granted	28
treatment of hypertension	28
coronary artery disease	28
the production of	28
for this preprint	28
medrxiv a license	28
most of the	28
in the c	28
receptor for the	28
data not shown	28
have been shown	28
was found to	28
license to display	28
copyright holder for	28
ace is a	28
the preprint in	28
the action of	27
the renin angiotensin	27
the structure of	27
the severity of	27
between the two	27
understanding of the	27
receptor blockers on	27
the at r	27
an important role	27
such as the	27
to that of	27
and severity of	27
significantly increased in	27
an ace inhibitor	27
compared to the	27
in the regulation	27
patients with coronavirus	27
of heart failure	27
which in turn	26
to angiotensin ii	26
patients with heart	26
found in the	26
during the covid	26
patients with diabetes	26
chronic kidney disease	26
for patients with	26
according to the	26
conversion of ang	26
inhibitors or arbs	26
is the most	26
the cell surface	26
with novel coronavirus	26
the most common	26
blood pressure in	26
in the human	26
related to the	26
ace protein expression	26
hereditary angioneurotic edema	26
effect on the	26
acts as a	25
to have a	25
addition to the	25
the amount of	25
in this study	25
of the human	25
cells of the	25
and risk of	25
of these drugs	25
present in the	25
the inhibition of	25
appears to be	25
is responsible for	25
of human angiotensin	25
conflict of interest	25
in hypertensive patients	25
ang ii in	25
in this regard	25
and in the	25
of the first	25
the sars coronavirus	25
activity in the	25
the ang ii	25
the ace gene	25
and at r	25
the central nervous	25
left ventricular dysfunction	25
been reported in	24
have shown that	24
severity of covid	24
inhibition and angiotensin	24
the entry of	24
ace ace activity	24
should not be	24
be involved in	24
ace activity was	24
in the case	24
of hypertension and	24
as shown in	24
was shown to	24
of patients infected	24
chronic heart failure	24
in the n	24
converts angiotensin i	24
i and arbs	24
of factor xii	24
the existence of	24
the mas receptor	24
the severe acute	24
entry into the	24
the lack of	24
effects of angiotensin	24
among patients with	24
it is not	24
the case of	24
receptor for sars	24
and heart failure	24
ii in the	24
a role for	23
enzyme in the	23
used for the	23
pneumonia in wuhan	23
in the lung	23
aldosterone system inhibitors	23
may also be	23
with hereditary angioedema	23
was significantly increased	23
is a potent	23
binding to the	23
expression of the	23
patients with chronic	23
the function of	23
formation of ang	23
transforming growth factor	23
blood pressure regulation	23
compared with the	23
db mice at	23
more likely to	23
the possibility of	23
intensive care unit	23
the majority of	23
of myocardial infarction	23
to cont mice	23
the administration of	22
enzyme inhibition and	22
society of cardiology	22
is known to	22
of ace is	22
and treatment of	22
of the covid	22
at and at	22
has been suggested	22
angiotensin i and	22
to ace inhibitors	22
spike protein of	22
to be a	22
sodium and water	22
it is a	22
have been reported	22
of nitric oxide	22
the ace inhibitor	22
ace inhibitor and	22
use of angiotensin	22
cardiovascular and renal	22
of the raas	22
in spontaneously hypertensive	22
ace expression in	22
hae type iii	22
the generation of	22
a functional receptor	22
infected with sars	22
may contribute to	22
expressed in the	22
a retrospective cohort	22
ace inhibition in	22
inhibition of the	22
i and ii	22
as a potential	22
there was no	21
early and late	21
of the active	21
wide range of	21
hypertension and diabetes	21
the increase in	21
syndrome coronavirus infection	21
a novel coronavirus	21
the c nh	21
with severe covid	21
ace and the	21
the angiotensin converting	21
the onset of	21
morbidity and mortality	21
effects of ang	21
systematic review and	21
crystal structure of	21
retrospective cohort study	21
in patients who	21
contributes to the	21
the levels of	21
acute myocardial infarction	21
angiotensin ii in	21
c esterase inhibitor	21
of c inhibitor	21
of hereditary angioedema	21
and the risk	21
and late stages	21
the rate of	21
a crucial role	21
of the patients	21
in combination with	21
by peer review	21
a decrease in	21
and cardiovascular disease	21
of recombinant human	20
in the development	20
member of the	20
the author funder	20
was observed in	20
this version posted	20
the world health	20
the time of	20
domains of human	20
in vitro and	20
was not certified	20
the progression of	20
angiotensin i converting	20
mechanism of action	20
gene expression and	20
the metabolism of	20
converting enzyme activity	20
for the sars	20
the interaction of	20
not certified by	20
members of the	20
in the pathogenesis	20
certified by peer	20
novel coronavirus in	20
reported to be	20
is a functional	20
this preprint this	20
within the kidney	20
to those of	20
a wide range	20
the european society	20
preprint this version	20
binds to the	20
of ace activity	20
the influence of	20
is important to	20
the binding of	20
important role in	20
hospitalized patients with	20
version posted june	20
have been identified	20
the gastrointestinal tract	20
enzyme is a	20
to assess the	20
the immune system	20
campbell et al	20
converting enzyme angiotensin	20
may result in	20
the ras in	20
is the author	20
been reported to	20
higher risk of	20
i to ang	19
increased levels of	19
protein of sars	19
ace inhibitors arbs	19
a patient with	19
by ace inhibitors	19
gene expression in	19
that ace is	19
of sars coronavirus	19
li et al	19
disease in china	19
distribution of ace	19
associated with an	19
two active sites	19
of amino acids	19
clinical course and	19
at r and	19
have also been	19
vascular endothelial cells	19
play a role	19
the right ventricle	19
active sites of	19
patients with hereditary	19
viral entry into	19
and management of	19
the concentration of	19
characteristics of coronavirus	19
at increased risk	19
levels in the	19
at the time	19
in rats with	19
review of the	19
that ace inhibitors	19
actions of ang	19
ace inhibitors are	19
the results of	19
ang i to	19
coronavirus disease in	19
human angiotensin i	19
benefits of ace	19
due to its	19
of the population	19
the cleavage of	19
inhibitors in patients	19
mortality in patients	19
presence of a	19
the degradation of	19
the impact of	18
review and meta	18
known to be	18
increased in db	18
an angiotensin ii	18
ace and tmprss	18
and ace in	18
may be a	18
for coronavirus disease	18
angiotensin ii and	18
function of the	18
beneficial effects of	18
been associated with	18
of angiotensinconverting enzyme	18
of the protein	18
the prevalence of	18
a novel angiotensin	18
in individuals with	18
enzyme protects from	18
the end of	18
and mortality in	18
have not been	18
ang ii is	18
portion of the	18
of the heart	18
expression of angiotensin	18
depends on ace	18
it is important	18
the incidence of	18
regulation of the	18
the host cell	18
risk of severe	18
ang ii at	18
activity of ace	18
in the covid	18
characterization of a	18
the control of	18
the potentiation of	18
risk for covid	18
caused by the	18
and ace inhibitors	18
the cardiovascular system	18
receptors in the	18
treatment with ace	18
used in the	18
of nonallergic angioedema	18
treated with ace	18
with the use	18
sympathetic nervous system	18
the actions of	18
of the bradykinin	18
it should be	18
no effect on	18
the combination of	18
activity in db	17
on ace and	17
c inhibitor deficiency	17
with hypertension and	17
a risk factor	17
hnrnp f overexpression	17
leading to the	17
in the circulation	17
not associated with	17
evidence for a	17
depending on the	17
converting enzyme protects	17
of the novel	17
domain of sace	17
were treated with	17
c inhibitor gene	17
in a variety	17
acute lung failure	17
the heart and	17
have been described	17
potentiation of the	17
a combination of	17
in a study	17
in association with	17
by ang ii	17
of the left	17
the benefits of	17
and at receptors	17
did not modify	17
angiotensin system and	17
results of the	17
insertion deletion polymorphism	17
college of cardiology	17
risk of sars	17
severe acute lung	17
the cell membrane	17
of cardiovascular disease	17
the general population	17
american college of	17
to the c	17
of the classical	17
that there is	17
be due to	17
from the cell	17
and safety of	17
yet to be	17
the involvement of	17
that ang ii	17
increased risk for	17
patients treated with	17
of a novel	16
is blocked by	16
downregulation of ace	16
the context of	16
blockers on cardiac	16
changes in the	16
mortality in covid	16
and expression of	16
that patients with	16
reactive oxygen species	16
end of the	16
essential regulator of	16
use of arbs	16
is an essential	16
implicated in the	16
of heart function	16
for ang ii	16
and tmprss and	16
and in vivo	16
its role in	16
in which the	16
increase in ace	16
is present in	16
efficacy and safety	16
with left ventricular	16
revealed that the	16
to the ace	16
a total of	16
and blood pressure	16
at receptors in	16
contributed to the	16
the most important	16
insights into the	16
observed in the	16
an essential regulator	16
protein expression was	16
ace activity is	16
of at receptors	16
thought to be	16
the ability of	16
referred to as	16
high blood pressure	16
regulator of heart	16
inhibitor and arb	16
in sars coronavirus	16
ace activity and	16
tmprss and is	16
it may be	16
cells in the	16
than that of	16
is thought to	16
at r c	16
interaction with the	16
a review of	16
by ace inhibition	16
been suggested that	16
from severe acute	16
in ang ii	16
the association of	16
bork et al	16
the setting of	16
blockers and the	16
and functional expression	16
is the only	16
been identified as	16
clinical features of	16
coronavirus spike protein	16
and risk factors	16
similar to that	16
likely to have	16
blocked by a	16
animal models of	16
is replaced by	16
in the context	16
led to the	16
of at receptor	16
inflammation and fibrosis	16
ace activity ratio	16
on cardiac angiotensin	16
a mouse model	15
is unable to	15
therapy in patients	15
in ace activity	15
mice at early	15
with the ace	15
and is blocked	15
consistent with the	15
in angiotensin ii	15
conditions such as	15
cloning and functional	15
was found in	15
ang ii receptor	15
there were no	15
is no evidence	15
the addition of	15
treatment of acute	15
likely to be	15
left ventricular hypertrophy	15
peptides by human	15
proven protease inhibitor	15
increased expression of	15
protects from severe	15
highly expressed in	15
with an increased	15
allowed without permission	15
and diabetes mellitus	15
for disease control	15
a clinically proven	15
crucial role of	15
the two domains	15
results in a	15
clinically proven protease	15
are involved in	15
may be the	15
control and prevention	15
entry depends on	15
of the receptor	15
there was a	15
a reduction in	15
all rights reserved	15
patients who are	15
has been identified	15
in the setting	15
the study of	15
with severe acute	15
there are no	15
well as in	15
is able to	15
of the s	15
nitric oxide synthase	15
with ace inhibitor	15
cov and sars	15
studies have shown	15
a study of	15
by a clinically	15
inhibitors and arb	15
women with hae	15
no reuse allowed	15
similar to those	15
the american college	15
and lung injury	15
was used to	15
patients with type	15
in acute respiratory	15
the s protein	15
cell entry depends	15
the association between	15
angiotensin ii receptors	15
is expressed in	15
reuse allowed without	15
expression as a	15
of human ace	15
two homologous domains	15
myocardial infarction and	15
the c domain	15
in the early	15
as part of	15
a report of	15
ii and ang	15
alveolar epithelial cells	15
function of ace	15
may lead to	15
ang ii by	15
the clinical course	14
and the ace	14
a rat model	14
of the reninangiotensin	14
is one of	14
chronic obstructive pulmonary	14
lower respiratory tract	14
in these patients	14
observed in patients	14
of the brain	14
a and b	14
effects on the	14
tissue distribution of	14
mouse model of	14
the c inhibitor	14
the efficacy and	14
functional expression as	14
respiratory syncytial virus	14
disease control and	14
for sars coronavirus	14
of the ang	14
deletion of the	14
at receptor stimulation	14
presence of ace	14
of the infection	14
by angiotensin ii	14
vitro and in	14
system blockers and	14
from ang ii	14
at receptor blockade	14
is based on	14
a human homolog	14
in hereditary angioedema	14
increase the risk	14
hospitalized with covid	14
type i and	14
and the renin	14
angioedema associated with	14
in the management	14
infected with novel	14
of the b	14
type i hereditary	14
i and bradykinin	14
angiotensin system blockers	14
in the first	14
of left ventricular	14
to play a	14
inhibitors of the	14
patients admitted to	14
the spread of	14
are known to	14
there is evidence	14
associated with severe	14
lung injury and	14
in clinical trials	14
in a patient	14
blockade of the	14
of at least	14
the n domain	14
and it is	14
of the bk	14
converting enzyme from	14
the control group	14
human homolog of	14
in a mouse	14
the relationship between	14
in the literature	14
is essential for	14
of which are	14
clinical course of	14
the left ventricle	14
contrast to the	14
seems to be	14
as a consequence	14
case fatality rate	14
a key role	14
lead to a	14
cleavage of the	14
administration did not	14
ace inhibitors in	14
ill patients with	14
has been demonstrated	14
to patients with	14
patients in the	14
leads to the	14
ace inhibitor or	14
with hypertension hospitalized	14
is mediated by	14
insulin administration did	14
is a key	14
cardiovascular disease and	14
were associated with	14
with acute respiratory	14
the present study	14
of severe covid	14
inhibitor of the	14
in the same	14
the authors declare	14
increase in the	14
at receptor antagonist	14
enzyme is an	14
american heart association	13
of the severe	13
system inhibitors in	13
active site of	13
located in the	13
treatment of patients	13
the ras system	13
might be a	13
the mitral valve	13
of these enzymes	13
an increased risk	13
in the course	13
are patients with	13
heart failure with	13
of middle east	13
domains of sace	13
results of a	13
years of age	13
for the management	13
between ace inhibitors	13
inpatients with covid	13
an at receptor	13
angioedema caused by	13
for mortality of	13
patients with novel	13
course and risk	13
i d polymorphism	13
deaths due to	13
amino acid residues	13
wang et al	13
plasma levels of	13
death in patients	13
bradykinin b receptor	13
mellitus at increased	13
protective role of	13
of the right	13
is likely to	13
patients with mild	13
together with the	13
compared to those	13
authors declare that	13
the design of	13
effect of the	13
of adult inpatients	13
in a rat	13
biological peptides by	13
the s subsite	13
the ras and	13
the pathophysiology of	13
et al a	13
it is reasonable	13
receptor blockers with	13
in diabetic mice	13
of the european	13
key role in	13
data from the	13
decrease in ace	13
mediated by the	13
of sars cov	13
regulation of blood	13
the contact system	13
in terms of	13
should be avoided	13
the right coronary	13
from the c	13
shown to have	13
homolog of angiotensin	13
conversion of angiotensin	13
associated with covid	13
is increased in	13
sequence of the	13
as a captopril	13
an inhibitor of	13
with regard to	13
axis of the	13
esterase inhibitor deficiency	13
mortality of adult	13
treatment of sars	13
patients with a	13
adult inpatients with	13
of diabetic nephropathy	13
in critically ill	13
diabetes mellitus at	13
is reduced in	13
factors for mortality	13
the hypothesis that	13
be related to	13
into host cells	13
on blood pressure	13
and arbs in	13
had no effect	13
receptor in the	13
hydrolysis of biological	13
at the end	13
results in the	13
patients with sars	13
features of patients	13
a major role	13
ace ang ii	13
ii receptor blocker	13
inhibitors of angiotensin	13
the surface of	13
of biological peptides	13
the reduction of	13
at early and	13
in the central	13
and outcomes of	13
the in vivo	13
of angioedema attacks	13
i and ang	13
ang ii formation	13
study of the	13
tumor necrosis factor	13
blood glucose levels	13
to be effective	12
the need for	12
this suggests that	12
ace i d	12
but not in	12
is used for	12
bind to the	12
clinical trial of	12
that the use	12
in vivo and	12
higher in the	12
in the active	12
by human angiotensin	12
inhibitors and at	12
kim et al	12
side effects of	12
wei et al	12
half of the	12
of ace protein	12
of the inhibitor	12
of the kidney	12
ace and neprilysin	12
hereditary angioneurotic oedema	12
are shown in	12
donoghue et al	12
be used for	12
ace d d	12
in the control	12
factors associated with	12
ras in the	12
with cardiovascular disease	12
was reported to	12
of the rat	12
in the metabolism	12
to explain the	12
the ratio of	12
infection with sars	12
role for the	12
the left and	12
high level of	12
could be a	12
is possible that	12
domain of ace	12
ace has been	12
and inhibition of	12
is consistent with	12
are related to	12
levels of the	12
that can be	12
plasminogen activator inhibitor	12
in the elderly	12
and ace activity	12
in this section	12
from ang i	12
in the renal	12
enzyme activity in	12
ace is an	12
and renal disease	12
both ace and	12
is identical to	12
characterization of the	12
homologous domains of	12
the human ace	12
ace inhibitor treatment	12
activation of factor	12
polymerase chain reaction	12
the proportion of	12
and smooth muscle	12
the prevention of	12
that they have	12
by using the	12
level of the	12
the synthesis of	12
number of deaths	12
of the study	12
is a major	12
patients with acute	12
evidence for the	12
the coronavirus disease	12
and progression of	12
a first step	12
result in the	12
study of patients	12
the functional receptor	12
contraction of the	12
myocardial infarction in	12
has been found	12
diagnosis and treatment	12
is not yet	12
blood pressure by	12
which is a	12
b and b	12
structure and function	12
mas receptor axis	12
the two homologous	12
patients with cardiovascular	12
is capable of	12
expression was significantly	12
inhibitors angiotensin receptor	12
of the lungs	12
the ace d	12
edema of the	12
inhibitors and risk	12
identical to the	12
of the pandemic	12
are present in	12
ace and ang	12
by the spike	12
of the viral	12
lung injury in	12
the plasma membrane	12
no conflict of	12
ace inhibition is	12
patients with left	12
corradi et al	12
it is also	12
our understanding of	12
it is possible	12
such as ace	12
resulting in a	12
obstructive pulmonary disease	12
is located on	12
at least in	12
to evaluate the	12
proven to be	12
shedding of the	12
is an important	12
the frequency of	12
proximal tubular cells	12
entry of the	12
such as hypertension	12
heart rate and	12
plasma renin activity	12
treatment of hae	12
and properties of	12
to the use	12
of somatic ace	12
shown in figure	12
a member of	12
and neprilysin in	12
in the current	12
all of the	12
studies on the	11
understanding sars pathogenesis	11
have a role	11
characteristics of patients	11
may play a	11
potent inhibitor of	11
angiotensin receptor blocker	11
acute attack therapy	11
change in the	11
that hnrnp f	11
were found to	11
of hnrnp f	11
of the most	11
angiotensin ii at	11
hoffmann et al	11
the brain of	11
ace d allele	11
concentrations of c	11
type ii pneumocytes	11
also been shown	11
of a new	11
for the first	11
have been found	11
the host cells	11
the intensive care	11
generation of ang	11
quality of life	11
randomized clinical trial	11
may have a	11
the ras is	11
located on the	11
of big et	11
db db mice	11
diagnosis of hae	11
clinical manifestations of	11
domain of human	11
by the ace	11
of the potentiating	11
the interventricular septum	11
of neutral endopeptidase	11
identified in the	11
hypertension and heart	11
of the spike	11
of hospitalized patients	11
mechanism by which	11
the aim of	11
enzyme inhibition in	11
of the m	11
the sars virus	11
the x chromosome	11
axes of the	11
increase in blood	11
higher risk for	11
ace knockout mice	11
the venom of	11
detected in the	11
of hae attacks	11
council on hypertension	11
of the cardiac	11
site of human	11
addition to its	11
of this system	11
in cardiovascular disease	11
blood flow and	11
with a higher	11
right coronary artery	11
the drosophila ace	11
the ace axis	11
caused by c	11
is not a	11
potential therapeutic target	11
approved by the	11
less likely to	11
the two active	11
risk of death	11
blood pressure lowering	11
influenza a virus	11
converted to ang	11
system inhibitors and	11
profiling of ace	11
indicates that the	11
s and s	11
of critically ill	11
there is an	11
of this enzyme	11
with an increase	11
report of the	11
shown in fig	11
specificity of the	11
impact on the	11
and vascular smooth	11
is reasonable to	11
lead to the	11
of the physicians	11
and at receptor	11
sars and mers	11
system in the	11
of the molecule	11
depends on the	11
the active sites	11
entry of sars	11
hypertensive patients with	11
enzyme inhibitors angiotensin	11
mortality and morbidity	11
in relation to	11
ace is the	11
coronavirus in wuhan	11
to be associated	11
the hungarian hae	11
enzyme from the	11
it is used	11
in those with	11
of action of	11
the mineralocorticoid receptor	11
in understanding sars	11
dependent on the	11
concentration of c	11
by the sars	11
the virus may	11
role for ace	11
in hospitalized patients	11
from patients with	11
evidence that the	11
and prevention of	11
plasma concentrations of	11
cases of covid	11
presence of the	11
cornell et al	11
in vitro studies	11
circulating ang ii	11
the receptor for	11
remains to be	11
to the n	11
is believed to	11
molecular weight kininogen	11
at late stage	11
and the presence	11
of an ace	11
of ischemic stroke	11
the site of	11
epithelial cells of	11
act as a	11
been found to	11
is the main	11
used to treat	11
a clinical trial	11
first step in	11
with a new	11
is highly expressed	11
outbreak in china	11
reduced ejection fraction	11
as a sars	11
not yet been	11
enzyme and angiotensin	11
endothelial dysfunction in	11
a marker of	11
on the cell	11
to determine the	11
be able to	11
of angiotensin receptor	11
is an endogenous	11
binding of the	11
a consequence of	11
are able to	11
c domain of	11
enzyme inhibition on	11
step in understanding	11
development of a	11
expression profiling of	11
may be due	11
patients receiving ace	11
hfsa acc aha	11
of type diabetes	11
stage as compared	11
guidelines for the	11
at receptor activation	11
to prevent the	11
for heart failure	11
sequence identity with	11
as high as	11
angiotensin type receptor	11
statement of the	11
basis of the	11
ischaemic heart disease	11
et al b	11
the balance between	11
at higher risk	11
of at r	11
or ace inhibitors	11
the processing of	11
can also be	11
between ace and	11
c and c	11
crackower et al	11
in the late	11
db and cont	10
can be used	10
suggests that the	10
inpatient use of	10
patients who have	10
a series of	10
novel coronavirus from	10
a new coronavirus	10
this is the	10
the severity and	10
and severe acute	10
with acquired c	10
effects of captopril	10
with type diabetes	10
could potentially be	10
cardiac and renal	10
chen et al	10
included in the	10
in vascular endothelial	10
these data suggest	10
human coronavirus nl	10
and protein expression	10
in the uk	10
mechanisms and potential	10
entry into cells	10
patients with pneumonia	10
could be used	10
of icu admission	10
not modify ace	10
db as compared	10
homologue of angiotensin	10
the recognition of	10
t t t	10
coronary blood flow	10
recombinant human ace	10
comparison of the	10
protein expression in	10
the duration of	10
differences in ace	10
type diabetes and	10
large number of	10
to human ace	10
syndrome coronavirus spike	10
as compared with	10
activity was significantly	10
ang ii or	10
severe lethal disease	10
and can be	10
is the first	10
region of the	10
and patients with	10
in the last	10
epithelial cells and	10
be effective in	10
course of sars	10
and type ii	10
of the new	10
shown that the	10
with essential hypertension	10
of angiotensin peptides	10
actions of angiotensin	10
in substrate specificity	10
admitted to the	10
of hypertension in	10
was followed by	10
the reninangiotensin system	10
has been described	10
action of angiotensin	10
of hae and	10
it has a	10
of the mas	10
injury induced by	10
both catalytically active	10
i arbs treatment	10
ratio was significantly	10
with mortality among	10
evidence that ace	10
mortality among patients	10
patients hospitalized with	10
polymorphism in the	10
with myocardial infarction	10
effects of at	10
can lead to	10
leads to a	10
factor xii activation	10
of the literature	10
in women with	10
heart failure patients	10
is expressed on	10
its interaction with	10
the action potential	10
ace inhibitors have	10
to reduce the	10
need to be	10
a potent inhibitor	10
a recent study	10
ferrario et al	10
shedding of ace	10
chinese center for	10
demonstrated that the	10
the mechanism of	10
plays an important	10
in some cases	10
and morbidity in	10
of natriuretic peptides	10
and arbs have	10
be used to	10
linked to the	10
the distribution of	10
and on the	10
by binding to	10
smooth muscle contraction	10
viral load and	10
the crystal structure	10
of the clinical	10
aldosterone system blockers	10
more susceptible to	10
in akita hnrnpf	10
to the human	10
is composed of	10
on the role	10
in the shr	10
is difficult to	10
plays a role	10
and potential therapeutic	10
in different tissues	10
high levels of	10
dysfunction after myocardial	10
the decrease in	10
of the lung	10
supplied by the	10
higher levels of	10
been linked to	10
of bradykinin by	10
discovery of ace	10
in endothelial cells	10
molecular mechanisms and	10
mas receptor in	10
have been made	10
oxidative stress and	10
the reduction in	10
very severe lethal	10
upregulation of ace	10
major role in	10
receptor blockers and	10
knowledge of the	10
inhibition of sars	10
this leads to	10
converting enzyme homologue	10
in the mouse	10
increased in the	10
of lung injury	10
in patients taking	10
mutations in the	10
is required for	10
in this context	10
i or arbs	10
in cardiovascular and	10
isolation and characterization	10
of patients in	10
ventricular dysfunction after	10
with respect to	10
rats with myocardial	10
the proximal tubule	10
are likely to	10
of mammalian ace	10
in vascular smooth	10
center for disease	10
for ace in	10
raas antagonists in	10
may be involved	10
of c nh	10
the diagnosis and	10
results suggest that	10
severe heart failure	10
are required to	10
component of the	10
with that of	10
are both catalytically	10
in the liver	10
residues in the	10
to the sars	10
converting enzyme by	10
into the cell	10
bradykinin potentiating peptides	10
appear to be	10
that of the	10
insulin administration significantly	10
with chronic heart	10
were observed in	10
is seen in	10
from the venom	10
arbs in the	10
ectodomain shedding of	10
and nitric oxide	10
gender differences in	10
of this study	10
presence of an	10
to an increase	10
for this reason	10
upper respiratory tract	10
ii receptor antagonist	10
to be involved	10
i and arb	10
at least one	9
blockers with mortality	9
are responsible for	9
was shown that	9
by severe acute	9
did not show	9
action of ace	9
the components of	9
of local renin	9
a protective role	9
in the cleavage	9
therapy in the	9
patients suffering from	9
head to head	9
in this review	9
via the at	9
chronic use of	9
is found in	9
induced acute lung	9
of endothelial cells	9
use of aceis	9
the virus can	9
of these two	9
susceptibility to sars	9
in patients receiving	9
protective effect of	9
in the adrenal	9
at receptor antagonists	9
substrate specificity and	9
of functional c	9
lung injury by	9
may be associated	9
harmful effects of	9
mild to moderate	9
interaction with ace	9
hormone replacement therapy	9
ischemic heart disease	9
of the central	9
statement addresses concerns	9
at receptor gene	9
evaluation of the	9
and function of	9
the availability of	9
identified as a	9
activated protein kinase	9
viral rna in	9
with an ace	9
ace inhibition and	9
and endothelial dysfunction	9
expressed in a	9
recombinant human angiotensin	9
showed that ace	9
when compared with	9
the aortic valve	9
account for the	9
angiotensinconverting enzyme inhibitors	9
risk of infection	9
complexity of the	9
the beneficial effects	9
blood pressure control	9
suggest that the	9
i and type	9
of death in	9
a recent report	9
at tissue sites	9
addresses concerns re	9
seem to be	9
been demonstrated to	9
viral entry and	9
reported in the	9
low molecular weight	9
patients at high	9
novel coronavirus sars	9
severe coronavirus disease	9
there may be	9
the mechanisms of	9
characteristics of the	9
at a receptor	9
activity of pgl	9
the coronary sinus	9
due to covid	9
should be considered	9
reduced risk of	9
the sympathetic nervous	9
be used as	9
in the plasma	9
increasing amounts of	9
discovery of the	9
disseminated intravascular coagulation	9
the hemoregulatory peptide	9
significantly higher in	9
activation of prorenin	9
be useful in	9
clinical trials are	9
expressed on the	9
the included studies	9
degradation of bradykinin	9
a protective effect	9
in recent years	9
position statement of	9
was the first	9
inh concentration and	9
correlated inversely with	9
similar to the	9
diabetic kidney disease	9
elevated levels of	9
of antihypertensive drugs	9
use in the	9
with hae have	9
inhibitors or arb	9
azizi et al	9
et al found	9
health and disease	9
terminal domain of	9
early in the	9
using raas antagonists	9
located on chromosome	9
number of patients	9
coronavirus infection in	9
role of ang	9
in the other	9
effect of losartan	9
acts as an	9
the chinese center	9
the disease and	9
high prevalence of	9
ace inhibitors on	9
r c allele	9
outcomes of patients	9
roles of the	9
shown in table	9
seen in the	9
by inhibiting the	9
reported in patients	9
increased ang ii	9
many of the	9
they have no	9
of the d	9
the left atrium	9
declare that they	9
systematic review of	9
in the study	9
of the total	9
has led to	9
activation of ace	9
the lung and	9
the right atrium	9
no evidence of	9
leads to increased	9
n domain of	9
enzyme are both	9
is supplied by	9
frequency of the	9
the extent of	9
the left ventricular	9
for the development	9
in humans and	9
duration of action	9
association between ace	9
of inpatient use	9
which can be	9
hyperoxia with normoxic	9
of hereditary angioneurotic	9
and clinical implications	9
the ace ang	9
is also a	9
the response to	9
at receptor is	9
into the host	9
more severe disease	9
ventricular outflow tract	9
ii receptor subtypes	9
patients taking ace	9
found that ace	9
ang ii degradation	9
contributing to the	9
group of patients	9
and death in	9
integral membrane protein	9
the significance of	9
by the use	9
than in the	9
regulator of the	9
identification of a	9
the virus and	9
new york city	9
at receptor expression	9
the loss of	9
by the severe	9
the nervous system	9
antagonists in covid	9
wall of the	9
tissue levels of	9
association of inpatient	9
those of the	9
been implicated in	9
and oxidative stress	9
increased amounts of	9
use of the	9
the kinin system	9
has been used	9
of women with	9
basis for the	9
the rbd ace	9
the classical ras	9
were shown to	9
tatei et al	9
role in reproduction	9
to bind to	9
terminal catalytic domain	9
the identification of	9
is and arbs	9
enzyme and its	9
is caused by	9
risk of icu	9
at the level	9
caused by a	9
by the action	9
along with the	9
but not by	9
released into the	9
wide variety of	9
have been used	9
no difference in	9
to understand the	9
captopril on mortality	9
of convalescent plasma	9
it is an	9
converting enzyme are	9
has been proposed	9
such as diabetes	9
we found that	9
of ace on	9
hydroxychloroquine and azithromycin	9
hypertension hospitalized with	9
it was shown	9
a wide variety	9
serve as a	9
as described previously	9
properties of the	9
mediated by at	9
at the same	9
of the mammalian	9
a higher risk	9
with the c	9
human coronavirus e	9
lung injury angiotensin	9
the right ventricular	9
in human lung	9
breakdown of bradykinin	9
that the ace	9
negative regulator of	9
angiotensin and bradykinin	9
in serum samples	9
impact of the	9
the m family	9
the process of	9
enzyme responsible for	9
in china clinical	9
ance and acer	9
there have been	9
outcomes in patients	9
lung injury induced	9
inactivation of the	9
the distinction between	9
of ace gene	9
acc aha statement	9
in comparison with	9
of the esc	9
in patients treated	9
in the angiotensin	9
inh concentrate is	9
amino acid sequence	9
response to ang	9
vickers et al	9
with normoxic recovery	9
be responsible for	9
guinea pig ileum	9
novel coronavirus pneumonia	9
virus entry into	9
in contrast with	9
and therapeutic implications	9
has recently been	9
angiotensin ii to	9
of the effects	9
coupled receptor mas	9
studies showed that	9
to investigate the	8
which is the	8
the p position	8
stimulation of the	8
the brain and	8
no evidence that	8
of captopril on	8
management of patients	8
related to increased	8
shows that the	8
is induced by	8
significant differences in	8
all of these	8
ace may be	8
smooth muscle cell	8
in coronavirus disease	8
in severe covid	8
could not be	8
under a author	8
influence of angiotensin	8
structural and functional	8
renin prorenin receptor	8
mrna expression profiling	8
hungarian hae center	8
of brain ras	8
treatment and prevention	8
rat model of	8
in an open	8
this could be	8
this type of	8
converting enzyme ii	8
the angiotensin i	8
recorded diagnosis of	8
on ace inhibitors	8
from that of	8
yang et al	8
hypertension on ace	8
ras has been	8
on the surface	8
liu et al	8
oxidative stress in	8
that it is	8
the introduction of	8
patients presenting with	8
management of hypertension	8
quantitative mrna expression	8
a novel homologue	8
is a type	8
performance liquid chromatography	8
form of ace	8
drosophila angiotensin i	8
in the formation	8
ang ii are	8
it is difficult	8
therapeutic benefits of	8
hemodynamic effects of	8
angiotensin system inhibitors	8
the upregulation of	8
at early stage	8
of human and	8
a receptor for	8
only in the	8
to explore the	8
late stage as	8
followed by the	8
analysis revealed that	8
ace deletion polymorphism	8
these drugs are	8
and is associated	8
point of view	8
fold increase in	8
glomerular injury in	8
were no differences	8
in a number	8
binding of sars	8
the occurrence of	8
of ace was	8
analysis of randomized	8
depend on the	8
activity ratio in	8
were found in	8
in a cohort	8
of aceis arbs	8
vivo and in	8
was increased in	8
and clinical characteristics	8
cd t cells	8
is dependent on	8
transmembrane serine protease	8
in transgenic mice	8
the first time	8
and kidney diseases	8
a better understanding	8
the immune response	8
human immunodeficiency virus	8
in the host	8
due to increased	8
symptomatic heart failure	8
relationship between ace	8
and of the	8
release from the	8
hereditary angioedema with	8
the d allele	8
regulation of ace	8
what is the	8
at r axis	8
a group of	8
administration of ace	8
due to a	8
of the mitral	8
the esc council	8
effects of ras	8
for the diagnosis	8
to conclude that	8
use of these	8
arbs or ace	8
been observed in	8
activity is reduced	8
lung epithelial cells	8
formation of the	8
the outcome of	8
on the clinical	8
the white group	8
of acute respiratory	8
with normal c	8
nitric oxide and	8
in the clinical	8
through activation of	8
absence of a	8
implications for the	8
in the binding	8
to block the	8
indicate that the	8
on the x	8
did not affect	8
is shown as	8
and ace activities	8
angiotensin ii levels	8
the observation that	8
a author funder	8
failure with reduced	8
useful in the	8
with reduced ejection	8
seen in patients	8
parts of the	8
of probable bat	8
used as a	8
type receptor in	8
ii receptor type	8
ii levels in	8
blood flow to	8
first case of	8
for the use	8
polymorphism is associated	8
risk of pneumonia	8
human lung fibroblasts	8
and endothelial cells	8
not appear to	8
kidney disease and	8
compared with non	8
coronavirus of probable	8
chronic hepatitis c	8
a model of	8
as ace inhibitors	8
license it is	8
are needed to	8
the zinc ion	8
is made available	8
and cont mice	8
china clinical characteristics	8
dependent inherited angioedema	8
r and at	8
exposed to hyperoxic	8
of an angiotensin	8
evidence suggests that	8
for use in	8
evidence of the	8
the ace angiotensin	8
in some patients	8
the replication of	8
hypoxic pulmonary vasoconstriction	8
human c inhibitor	8
third of the	8
by using a	8
activity was measured	8
coronavirus in the	8
to improve the	8
severity of the	8
isolated perfused rat	8
with and without	8
gp recorded diagnosis	8
a potent vasoconstrictor	8
for entry into	8
a negative regulator	8
in the cardiac	8
may be more	8
none of the	8
no differences in	8
and the cardiovascular	8
outbreak associated with	8
patients with symptomatic	8
the lungs of	8
protein kinase c	8
to compare the	8
during ace inhibition	8
in health and	8
and angiotensin converting	8
peptide levels in	8
belong to the	8
made available under	8
to the increased	8
international license it	8
well as their	8
data on the	8
studies of the	8
patients with acquired	8
as described in	8
considered to be	8
setting of covid	8
from the coronavirus	8
is required to	8
of bradykinin in	8
new coronavirus of	8
the g protein	8
differences between the	8
lead to an	8
the study was	8
late stages of	8
in plasma and	8
drosophila ace orthologous	8
it is made	8
of the different	8
listed in table	8
gene insertion deletion	8
is a natural	8
time of the	8
characteristics of and	8
host cell entry	8
left and right	8
and kidney disease	8
in the proximal	8
actions of ace	8
and neutral endopeptidase	8
with chronic kidney	8
in the processing	8
significant role in	8
is used to	8
better understanding of	8
aceis or arbs	8
renin and prorenin	8
at receptor and	8
suggesting that the	8
in one study	8
cd and cd	8
type natriuretic peptide	8
the same time	8
has shown that	8
human angiotensinconverting enzyme	8
the adrenal gland	8
in the kidneys	8
as discussed in	8
was detected in	8
with c inhibitor	8
that ace activity	8
in the male	8
associated with acute	8
enzyme inhibitors in	8
prevention and treatment	8
with human ace	8
inhibitors of ace	8
at receptor blockers	8
that may be	8
features of the	8
treatment of chronic	8
isaac et al	8
recombinant human c	8
from the chinese	8
the classical pathway	8
ace inhibition may	8
of patients admitted	8
is a novel	8
the blood pressure	8
in vivo studies	8
the receptor binding	8
a pneumonia outbreak	8
mice as compared	8
and characteristics of	8
a significant role	8
mrna and protein	8
heart failure angiotensin	8
day follow up	8
the binding site	8
for the n	8
on hypertension on	8
in support of	8
ace inhibitors is	8
act on the	8
report of cases	8
was based on	8
be prevented by	8
high molecular weight	8
were performed in	8
was not peer	8
and the at	8
with diabetes and	8
release of the	8
estimates of the	8
a broad spectrum	8
with diabetes mellitus	8
difference in the	8
in light of	8
did not have	8
at room temperature	8
the ability to	8
on the contrary	8
of severe disease	8
risk of mortality	8
have been associated	8
in a similar	8
aha statement addresses	8
needs to be	8
esc council on	8
its effect on	8
is not known	8
tipnis et al	8
structural basis for	8
santos et al	8
of cases from	8
a significant increase	8
the interaction between	8
was able to	8
for a wide	8
may not be	8
probable bat origin	8
important role of	8
in the airways	8
for ace inhibitors	8
reduction in the	8
to determine whether	8
circulating levels of	8
of the septum	8
and the right	8
the catalytic zinc	8
have been proposed	8
available under a	8
in the usa	8
and that the	8
burnham et al	8
fluid and electrolyte	8
of the effect	8
for angiotensin ii	8
as opposed to	8
is characterized by	8
diseases such as	8
this is not	8
should be noted	8
binding to ace	8
has been observed	8
in akita mice	8
elucidation of the	8
coronavirus spike proteins	8
ace have been	8
to be an	8
the creative commons	8
been shown that	8
a mortality rate	8
endothelial nitric oxide	8
in the risk	8
novel homologue of	8
and the c	8
in the general	8
pneumonia outbreak associated	8
not been shown	8
acute coronary syndrome	8
of infected patients	8
both early and	8
that has been	8
at high risk	8
clinical and biochemical	8
converting enzyme the	8
of reactive oxygen	8
metabolism in the	8
ii at receptor	8
data suggest that	8
ratio in db	8
of the isolated	8
to the development	7
those in the	7
as an endopeptidase	7
need for a	7
with a high	7
administration of the	7
angiotensin ii on	7
administration significantly decreased	7
there has been	7
infected patients linked	7
the catalytic site	7
identification and characterization	7
task force on	7
specific substrate of	7
ace gene polymorphisms	7
was carried out	7
danser et al	7
accumulation of ang	7
is in the	7
side effects are	7
have been infected	7
found that the	7
generation of angiotensin	7
for inhibitor binding	7
is due to	7
with permission from	7
this results in	7
endothelial growth factor	7
antihypertensive effects of	7
prevalence of hypertension	7
ratio of ace	7
the idea that	7
the death rate	7
differential regulation of	7
increase in ang	7
structures of the	7
shown as sticks	7
was higher in	7
ncov infected patients	7
increased in ad	7
is the primary	7
structural requirements for	7
even in the	7
study clinical characteristics	7
the arrival of	7
viral loads and	7
the safety and	7
incubation period of	7
also associated with	7
of acquired c	7
of this article	7
may have been	7
an essential role	7
of ace as	7
that have been	7
as in ace	7
homologue of ace	7
potential influence of	7
due to their	7
linked to viral	7
for the recognition	7
been detected in	7
molecular basis of	7
for many years	7
to viral loads	7
those who have	7
the frequency and	7
is similar to	7
involvement of the	7
fetal lung fibroblasts	7
of the activation	7
cardiovascular implications of	7
of and important	7
treated with the	7
not included in	7
characteristics of hospitalized	7
degradation of the	7
trial of recombinant	7
ii levels were	7
bar chart showing	7
stages as compared	7
differential expression of	7
reprinted with permission	7
catalytic domains of	7
may be beneficial	7
characteristics and outcomes	7
not result in	7
restricted to the	7
i arbs therapy	7
substrate and c	7
ace gene polymorphism	7
a type i	7
of morbidity and	7
noted that the	7
biochemical indexes from	7
spike glycoprotein of	7
increased ace expression	7
mediates regulated ectodomain	7
on ang ii	7
focus on the	7
lessons from the	7
and specific substrate	7
may be increased	7
for management of	7
to generate ang	7
therapeutic implications in	7
em structure of	7
of cases of	7
a small study	7
implications for covid	7
of the cell	7
identification of the	7
is independent of	7
downregulation of the	7
in conjunction with	7
were exposed to	7
mortality associated with	7
natural and specific	7
diagnosis of covid	7
may be useful	7
the primary outcome	7
that could be	7
and an increase	7
the side effects	7
and in vitro	7
substrate and inhibitor	7
have been developed	7
cough and angioedema	7
decrease in blood	7
does not require	7
of the anoace	7
the specificity of	7
treatment with the	7
myocardial o consumption	7
gene expression levels	7
effects of bradykinin	7
chappell et al	7
angiotensin iconverting enzyme	7
components in the	7
the gene encoding	7
on the ace	7
was approved by	7
changes were observed	7
in the cns	7
linz et al	7
can be activated	7
by bork et	7
beta blocker therapy	7
the last decade	7
a pilot clinical	7
ras and the	7
pro is a	7
in the ras	7
b and c	7
distribution of angiotensin	7
hemoregulatory peptide n	7
mice at late	7
be attributed to	7
we did not	7
this study was	7
can result in	7
lung fibrosis in	7
in of patients	7
shown to inhibit	7
attacks of hae	7
and drug administration	7
blood pressure homeostasis	7
insect ace homologues	7
at the c	7
of the drug	7
inh deficiency is	7
the bradykinin receptor	7
demonstrated that ace	7
but it is	7
it was found	7
an analysis of	7
were able to	7
the development and	7
i into angiotensin	7
the result of	7
loss of ace	7
to the ras	7
of the action	7
and uncaric acid	7
on the renin	7
in of the	7
and found that	7
be avoided in	7
presence of at	7
diabetes and covid	7
no evidence for	7
been attributed to	7
has been detected	7
to what extent	7
the understanding of	7
severe disease and	7
chloroquine and hydroxychloroquine	7
and did not	7
metabolism of ang	7
expression levels in	7
to infect humans	7
was supported by	7
of potentiating peptides	7
testing positive for	7
interactions with the	7
may be of	7
of the substrate	7
from the same	7
regulation of angiotensin	7
in host cells	7
plasma and tissue	7
to identify the	7
substrate of the	7
is the major	7
effective refractory period	7
ace insertion deletion	7
was obtained from	7
the way for	7
of the main	7
was significantly higher	7
vascular endothelial growth	7
was linked to	7
because of its	7
the renin receptor	7
there are several	7
is available to	7
soluble human ace	7
its ability to	7
terminal active site	7
there are also	7
binds to ace	7
ang ii were	7
increase in myocardial	7
higher prevalence of	7
patients with c	7
resulted in a	7
using ace inhibitors	7
a history of	7
and increase in	7
the raas is	7
severe lethal covid	7
development of the	7
and functional characterization	7
after adjusting for	7
and ulcerative colitis	7
coronavirus associated with	7
in the gastrointestinal	7
cases from the	7
have not yet	7
the respiratory tract	7
lowering blood pressure	7
be activated by	7
ang ii was	7
at receptor axis	7
as a model	7
of arterial hypertension	7
in myocardial infarction	7
an in vitro	7
functions as a	7
effects of losartan	7
not be used	7
ang ii generation	7
with angiotensin ii	7
drugs in the	7
physiological relevance and	7
is cleaved by	7
hae and aae	7