trigram

This is a table of type trigram and their frequencies. Use it to search & browse the list to learn more about your study carrel.

trigram	frequency
of ebola virus	404
ebola virus disease	225
the ebola virus	187
ebola virus infection	173
ebola virus glycoprotein	160
as well as	126
of the ebov	116
of the ebola	110
ebola hemorrhagic fever	103
ebola and marburg	102
of ebov gp	100
vesicular stomatitis virus	96
of marburg virus	94
the presence of	93
virus disease in	88
democratic republic of	85
due to the	77
respiratory syndrome coronavirus	77
for ebola virus	76
the plasma membrane	73
the role of	70
vero e cells	69
of the congo	66
republic of the	65
severe acute respiratory	64
in west africa	63
virus infection in	63
the development of	62
of the virus	62
and marburg viruses	62
the absence of	61
the cell surface	61
in this study	60
against ebola virus	60
acute respiratory syndrome	59
in order to	58
was used to	57
middle east respiratory	57
east respiratory syndrome	56
in the absence	55
based on the	55
human immunodeficiency virus	54
of the viral	54
one of the	54
the use of	54
of ebola hemorrhagic	54
the treatment of	54
structure of the	53
ebola virus vp	53
type i ifn	50
in the presence	50
marburg hemorrhagic fever	50
been shown to	50
ebola virus in	49
in sierra leone	47
a number of	47
analysis of the	47
for h at	47
ebola virus entry	46
is essential for	46
world health organization	45
ebov gp pseudotyped	45
the number of	44
treatment of ebola	44
at room temperature	43
the ebov gp	43
with ebola virus	43
in nonhuman primates	43
according to the	41
the ability of	41
hemorrhagic fever in	41
of ebola and	40
ebola haemorrhagic fever	40
hepatitis c virus	40
the course of	40
the united states	39
in ebola virus	38
shown to be	38
as previously described	38
transcription and replication	38
the context of	37
of type i	37
we found that	37
the cells were	37
in addition to	37
the democratic republic	37
it has been	37
ebov and marv	36
t cells were	36
the efficacy of	36
the risk of	36
gp and gp	36
the identification of	36
the expression of	35
for the treatment	35
well as the	35
in patients with	34
in the context	34
to ebola virus	34
compared to the	34
is required for	33
cd t cells	33
for min at	33
were used to	32
a total of	32
to assess the	32
in vitro and	32
ebov infection in	32
thermo fisher scientific	31
the surface of	31
in the democratic	31
recombinant vesicular stomatitis	31
lethal ebola virus	31
has been shown	31
vp and vp	31
the importance of	31
a role in	31
the west african	31
the ebov infection	30
part of the	30
nonhuman primates against	30
is associated with	30
and marburg virus	30
such as the	30
zaire ebola virus	30
patients with ebola	29
found to be	29
on the cell	29
the host cell	29
republic of congo	28
immunodeficiency virus type	28
dependent rna polymerase	28
in western africa	28
to determine the	28
of the host	28
type i interferon	28
of ebov and	28
was added to	28
of ebov infection	27
the detection of	27
a variety of	27
the activity of	27
geisbert et al	27
that can be	27
as compared to	27
used for the	27
and in vivo	27
were transfected with	27
is able to	27
marburg and ebola	27
the case of	27
the viral envelope	27
colored fruit bat	26
npc domain c	26
influenza a virus	26
of the gp	26
in the ebov	26
vitro and in	26
processing of the	26
from ebola virus	26
in the case	26
has also been	26
on the surface	26
to ebov gp	26
of the disease	26
in humans and	26
infected with ebov	25
of the human	25
the production of	25
with ebov gp	24
the effect of	24
the onset of	24
has not been	24
by ebola virus	24
the lack of	24
the formation of	24
of the most	24
ebola virus and	24
to ebov infection	24
was used for	23
the extent of	23
were treated with	23
most of the	23
the effects of	23
in the united	23
of the nucleocapsid	23
in contrast to	23
have been shown	23
role of the	23
in our study	23
for ebov gp	23
as described previously	23
associated with the	23
cells were transfected	23
have been developed	23
in the viral	23
samples were collected	23
and ebola viruses	23
in vero e	23
viruses such as	22
protects nonhuman primates	22
members of the	22
pathogenesis of ebola	22
observed in the	22
on the other	22
the fact that	22
cells were seeded	22
each of the	22
as shown in	22
mcelroy et al	22
entry and infection	22
in rhesus macaques	22
used as a	22
the ebov genome	22
a and b	22
have been reported	22
ebola virus gp	22
ebov gp and	22
characterization of the	22
against a lethal	22
role in the	22
the present study	21
in cell culture	21
added to the	21
gp on the	21
virus entry requires	21
found in the	21
disease in guinea	21
wide range of	21
type i ifns	21
ebov gp cl	21
of patients with	21
of zaire ebolavirus	21
development of a	21
there was no	21
the ifn system	20
outbreak of ebola	20
appears to be	20
detected in the	20
the glycan cap	20
entry mediated by	20
cholesterol transporter niemann	20
contribute to the	20
some of the	20
linked immunosorbent assay	20
can be used	20
to be the	20
ebov gp is	20
at h post	20
against lethal ebola	20
probability of introduction	20
the structure of	20
entry requires the	20
were added to	20
centers for disease	20
of infectious diseases	20
the other hand	20
virus glycoprotein is	20
host cell entry	20
drnpc domain c	20
the level of	20
changes in the	20
there is a	20
in response to	20
because of the	20
an important role	20
of the vsv	20
high levels of	20
the addition of	20
in cynomolgus macaques	20
cells were washed	20
viral replication and	20
to be a	20
showed that the	20
of severe acute	20
mediated by the	20
and nonhuman primates	20
one or more	20
fruit bat cells	19
the probability of	19
volchkov et al	19
in combination with	19
enclomiphene and zuclomiphene	19
remains to be	19
antiviral activity of	19
hemorrhagic fever virus	19
the generation of	19
extent of fusion	19
t cell responses	19
the majority of	19
not required for	19
for the detection	19
and sierra leone	19
were obtained from	19
by the ebola	19
have been used	19
and ebola virus	19
has been reported	19
a mouse model	19
in which the	19
the ability to	19
a wide range	19
in the current	19
marburg virus disease	19
hek t cells	19
has been used	18
disease control and	18
to the plasma	18
thought to be	18
the spread of	18
against the ebov	18
gp and ebov	18
associated with fatal	18
of marburg hemorrhagic	18
data not shown	18
to the cell	18
were found to	18
were infected with	18
ebola virus glycoproteins	18
marburg virus infection	18
the immune system	18
important role in	18
during the outbreak	18
for disease control	18
ebola virus replication	18
in the plasma	18
immune responses in	18
the inhibition of	18
was found to	18
the cholesterol transporter	18
been detected in	18
was removed and	18
activity of the	18
replication and transcription	18
analysis of ebola	18
resulted in a	18
be used to	18
as described in	17
against ebov infection	17
case fatality rate	17
may not be	17
of the family	17
the respiratory tract	17
included in the	17
assembly and budding	17
is necessary for	17
control and prevention	17
the marburg virus	17
suggest that the	17
essential for ebola	17
as a result	17
balb c mice	17
to that of	17
indicating that the	17
in the treatment	17
west nile virus	17
of middle east	17
a single dose	17
a human survivor	17
haemorrhagic fever in	17
transmission of ebola	17
b and l	17
differences in the	17
of the protein	17
for treatment of	17
be due to	17
of this study	17
infection ebola virus	17
protection against lethal	17
are associated with	17
gp pseudotyped hiv	17
was carried out	17
results suggest that	17
proteolysis of the	17
is thought to	17
similar to the	17
ebola virus challenge	16
has been demonstrated	16
respiratory syncytial virus	16
it is possible	16
marburg virus and	16
pick c is	16
tumor necrosis factor	16
responsible for the	16
min at room	16
small molecule inhibitors	16
and ebov gp	16
from a human	16
c is essential	16
an moi of	16
virus entry by	16
outbreak in the	16
antiviral activity against	16
is responsible for	16
used in the	16
in terms of	16
all of the	16
protection against ebov	16
is shown in	16
interact with the	16
of the patients	16
the age of	16
proteolytic processing of	16
presence of the	16
was determined by	16
at least one	16
it is not	16
antisera and f	16
a case of	16
of filovirus infection	16
there is no	16
was shown to	16
to bind to	16
the family filoviridae	16
of the immune	16
live ebov neutralisation	16
passive transfer of	16
and stored at	16
cd t cell	16
an outbreak of	16
of the ifn	16
as described above	16
have also been	16
requires the cholesterol	15
that of the	15
course of the	15
the envelope glycoprotein	15
no competing interests	15
elevated levels of	15
during the course	15
i ifn signaling	15
the induction of	15
in the philippines	15
by flow cytometry	15
of zaire ebola	15
components of the	15
safety and immunogenicity	15
insight into the	15
is one of	15
dendritic cells and	15
mechanism of action	15
and immunogenicity of	15
the pathogenesis of	15
equine encephalitis virus	15
is consistent with	15
region of the	15
the impact of	15
been associated with	15
during the west	15
by ebola and	15
similar to that	15
the matrix protein	15
in the development	15
interfere with the	15
was associated with	15
a range of	15
play a role	15
the need for	15
the binding site	15
for detection of	15
virus infection with	15
the viral genome	15
protection against ebola	15
the potential to	15
were observed in	15
involved in the	15
the gp subunit	15
and in the	15
cellular entry by	15
a combination of	15
protective efficacy of	15
ebola virus is	15
ebola virus transmission	15
have been identified	15
comparison of the	15
disease in humans	15
of the fusion	14
the medium was	14
of the epidemic	14
emerging infectious diseases	14
of proinflammatory cytokines	14
diagnosis of ebola	14
in guinea pigs	14
and found that	14
molecule inhibitors reveal	14
response to the	14
host immune response	14
fetal bovine serum	14
a dose of	14
virus vp protein	14
single amino acid	14
is an important	14
glycoproteins of ebola	14
is critical for	14
in the future	14
cells were treated	14
into host cells	14
of influenza a	14
the authors declare	14
of the filovirus	14
glycoprotein bound to	14
extents of fusion	14
the entry of	14
high degree of	14
u a u	14
a u a	14
demonstrated that the	14
in the present	14
evaluation of the	14
their ability to	14
the binding of	14
added to each	14
to each well	14
of infected cells	14
west african ebola	14
shown in fig	14
for marburg virus	14
virus in the	14
was detected in	14
matrix protein vp	14
of the three	14
vaccine protects nonhuman	14
we show that	14
is possible that	14
west african outbreak	14
disease in the	14
incubated for h	14
inhibitors reveal niemann	14
the three areas	14
is composed of	14
outbreak in west	14
shown in figure	14
the viral rna	14
of the cell	14
may be a	14
of up to	14
viral hemorrhagic fever	14
and incubated at	14
rift valley fever	14
in south korea	14
and target cells	14
the recent outbreak	14
emerging and re	14
supplementary figure s	14
obtained from the	14
was detected by	14
lethal dose of	14
the filoviridae family	14
fusion of the	13
for the development	13
the evolution of	13
and it is	13
be used as	13
the possibility of	13
are thought to	13
of ebov in	13
of guinea pigs	13
and marburg hemorrhagic	13
it is likely	13
equine antisera and	13
jui government hospital	13
ebola virus outbreak	13
in the ebola	13
at the same	13
antibodies to ebola	13
the end of	13
antibodies against ebov	13
cell entry of	13
sign and dc	13
replication of the	13
with or without	13
crystal structure of	13
the a u	13
could not be	13
the ebov glycoprotein	13
prior to the	13
of nonhuman primates	13
of viral rna	13
the discovery of	13
for host cell	13
increasing or decreasing	13
the finding that	13
the transmembrane domain	13
on the plasma	13
african green monkey	13
cells were infected	13
in the lungs	13
is involved in	13
disseminated intravascular coagulation	13
due to their	13
of viral replication	13
innate and adaptive	13
amino acid residues	13
disease in west	13
it is important	13
highly pathogenic avian	13
guinea pigs and	13
the selection of	13
were determined by	13
that ebov gp	13
this work was	13
of enveloped viruses	13
after the onset	13
can also be	13
the receptor binding	13
emergence of zaire	13
into the host	13
the outbreak in	13
to account for	13
ebov infection and	13
case fatality rates	13
of a novel	13
in the mouse	13
by a single	13
of experimental ebola	13
fever in cynomolgus	13
in evd patients	13
the time of	13
entry of ebola	13
the host immune	13
both ebov and	13
lake victoria marburgvirus	13
were incubated with	13
fatality rate of	13
patients with severe	13
associated with an	13
be able to	13
to evaluate the	13
luciferase reporter gene	13
the function of	13
at least in	13
the determination of	13
lethal ebov infection	13
the replication of	13
resulted in the	13
fda approved drugs	13
type i interferons	13
member of the	12
the potential of	12
cleaved ebov gp	12
and characterization of	12
gp pseudotyped vsv	12
depending on the	12
were carried out	12
ebov gp on	12
simian immunodeficiency virus	12
huang et al	12
the basis of	12
a series of	12
necessary for infection	12
this study was	12
the in vitro	12
are important for	12
this is a	12
were able to	12
were collected and	12
large number of	12
pseudotyped virus neutralisation	12
the levels of	12
associated with a	12
regions of the	12
efficacy of remdesivir	12
factors that influence	12
a panel of	12
during the recent	12
were seeded in	12
om and esom	12
are required for	12
clomiphene and toremifene	12
for use in	12
and endothelial cells	12
in the field	12
the gp gene	12
h n virus	12
of viral entry	12
to public health	12
personal protective equipment	12
are able to	12
increased risk of	12
endosomal proteolysis of	12
cytokines and chemokines	12
been reported to	12
the prevalence of	12
innate immune responses	12
the most potent	12
polymerase chain reaction	12
of gp and	12
the middle east	12
is likely that	12
in a mouse	12
inhibit ebola virus	12
virus infection is	12
in healthy adults	12
venezuelan equine encephalitis	12
creative commons license	12
medium was replaced	12
cleavage of gp	12
adaptive immune responses	12
glycoprotein is necessary	12
the release of	12
h n influenza	12
pathogenic avian influenza	12
and public health	12
the world health	12
of years of	12
a case fatality	12
glycoprotein by the	12
viral fusion proteins	12
based on a	12
were generated by	12
influenza a viruses	12
cleavage of ebov	12
virus can be	12
are needed to	12
were transferred to	12
were performed in	12
formation of the	12
serial dilutions of	12
during the first	12
immune response to	12
ebola virus infections	12
from infected cells	12
of ebola viruses	12
of evd patients	12
play an important	12
to infection by	12
national institutes of	12
is important to	12
the length of	12
as seen in	12
the endoplasmic reticulum	12
it should be	12
that it is	12
the causative agent	12
the type i	12
followed by a	12
have shown that	12
with regard to	12
ebola outbreak in	12
caused by the	12
to a lesser	12
of public health	12
the mechanism of	12
marv and ebov	12
virus glycoprotein by	12
phase i clinical	12
end of the	12
the amount of	12
between the two	12
were purchased from	12
in the same	12
a single amino	12
binding in the	11
in african straw	11
higher levels of	11
dependent on the	11
was expressed with	11
and vlp entry	11
activity against ebov	11
the outbreak of	11
infection is associated	11
the activation of	11
is mediated by	11
after the first	11
derived from the	11
the first time	11
it is also	11
patients with evd	11
a review of	11
at the plasma	11
of pseudotyped virus	11
institutes of health	11
years of epidemiological	11
infection in the	11
of mg kg	11
pick c small	11
specific for the	11
gp is cleaved	11
used in this	11
suggests that the	11
entry into the	11
ebov infection is	11
of monoclonal antibodies	11
compared with pbs	11
and h n	11
internal fusion loop	11
at the time	11
a compendium of	11
assessment of the	11
the ebov is	11
of human immunodeficiency	11
advanced ebola virus	11
and laboratory studies	11
transmission during the	11
entry driven by	11
hepatitis b virus	11
multiple sequence alignment	11
in insect cells	11
terminal heptad repeat	11
were found in	11
a broad range	11
and cd t	11
g for min	11
egyptian fruit bats	11
in the golgi	11
virion glycoproteins of	11
a lethal dose	11
and gp i	11
avian influenza virus	11
a result of	11
interactions with the	11
reported to be	11
the possibility that	11
glycan cap and	11
infected with the	11
of host immune	11
located in the	11
all animals were	11
the low ph	11
of the conserved	11
the percentage of	11
patients in the	11
single dose of	11
viral rna polymerase	11
cellular entry of	11
likely due to	11
form of the	11
the emergence of	11
fecal samples were	11
cells were then	11
led to the	11
viral life cycle	11
ebov entry and	11
or decreasing trend	11
of a small	11
food and drug	11
cell lines were	11
among the three	11
clinical management of	11
number of cases	11
the site of	11
the virus to	11
is known to	11
in the cytoplasm	11
transfected with the	11
were associated with	11
pick type c	11
ebov and mers	11
binding of the	11
ebola virus can	11
was supported by	11
compendium of years	11
characterization of a	11
in the early	11
i and s	11
during an outbreak	11
broad range of	11
simons et al	11
show that the	11
and huh cells	11
to an antibody	11
the gp of	11
the antiviral activity	11
patients infected with	11
and neutralizing antibody	11
were shown to	11
the results of	11
with pbs group	11
was observed in	11
of neutralizing antibodies	11
h at room	11
bind to ebov	11
there was a	11
of vesicular stomatitis	11
in an outbreak	11
proinflammatory cytokines and	11
due to a	11
the potential for	11
to the ebov	11
into the eu	11
the gxxxg motif	11
dendritic cells are	11
of the different	11
mouse model of	11
in transfected cells	11
cell lines and	11
nature of the	11
understanding of the	11
assumed to be	11
present in the	11
h post transfection	11
to determine whether	11
the survival of	11
vp is essential	11
ebola virus nucleocapsid	11
identification of a	11
data indicate that	11
the current study	11
in rhesus monkeys	11
the plates were	11
a type i	11
and mucin domain	11
of rhesus macaques	11
sanchez et al	11
of remdesivir in	11
in recent years	11
with the ebov	11
have demonstrated that	11
there have been	11
of these viruses	11
are shown in	11
e cells were	10
direct contact with	10
based on these	10
motif in the	10
expressing ebola virus	10
signs of disease	10
virus glycoprotein bound	10
against ebov gp	10
reid et al	10
in human cells	10
yellow fever virus	10
an antibody from	10
can be effectively	10
early and sustained	10
those of the	10
immunoglobulin and mucin	10
the application of	10
viral spread in	10
were used for	10
length of the	10
the effectiveness of	10
inoculum was removed	10
for binding to	10
with a high	10
carette et al	10
cell immunoglobulin and	10
in ebov infected	10
the common marmoset	10
of the zaire	10
up to days	10
marburg virus infections	10
cathepsins b and	10
final concentration of	10
which is a	10
the ebov lifecycle	10
to the viral	10
depends on the	10
identification of the	10
many of the	10
animal models of	10
are early and	10
the ratio of	10
filovirus entry into	10
for the ebov	10
we used the	10
performed as described	10
steps in the	10
were performed using	10
over the course	10
months of the	10
the fusion loop	10
not yet been	10
in the gp	10
been observed in	10
the vp protein	10
monocytes and macrophages	10
and use of	10
the epidemic and	10
of evd and	10
role of vp	10
antibody from a	10
the genome of	10
of filovirus entry	10
work was supported	10
like particles produced	10
and interpretation of	10
fusion induced by	10
interpretation of data	10
host immune responses	10
in viral entry	10
the existence of	10
activation of the	10
to filovirus infection	10
persistence of ebola	10
the mouse model	10
as a potential	10
and sustained targets	10
was able to	10
surface of the	10
insights into the	10
sexual transmission of	10
to this end	10
required for ebov	10
were detected in	10
when compared to	10
for the first	10
characterization of ebola	10
on the basis	10
was quantified using	10
and vesicular stomatitis	10
composed of a	10
of the antibody	10
it may be	10
at an moi	10
yang et al	10
innate immunity and	10
of advanced ebola	10
appear to be	10
furin cleavage site	10
is not a	10
of ph n	10
of sars coronavirus	10
in infected cells	10
mutations in the	10
low levels of	10
the sites of	10
been found to	10
for membrane fusion	10
and replication of	10
in the late	10
our understanding of	10
the amino acid	10
along with the	10
we propose that	10
of the year	10
of the outbreak	10
the observation that	10
by interfering with	10
leroy et al	10
removal of the	10
entry by ebola	10
binding site of	10
of the rna	10
may result in	10
green monkey kidney	10
could be used	10
for viral entry	10
multiplicity of infection	10
the time to	10
contributed to the	10
no evidence of	10
is believed to	10
virus infection by	10
cases in the	10
supernatants were harvested	10
estimates of d	10
been shown that	10
successful treatment of	10
by reverse transcription	10
suggesting that the	10
the persistence of	10
an inhibitor of	10
the viral glycoprotein	10
valley fever virus	10
from the same	10
was obtained from	10
the stability of	10
risk factors for	10
against ebola and	10
was observed for	10
ebola virus nucleoprotein	10
humoral immune response	10
these results suggest	10
clomiphene and enclomiphene	10
bound to an	10
nonhuman primate model	10
inhibition of the	10
amino acid residue	10
a concentration of	10
a fraction of	10
infection of rhesus	10
and guinea pigs	10
the tnc mutations	10
none of the	10
and drug administration	10
the transmission of	10
blood samples from	10
dcs and macrophages	10
marburg virus in	10
is internalized into	10
antibodies against ebola	10
the size of	10
vero e and	10
of vp in	10
seen in figure	10
the viral life	10
of the glycoprotein	10
mev rpv pprv	10
gp to the	10
infection resulted in	10
of at least	10
ebola virus from	10
of the evd	10
a u and	10
is the most	10
of the risk	10
ebov or marv	10
a lethal challenge	10
is a key	10
is dependent on	10
be considered as	10
c small molecule	10
morbidity and mortality	10
virus entry and	10
of the spike	10
in central africa	10
to be effective	10
in the range	9
early stages of	9
consistent with the	9
immunity and with	9
with fetal bovine	9
is important for	9
absence of other	9
the inoculum was	9
of a single	9
to provide a	9
antibody responses against	9
post disease onset	9
were maintained in	9
was detected using	9
as determined by	9
analysis and interpretation	9
at the cell	9
are known to	9
binding domain of	9
as part of	9
inhibition of ebov	9
functional characterization of	9
cells were maintained	9
ebov has been	9
of the type	9
virus infection ebola	9
are based on	9
strain of ebov	9
derived dendritic cells	9
was dependent on	9
targets of infection	9
infection in humans	9
cathepsin b and	9
peripheral blood mononuclear	9
that had been	9
the population coverage	9
ibrahim et al	9
mhc class i	9
the proportion of	9
has been described	9
of therapeutic antisera	9
of the two	9
with the endosomal	9
into the cytoplasm	9
effector and target	9
was based on	9
to have a	9
the external solution	9
binding to the	9
virus matrix protein	9
proportion of the	9
to interfere with	9
sustained targets of	9
the immune response	9
the interaction of	9
be important for	9
is cleaved by	9
and for the	9
a h n	9
the characterization of	9
plasmacytoid dendritic cells	9
a receptor for	9
of infection in	9
ebov infected mice	9
spike protein of	9
antibodies directed against	9
ebola virus origin	9
e and f	9
the total number	9
the equine antisera	9
a high degree	9
of sexual transmission	9
in both the	9
independent of ph	9
infection with a	9
causative agent of	9
virus origin and	9
h at rt	9
related to the	9
complete protection against	9
weeks after the	9
united states and	9
conflicts of interest	9
the h n	9
models of ebola	9
of the marburg	9
been reported in	9
is characterized by	9
three of the	9
dependent enhancement of	9
with an aberrant	9
in filovirus entry	9
high viral load	9
average number of	9
by macropinocytosis and	9
efficacy of the	9
been used to	9
approved by the	9
a biosafety level	9
and a u	9
in the environment	9
in the exporting	9
in the host	9
of human infection	9
and incubated with	9
class i fusion	9
that dendritic cells	9
a large number	9
pathology of experimental	9
prevention and control	9
virus and the	9
analysis was performed	9
to reduce the	9
released into the	9
the exporting country	9
h n and	9
been used in	9
incubation period of	9
the need to	9
a recent study	9
neutralizing antibody responses	9
aberrant innate immunity	9
c and d	9
size of the	9
in animal models	9
mediated restriction of	9
infected with ebola	9
prime boost vaccination	9
in evd survivors	9
and incubated for	9
in nonendemic countries	9
blood mononuclear cells	9
and control of	9
percentage of ebov	9
in experimentally infected	9
patients during the	9
ebola vaccine in	9
be explained by	9
directed against the	9
ebov gp to	9
should be noted	9
produced in insect	9
e and huh	9
likely to be	9
be noted that	9
evd outbreak in	9
the quality of	9
significant difference in	9
with a recombinant	9
days after the	9
human ebov infection	9
strand rna viruses	9
conformational changes in	9
against lethal challenge	9
incorporated into virions	9
authors declare no	9
from the cell	9
the range of	9
of viral particles	9
open reading frame	9
treatment of ebov	9
of ebov entry	9
in t cells	9
surveillance elucidates ebola	9
ebov in vitro	9
to interact with	9
virus has been	9
studies have demonstrated	9
was used as	9
ebov and other	9
of host cell	9
of the cases	9
first months of	9
mediated protection against	9
ebov gp that	9
of evd in	9
virus infection of	9
health care workers	9
elucidates ebola virus	9
role of type	9
required for ebola	9
is conceivable that	9
have been found	9
through transcriptional editing	9
standard error of	9
in some cases	9
pi k akt	9
cells by macropinocytosis	9
factors such as	9
are likely to	9
mediated cell entry	9
to inhibit the	9
are not required	9
and with massive	9
in hela cells	9
the ebov outbreak	9
the advantage of	9
outbreak in kikwit	9
against ebov in	9
we used a	9
a lethal ebov	9
marburg virus glycoprotein	9
in a phase	9
suggested that the	9
the feasibility of	9
it is conceivable	9
activity against ebola	9
we investigated the	9
at or dpi	9
liberia and sierra	9
was confirmed by	9
small animal model	9
effector cells were	9
gp or gp	9
infection and vlp	9
case of marburg	9
the spike protein	9
were stained with	9
study was to	9
has recently been	9
innate immune response	9
its ability to	9
addition to the	9
in agreement with	9
the most promising	9
of human infections	9
cells infected with	9
gp of ebola	9
from convalescent patients	9
was replaced by	9
of the envelope	9
a study of	9
with massive lymphocyte	9
the zaire ebov	9
the design of	9
were collected from	9
ebola virus ebola	9
selective estrogen receptor	9
and transmission during	9
van der waals	9
for the identification	9
was performed using	9
were exposed to	9
heptad repeat region	9
that have been	9
an aberrant innate	9
the viral membrane	9
colored fruit bats	9
for ebov and	9
origin and transmission	9
in the past	9
which has been	9
trvlp infection and	9
in accordance with	9
massive lymphocyte apoptosis	9
zaire ebolavirus and	9
the most common	9
the west africa	9
genomic surveillance elucidates	9
were unable to	9
primates against ebola	9
of the same	9
interfering with the	8
ranging from to	8
evidence that dendritic	8
and vp proteins	8
results indicate that	8
pcr in an	8
epidemic and forward	8
stages of infection	8
neutralizing antibodies were	8
as the main	8
the internal fusion	8
gp of the	8
binding free energy	8
been reported for	8
plasma membrane and	8
fever in zaire	8
experimental infection of	8
creative commons attribution	8
that could be	8
emerging viral pathogens	8
half of the	8
mice and guinea	8
increasing amounts of	8
that they are	8
with a case	8
kaletsky et al	8
period of time	8
of three independent	8
of rna viruses	8
change in the	8
in comparison to	8
mechanism by which	8
receptor binding domain	8
required for the	8
a decrease in	8
against infection with	8
to be determined	8
by incubation with	8
hemorrhagic fever with	8
postexposure protection of	8
washed with pbs	8
the human immunodeficiency	8
between filoviruses and	8
response in the	8
gp in the	8
bats as reservoirs	8
similar to those	8
the gps of	8
nematode anticoagulant protein	8
of equine antisera	8
were incubated at	8
is used as	8
by using pseudotyped	8
route of infection	8
human dendritic cells	8
was calculated as	8
specificity of the	8
of dendritic cells	8
fruit bats as	8
examination of the	8
reservoirs of ebola	8
has been detected	8
spread in tetherin	8
infection in rhesus	8
in a dose	8
neutralizing antibody titers	8
gene inhibition assay	8
the incubation period	8
in vivo studies	8
of cell surface	8
the contribution of	8
during the early	8
to be used	8
filoviral hemorrhagic fever	8
and forward projections	8
the sensitivity of	8
side of the	8
and lassa viruses	8
cells are early	8
and in trans	8
been isolated from	8
a critical role	8
discovery to vaccine	8
time to clinical	8
the cathepsin b	8
the creative commons	8
marburg haemorrhagic fever	8
onset of symptoms	8
used to assess	8
sierra leone and	8
a member of	8
anticoagulant protein c	8
of the respective	8
acute respiratory distress	8
to target cells	8
respiratory distress syndrome	8
disease outbreak in	8
and technical committee	8
the ebov in	8
fever virus infection	8
in more detail	8
sequence analysis of	8
passive microfluidic cartridge	8
u and a	8
transported to the	8
a lesser extent	8
cloned into the	8
gp pseudotyped virus	8
for patients with	8
infection by a	8
leading to the	8
as discussed below	8
human asymptomatic ebola	8
us department of	8
with the gp	8
effect on the	8
in cell cultures	8
to compare the	8
that the ebov	8
tetherin antagonism by	8
from discovery to	8
initial viral load	8
cleave ebov gp	8
of less than	8
on vero e	8
of the filoviridae	8
shedding of the	8
for ebov infection	8
murine leukemia virus	8
and activation of	8
huh cells were	8
yet to be	8
with the viral	8
the genomes of	8
it is unclear	8
transmission of ebov	8
postexposure protection against	8
outbreaks of ebola	8
the influenza virus	8
ebolavirus and marburgvirus	8
lethal challenge with	8
the virus was	8
of viral hemorrhagic	8
or dpi with	8
from filovirus disease	8
in the spleen	8
vp and gp	8
the capacity of	8
influenza a h	8
and evaluation of	8
replication in cell	8
of filoviruses in	8
entry of the	8
of marburg and	8
particles produced in	8
protect nonhuman primates	8
in a co	8
host cell cytoplasm	8
of health and	8
sign and l	8
at a final	8
filovirus cell entry	8
the live ebov	8
was performed in	8
in this work	8
the extents of	8
nonhuman primates with	8
cov and ebov	8
taylor et al	8
cells in the	8
african evd outbreak	8
factor viia tissue	8
error bars indicate	8
endosomal cysteine proteases	8
molecular dynamics simulations	8
is not essential	8
of fusion by	8
by ebov gp	8
attributed to the	8
after ebov infection	8
of personal protective	8
sodium dodecyl sulfate	8
stages of the	8
than that of	8
ebov infection of	8
the specificity of	8
have been described	8
virus envelope glycoprotein	8
in cis and	8
humoral and cellular	8
this article is	8
of the gxxxg	8
rapid diagnosis of	8
the viral protein	8
ebola virus during	8
mutation of the	8
mediated by ebov	8
kindly provided by	8
folate receptor alpha	8
functional analysis of	8
fatal and nonfatal	8
reporter gene inhibition	8
in the vlp	8
gp contains a	8
relative to the	8
amino acid sequences	8
avian influenza a	8
as reservoirs of	8
to the development	8
fever ebola virus	8
described in the	8
isolated from the	8
on ebov gp	8
mouse model for	8
rna secondary structures	8
and the cells	8
for zaire ebolavirus	8
the average number	8
log tcid ml	8
are composed of	8
are available from	8
supplemented with fetal	8
by the ebov	8
in the npc	8
have not been	8
cells treated with	8
in the pathogenesis	8
estrogen receptor modulators	8
protection from ebola	8
and dendritic cells	8
recombinant nematode anticoagulant	8
fatal zaire ebola	8
in a previous	8
lethal ebov challenge	8
have been detected	8
in semen of	8
ebov replication in	8
w a mutant	8
humans and nonhuman	8
h n challenge	8
human fatal zaire	8
were inoculated with	8
and assessment of	8
least in part	8
phase i studies	8
with an average	8
in a small	8
referred to as	8
found that a	8
sudan ebola virus	8
at least two	8
by marburg and	8
with bodily fluids	8
is due to	8
clinical signs of	8
found that the	8
in figure a	8
for ebola and	8
cis and in	8
vaccine for ebola	8
in saudi arabia	8
vaccine expressing ebola	8
in zone b	8
to study the	8
congo hemorrhagic fever	8
detection of antibodies	8
is a receptor	8
be associated with	8
imported case of	8
which can be	8
with a lethal	8
by western blot	8
identified as the	8
evd patients in	8
cells were incubated	8
had no effect	8
identified in the	8
the prevention of	8
nucleic acid amplification	8
impact on the	8
an outbreak setting	8
carried out in	8
for gp i	8
at the level	8
assembly of the	8
not be a	8
cytokine and chemokine	8
experimental ebola virus	8
that does not	8
a reduction in	8
ebov in a	8
international scientific and	8
viral fusion protein	8
of ebov during	8
viia tissue factor	8
of toremifene citrate	8
is the first	8
viral data sets	8
of the experiment	8
of the model	8
the filovirus family	8
for the production	8
although it is	8
that in the	8
scientific and technical	8
contact with bodily	8
vaccines have been	8
the introduction of	8
treated with thermolysin	8
gp and furin	8
component of the	8
ifn signaling in	8
mice from lethal	8
in the endoplasmic	8
total number of	8
amino acid changes	8
to calculate the	8
in exporting country	8
of the trailer	8
infection in a	8
residues in the	8
c mice were	8
as a host	8
three independent experiments	8
for virus entry	8
as measured by	8
the endosomal membrane	8
be sufficient to	8
copies of ebov	8
may have a	8
to minimize the	8
in mice and	8
generation of therapeutic	8
dose of mg	8
fusion mediated by	8
to the eu	8
is not required	8
stranded rna viruses	8
form of gp	8
the transcription and	8
a final concentration	8
the human ebov	8
is based on	8
risk of human	8
no effect on	8
no significant difference	8
a mtd of	8
wt and mutant	7
ebov entry into	7
removed from the	7
the nhp model	7
approved drugs for	7
virus circulation in	7
rhesus macaques infected	7
a ctl vaccine	7
outbreak of evd	7
the virion glycoproteins	7
the recovery process	7
as a function	7
congo basin mpxv	7
properties of the	7
were then incubated	7
collected during the	7
virus enters host	7
the acute phase	7
absence of a	7
therapeutic efficacy of	7
multiple cell types	7
as inhibitors of	7
of introduction for	7
of endosomal cathepsins	7
in two reading	7
and lake victoria	7
the secretion of	7
release of viral	7
hafl or vsv	7
human activated protein	7
acquisition of data	7
ebola virus rna	7
plates were incubated	7
of purifying selection	7
epidemic in western	7
the vesicular stomatitis	7
marburg virus vp	7
and at least	7
based vaccine protects	7
the united kingdom	7
brass et al	7
the raw product	7
in a single	7
using pseudotyped viruses	7
and supplementary figure	7
of cleaved gp	7
recombinant inhibitor of	7
were fixed with	7
two of the	7
shown that the	7
during the recovery	7
the past years	7
as has been	7
to counteract the	7
the interaction between	7
and adaptive immune	7
maps and institutional	7
are necessary for	7
of the united	7
cathepsin b inhibitor	7
stability of the	7
a consequence of	7
study in rhesus	7
we thank dr	7
expression of the	7
contact with infected	7
has yet to	7
for the virus	7
essential for replication	7
whether or not	7
isolation of genetically	7
higher than the	7
gene expression in	7
assays were performed	7
the higher ah	7
in the vsv	7
ability of the	7
for filovirus entry	7
macrophages and dendritic	7
was expressed in	7
so as to	7
could be a	7
signs and symptoms	7
to the cells	7
performed in the	7
ebola virus envelope	7
been identified as	7
the centers for	7
expression of vp	7
has been suggested	7
the regulation of	7
interacts with the	7
low ph pulse	7
from egyptian fruit	7
steps of the	7
an analysis of	7
was isolated from	7
virus and marburg	7
after the second	7
from lethal ebov	7
of the drug	7
host cells by	7
resistant to ebov	7
with a single	7
available in the	7
were regarded as	7
of clomiphene and	7
importance of the	7
and vero e	7
the congo basin	7
an increase in	7
to investigate whether	7
are encoded in	7
rhesus macaques with	7
use of a	7
study demonstrated that	7
directional changes in	7
predictor of outcome	7
uyeki et al	7
the current epidemic	7
were conducted in	7
in huh cells	7
and development of	7
was assumed to	7
development of an	7
the gp and	7
of infection and	7
amman et al	7
did not affect	7
outbreak setting and	7
on these data	7
the drug was	7
from a single	7
model of ebola	7
virus disease outbreak	7
as long as	7
at a concentration	7
genetically diverse marburg	7
the first months	7
the assembly of	7
boosted and weeks	7
sign mediate cellular	7
inhibitor of factor	7
compared to wt	7
of fusion is	7
t cells in	7
data on the	7
activated protein c	7
of a new	7
and other filoviruses	7
and used for	7
to test the	7
interferon regulatory factor	7
drafting or revising	7
radoshitzky et al	7
ebov gp by	7
avian influenza viruses	7
type lectins dc	7
in place of	7
of clomiphene citrate	7
important for ebov	7
needs to be	7
virus during the	7
viruses are encoded	7
the same for	7
safe and immunogenic	7
of a human	7
increase in the	7
a small animal	7
claims in published	7
i interferon response	7
using an anti	7
to increase the	7
were performed to	7
on the viral	7
specific cd t	7
origin of the	7
it was also	7
to jurisdictional claims	7
supported by the	7
green fluorescent protein	7
protection in the	7
frames and are	7
in one study	7
of gp is	7
of the mean	7
mpxv infection resulted	7
of hendra virus	7
protection from lethal	7
role in virus	7
infection of macrophages	7
essential for the	7
is safe and	7
used to infect	7
enhancement of ebola	7
primates from filovirus	7
the european union	7
receptor for zaire	7
are responsible for	7
was calculated by	7
of antibodies to	7
groups of mice	7
recent outbreak of	7
critical role in	7
different strategies to	7
cathepsins in entry	7
cells were lysed	7
for analysis of	7
neutralizing monoclonal antibodies	7
diverse marburg viruses	7
catb and catl	7
to address this	7
which may be	7
l and b	7
by using the	7
genome replication and	7
aids reagent program	7
a target for	7
to confirm the	7
replication in vitro	7
analysis of human	7
and functional characterization	7
the ebov trailer	7
such as hiv	7
host range of	7
patients with fatal	7
vp protein of	7
in the formation	7
represent the mean	7
prophylaxis protects nonhuman	7
and the role	7
species of ebola	7
a rapid and	7
prins et al	7
of the study	7
of ebolavirus infection	7
in the respiratory	7
amino acid change	7
the golgi apparatus	7
dronedarone and verapamil	7
an intracellular receptor	7
cd and cd	7
controlled trial of	7
in rhesus macaque	7
can lead to	7
not essential for	7
in the form	7
infected nonhuman primates	7
and influenza a	7
hemorrhagic fever by	7
cells stably expressing	7
ebolavirus and lake	7
and cells were	7
is difficult to	7
have been associated	7
ebola virus vaccines	7
t cell activation	7
an animal model	7
was performed to	7
and subjected to	7
lower respiratory tract	7
lead to the	7
ebola viruses are	7
monoclonal antibodies against	7
the early stages	7
encoded in two	7
viral transcription and	7
some of these	7
or revising the	7
of influenza virus	7
the protective efficacy	7
seems to be	7
assessment of patient	7
available from the	7
there is also	7
a highly conserved	7
in influenza virus	7
in scfv format	7
revealed that the	7
load as a	7
only a few	7
region of ebov	7
required for viral	7
for h in	7
a negative control	7
gxxxg motif in	7
prevalence in bats	7
two reading frames	7
for public health	7
by plaque assay	7
of ebov fusion	7
towner et al	7
cynomolgus macaques with	7
in infectious disease	7
by confocal microscopy	7
liver and spleen	7
hartman et al	7
viruses from egyptian	7
by the gp	7
roles in the	7
a mutation in	7
the ebov vp	7
fever by reverse	7
plasmids encoding the	7
treated with remdesivir	7
envelope glycoprotein gp	7
on day and	7
severe ebola virus	7
were subjected to	7
the virus in	7
a novel coronavirus	7
cathepsin l and	7
neutral with regard	7
molecular dynamics simulation	7
the african straw	7
exposure to the	7
a sucrose cushion	7
ebov outbreak in	7
may play a	7
version of this	7
np and vp	7
with monoclonal antibodies	7
human ebola virus	7
activity of a	7
of approved drugs	7
be related to	7
experimental inoculation of	7
during ebov infection	7
were reported in	7
the public health	7
postexposure antibody prophylaxis	7
the utility of	7
for the selection	7
collected from the	7
mediate cellular entry	7
in entry mediated	7
h n ha	7
for the determination	7
and the gp	7
outbreak of marburg	7
at neutral ph	7
found to have	7
acute phase of	7
humans infected with	7
the same time	7
immunity to ad	7
shown in table	7
was performed by	7
and analyzed by	7
to be an	7
of viral infection	7
common features pharmacophore	7
the management of	7
leung et al	7
virus in cis	7
been implicated in	7
identification of receptor	7
able to inhibit	7
expressing ebov gp	7
the sierra leone	7
are expressed through	7
expression on dcs	7
in host cells	7
with plasmids encoding	7
has not yet	7
be required to	7
ebov gp in	7
in published maps	7
figure a and	7
and supplementary fig	7
owing to the	7
development of new	7
west african countries	7
a luciferase reporter	7
viruses ebola virus	7
treatment was given	7
a host cell	7
short stretches of	7
to generate a	7
endosomal cathepsins in	7
and institutional affiliations	7
avian a subtypes	7
exposure treatment of	7
the evd patients	7
the patients were	7
as a predictor	7
recombinant human activated	7
genbank accession no	7
are susceptible to	7
was incubated with	7
our proposed epitope	7
given in table	7
and tumor necrosis	7
these results demonstrate	7
of the peptide	7
setting and assessment	7
existing immunity to	7
a guinea pig	7
ability to inhibit	7
activities in cell	7
results demonstrate that	7
it is of	7
secondary lymphoid tissues	7
with marburg virus	7
nature remains neutral	7
targets of the	7
virus prevalence in	7
the virus from	7
infection in mice	7
institute of infectious	7
the detection limit	7
in africa and	7
over the past	7
chou and fasman	7
were used as	7
nns rna viruses	7
been used for	7
neutralizing antibody response	7
influenza h n	7
information regarding the	7
virus disease ebola	7
prediction of the	7
to a single	7
fecal samples from	7
the ebov e	7
viral entry into	7
baize et al	7
springer nature remains	7
performed using the	7
virus entry into	7
into target cells	7
should be addressed	7
macaque model of	7
cell signaling pathways	7
in a similar	7
to show the	7
not lead to	7
in protection against	7
of the infected	7
the us department	7
the target cell	7
national institute of	7
disease in nonhuman	7
in the model	7
human influenza a	7
three viral data	7
antibody titers were	7
in cell lysates	7
published maps and	7
that has been	7
were analyzed for	7
of macrophages and	7
the glycoproteins of	7
as a surrogate	7
entry by using	7
deficient cell lines	7
use of the	7
infectious disease epidemiology	7
infection of the	7
viral hemorrhagic fevers	7
infection in an	7
nonhuman primates from	7
of immune responses	7
used as an	7
cells were grown	7
public health response	7
is an enveloped	7
of filovirus disease	7
of patient viral	7
were grown in	7
cell culture supernatant	7
detection of viral	7
of the viruses	7
virus persistence in	7
and immunogenic in	7
viral load on	7
role of endosomal	7
infection results in	7
remains neutral with	7
mediated by a	7
late endosomes and	7
acts as a	7
infected with a	7
the lipid bilayer	7
patient viral load	7
ministry of health	7
marburg viruses from	7
human cases of	7
host endosomal membranes	7
virus and ebola	7
a lethal ebola	7
was performed as	7
this is an	7
ebov neutralisation assay	7
african green monkeys	7
antibody prophylaxis protects	7
the form of	7
infected cells were	7
responses in ebola	7
the first immunization	7
ebola virus enters	7
mice vaccinated with	7
of h n	7
neutralizing antibodies against	7
that they have	7
revising the article	7
used to generate	7
the viral nucleoprotein	7
enters host cells	7
inhibition of ebola	7
in dendritic cells	7
of branch lengths	7
the study of	7
of gp to	7
culture supernatants were	7
human a subtypes	7
with the highest	7
strategies to counteract	7
domain of the	7
indicate that the	7
of the west	7
of the influenza	7
reading frames and	7
could be due	7
regard to jurisdictional	7
animals infected with	7
ebola virus proteins	7
of t cells	7
need to be	7
a function of	7
to counteract tetherin	7
vesicular stomatitis viruses	7
from the ncbi	7
plasma concentrations of	7
from the surface	7
aspects of the	7
plasma levels of	7
has resulted in	7
for the viral	7
to investigate the	7
in association with	7
viral load in	7
a surrogate for	7
cell activation and	7
humans and animals	7
structural basis for	7
a role for	7
of ebov vp	7
viral load as	7
base of the	7
clinical and laboratory	7
the viral load	7
expressed through transcriptional	7
of small molecules	7
from the ebov	7
of genetically diverse	7
as an ebola	7
are resistant to	7
using the same	7
natural reservoir hosts	7
amino acid sequence	7
data from the	7
of marv gp	7
is caused by	7
inhibitors of ebola	7
to obtain the	7
of ebov is	7
primary human monocytes	7
on day postexposure	7
attenuated recombinant vaccine	7
animals in the	7
to nonhuman primates	7
and are expressed	7
hsnpc domain c	7
determined by plaque	7
rna in the	7
increased vascular permeability	7
analyses showed that	7
a predictor of	7
were collected at	7
and yellow fever	7
jurisdictional claims in	7
gp is a	7
with blood transfusions	7
of the total	7
results in a	7
dependent protein kinase	6
antibody response against	6
absence of any	6
mediated immune responses	6
protective immunity against	6
for transcription and	6
estimated to be	6
is inhibited by	6
to the virus	6
fact that the	6
number of human	6
of the data	6
to be important	6
blood samples were	6
functions as a	6
the crystal structure	6
ebola virus makona	6
direct effect of	6
of convalescent plasma	6
characterization of host	6
at a dilution	6
the ebov and	6
gupta et al	6
clinically approved drugs	6
were mixed with	6
the online version	6
dependent inhibition of	6
spread of the	6
there are a	6
the active metabolite	6
glycoproteins and the	6
for determination of	6
sierra leone ebola	6
shown in the	6
wild great apes	6
against ebolavirus challenge	6
filovirus infection and	6
interaction with the	6
strong inflammatory response	6
evaluation of a	6
the availability of	6
editing of the	6
to viral spread	6
virus challenge in	6
infections in humans	6
for antibodies to	6
cell entry by	6
risk of ebola	6
ebola virus by	6
management of ebola	6
determined to be	6
k akt mtor	6
average weight loss	6
monoclonal antibodies in	6
guinea pigs were	6
infection prevention and	6
adjuvanted ebov vlps	6
human b type	6
ebola virus polymerase	6
isomers of clomiphene	6
of animal origin	6
not statistically significant	6
of endothelial cells	6
cov and sars	6
immune response in	6
macaques with monoclonal	6
the nucleocapsid proteins	6
of cell signaling	6
detected in a	6
the kinetics of	6
based ebola vaccine	6
virus polymerase and	6
broad antiviral activity	6
analyses were performed	6
infection and strong	6
the case for	6
common among the	6
taxonomy of viruses	6
for the two	6
into cell lines	6
were collected in	6
protection against the	6
t cells and	6
transfusions from convalescent	6
modulators inhibit ebola	6
the eye and	6
in hong kong	6
humoral and cell	6
monotherapy against lethal	6
protects against lethal	6
in different cell	6
potently neutralizing antibody	6
and marv are	6
bovine serum albumin	6
where it is	6
bind to the	6
authors declare that	6
to endo h	6
the national institute	6
cell responses are	6
the cell layer	6
and its isomers	6
to eu mss	6
of marv vp	6
and hepatitis c	6
has been associated	6
human embryonic kidney	6
each well and	6
host cells via	6
juvenile rousettus aegyptiacus	6
known to be	6
it can be	6
is antagonized by	6
produced in natural	6
experiments were performed	6
the appearance of	6
of the membrane	6
stage of the	6
by a potently	6
and weeks later	6
may be possible	6
is no evidence	6
due to its	6
we performed a	6
of factor viia	6
and zika virus	6
that clomiphene blocks	6
glycoproteins of marburg	6
susceptible to infection	6
macropinocytosis in a	6
is used to	6
are directed against	6
legal trade of	6
relative importance of	6
of the assay	6
ri rna synthesis	6
we focused on	6
also have a	6
in natural human	6
endothelial cells are	6
modifications of the	6
dynamics of the	6
type c disease	6
is available for	6
and in a	6
the results from	6
protein data bank	6
no conflicts of	6
the t cell	6
was then used	6
directly from the	6
as described for	6
the evd outbreak	6
or marv gp	6
persistence of phi	6
on the day	6
assessed by the	6
and pathogenesis of	6
for prediction of	6
viruses into the	6
survival in rhesus	6
of the c	6
cells were transduced	6
viruses can be	6
compared with the	6
as there is	6
t and vaccinia	6
are given in	6
the plate was	6
binding to ebola	6
dendritic cells in	6
endogenous viral elements	6
and a high	6
therapeutic antisera for	6
be needed to	6
replication cycle of	6
and verapamil inhibit	6
of hemorrhagic fever	6
and penicillin streptomycin	6
to develop a	6
the susceptibility of	6
have previously been	6
antiviral activity in	6
and cellular immunity	6
in humans infected	6
periods of increased	6
likely that the	6
membrane fusion and	6
assays have been	6
its interaction with	6
viral rna and	6
of the predicted	6
virus is edited	6
positive selection in	6
be attributed to	6
associated with ebov	6
of ebov was	6