trigram

This is a table of type trigram and their frequencies. Use it to search & browse the list to learn more about your study carrel.

trigram	frequency
and respiratory syndrome	2582
reproductive and respiratory	2500
porcine reproductive and	2492
respiratory syndrome virus	2133
of porcine reproductive	920
the presence of	314
in this study	235
the cells were	212
the expression of	207
as well as	189
infected with prrsv	184
the porcine reproductive	152
as shown in	151
in the lungs	144
according to the	140
syndrome virus infection	138
syndrome virus in	137
in the presence	137
of the virus	136
data not shown	135
to porcine reproductive	133
the development of	132
the number of	131
for h at	130
pigs infected with	129
shown in fig	125
with porcine reproductive	125
the role of	122
equine arteritis virus	121
were infected with	118
the present study	114
was used to	107
the replication of	105
of the porcine	104
porcine alveolar macrophages	102
been shown to	101
due to the	100
the absence of	100
the effect of	98
based on the	97
one of the	95
the immune response	95
of the prrsv	90
of prrsv infection	90
porcine circovirus type	90
the production of	90
immune response to	88
type i ifn	87
analysis of the	87
involved in the	87
has been shown	85
as described previously	83
pathogenic porcine reproductive	83
highly pathogenic porcine	82
of prrsv in	82
in order to	82
in porcine reproductive	81
for min at	80
the end of	79
the use of	78
it has been	77
of prrsv and	77
the united states	74
were transfected with	74
cells were infected	73
infected with porcine	73
meulenberg et al	72
of the viral	72
to determine the	71
an important role	71
response to prrsv	71
innate immune response	70
showed that the	70
in the present	70
was observed in	69
role in the	69
a and b	69
were treated with	69
cells were washed	69
in the lung	69
found to be	68
prrsv infection in	67
end of the	67
nsp and nsp	67
influenza a virus	67
respiratory syndrome viruses	66
in the absence	66
cd t cells	66
replication of prrsv	66
swine infertility and	65
was used as	65
type i interferon	65
infertility and respiratory	65
the formation of	64
the immune system	64
the detection of	64
the level of	64
was detected in	64
porcine epidemic diarrhea	63
in the united	63
syndrome virus and	62
member of the	62
immune responses to	62
shown to be	62
an moi of	62
virus infection in	62
at room temperature	62
as previously described	62
in response to	61
prrsv and pedv	61
the n protein	61
the induction of	61
that of the	61
of lelystad virus	61
the lungs of	61
innate and adaptive	60
gp and m	60
antiviral activity of	60
rna was extracted	60
to that of	60
important role in	60
porcine respiratory coronavirus	59
were used to	58
used as a	58
epidemic diarrhea virus	58
at an moi	58
replication of porcine	58
of type i	58
used in this	58
during prrsv infection	58
results showed that	58
strains of porcine	58
the prrsv genome	57
to prrsv infection	57
the ability of	57
transmissible gastroenteritis virus	57
as described above	56
of viral rna	56
in vitro and	56
strains of prrsv	56
severe acute respiratory	56
acute respiratory syndrome	55
we found that	55
prrsv n protein	55
against porcine reproductive	55
observed in the	55
a member of	54
is essential for	54
infection with prrsv	54
in pigs infected	54
the swine industry	54
cells were transfected	54
of the disease	53
compared to the	53
infection in pigs	53
comparison of the	53
syndrome virus by	53
total rna was	53
of swine infertility	53
nelsen et al	53
incubated for h	53
at h post	53
was added to	52
adaptive immune response	52
cells were then	52
well plates were	52
on the other	52
cells transfected with	52
gp and gp	51
was performed using	51
compared with the	51
allende et al	51
detected in the	51
the current study	51
the activation of	51
open reading frames	50
swine influenza virus	50
virus replication in	50
of prrsv nsp	50
immune responses in	50
innate immune responses	50
in a dose	50
the addition of	50
the north american	50
by porcine reproductive	49
the innate immune	49
was used for	49
in the cytoplasm	49
for porcine reproductive	49
in cell culture	49
assessment of the	49
of the immune	48
in addition to	48
expression of the	48
the efficacy of	48
of the most	48
the viral genome	48
isolate atcc vr	48
hepatitis c virus	48
to investigate the	48
of virus replication	47
of the arterivirus	47
the lack of	47
the e protein	47
were detected in	47
the other hand	47
were inoculated with	47
of pigs with	47
prrsv replication in	47
cells infected with	46
cells in the	46
indicated that the	46
in the infected	46
of n protein	46
the percentage of	46
and m proteins	45
a concentration of	45
for the detection	45
was found to	45
such as the	45
were incubated with	44
associated with the	44
in gnotobiotic pigs	44
adaptive immune responses	44
of neutralizing antibodies	44
the family arteriviridae	44
at a concentration	44
infected cells were	44
characterization of the	44
the infected pigs	44
and stored at	43
cells were incubated	43
the amount of	43
have shown that	42
strain of porcine	42
protein of porcine	42
vero cells were	42
with prrsv and	42
virus nucleocapsid protein	42
was determined by	42
the interaction between	42
have been identified	41
yuan et al	41
simian hemorrhagic fever	41
cell lysates were	41
structural proteins of	41
the frequency of	41
cells were treated	41
has been reported	41
type porcine reproductive	41
was carried out	41
mystery swine disease	41
respiratory syndrome coronavirus	40
western blot analysis	40
hemorrhagic fever virus	40
syndrome virus replication	40
is associated with	40
of the genome	40
to be a	40
a total of	40
the antiviral activity	40
detection of porcine	40
fetal bovine serum	40
pi k akt	40
with prrsv at	40
of the host	40
economic impact of	40
shi et al	40
in prrsv infection	40
of prrsv replication	40
the levels of	40
at h p	40
was extracted from	40
murtaugh et al	39
disease in the	39
related to the	39
a role in	39
the inhibition of	39
have been reported	39
understanding of the	39
host immune response	39
washed three times	39
shown in figure	39
responsible for the	39
of proinflammatory cytokines	39
li et al	39
humoral immune response	39
in the current	38
with that of	38
for h and	38
cells were harvested	38
syndrome virus isolates	38
demonstrated that the	38
immune response in	38
of porcine circovirus	38
is one of	38
consistent with the	38
the economic impact	38
expression of il	38
classical swine fever	38
belongs to the	38
results indicated that	38
is the most	38
the dub activity	38
there was no	38
the causative agent	38
the concentration of	38
open reading frame	37
effect on the	37
and adaptive immune	37
isolates of porcine	37
the host immune	37
development of a	37
were incubated for	37
infection of pigs	37
has not been	37
increase in the	37
is required for	37
for prrsv infection	36
infectious cdna clone	36
highly pathogenic prrsv	36
response to porcine	36
three times with	36
and in vivo	36
were observed in	36
the course of	36
derived from the	36
c for min	36
by western blot	36
the results of	36
virus in a	36
this study was	36
the order nidovirales	36
kim et al	36
and vero cells	36
studies have shown	36
at the time	35
of the economic	35
prrsv infection and	35
in the pig	35
the nucleocapsid protein	35
when compared to	35
used in the	35
a number of	35
of infected cells	35
of the orf	35
the mechanism of	35
exposed to prrsv	35
syndrome virus nucleocapsid	35
yoon et al	35
that prrsv nsp	35
virus porcine reproductive	35
the severity of	35
the emergence of	35
were analyzed by	35
play an important	35
on prrsv replication	35
the time of	35
g for min	35
of the two	35
the results showed	35
of the n	35
to determine whether	34
to assess the	34
most of the	34
revealed that the	34
the pi k	34
impact of porcine	34
expression levels of	34
multiplicity of infection	34
viral rna synthesis	34
the case of	34
were stained with	34
respiratory disease complex	34
expressed as the	34
derived dendritic cells	34
infected with the	34
is involved in	34
were found to	34
type i ifns	34
and incubated for	34
and simian hemorrhagic	33
structure of the	33
the isolation of	33
the control group	33
added to the	33
was supported by	33
of equine arteritis	33
found in the	33
abortion and respiratory	33
high levels of	33
orf a and	33
found that the	33
host innate immune	33
a combination of	33
presence of prrsv	33
cells were cultured	33
north american type	33
virus in the	33
wang et al	33
snijder et al	33
nelson et al	33
lungs of pigs	33
syndrome virus gp	33
in the serum	33
activation of the	33
dependent rna polymerase	33
indicating that the	33
a variety of	33
meng et al	33
cells were fixed	33
in our study	33
wensvoort et al	32
and porcine reproductive	32
of porcine epidemic	32
for detection of	32
syndrome virus the	32
of the gp	32
american and european	32
chen et al	32
in china and	32
porcine epidemic abortion	32
the respiratory tract	32
pigs exposed to	32
immune response against	32
pcd and vero	32
changes in the	32
peripheral blood mononuclear	32
experimental reproduction of	32
previous studies have	32
in the case	32
tian et al	32
blood mononuclear cells	32
the importance of	32
transfected with the	32
used as the	32
protective immune response	32
epidemic abortion and	32
reproductive failure in	32
swine fever virus	32
h n influenza	32
and in the	32
the majority of	31
prrsv is a	31
activity of the	31
in the same	31
contribute to the	31
porcine respiratory disease	31
the ability to	31
were subjected to	31
mengeling et al	31
the fact that	31
the adaptive immune	31
the effects of	31
of prrsv is	31
located in the	31
proteins of porcine	31
that the prrsv	31
in north america	31
human immunodeficiency virus	31
cells were maintained	31
zhang et al	31
have been shown	31
expression of ifn	31
in the netherlands	31
all of the	31
results suggest that	31
are involved in	31
of a novel	31
as a result	31
stadejek et al	31
immune response and	31
in the viral	30
north american and	30
it is possible	30
p and p	30
the infected cells	30
unfolded protein response	30
at dpi and	30
role of the	30
european porcine reproductive	30
virus nonstructural protein	30
in contrast to	30
plp dub activity	30
vitro and in	30
albina et al	30
indicate that the	30
prrsv strain jxwn	30
respiratory disease in	30
innate immunity and	30
of this study	30
at the same	30
of immune responses	30
some of the	30
proteins encoded by	30
or absence of	29
a highly pathogenic	29
pigs vaccinated with	29
the most important	29
is characterized by	29
samples were collected	29
be due to	29
presence or absence	29
at the indicated	29
immune responses and	29
that prrsv infection	29
causative agent of	29
for min and	29
genes involved in	29
the ratio of	29
pigs inoculated with	29
prrsv infection of	29
of the pigs	29
disease in pigs	29
to evaluate the	29
inhibitory effect on	29
in agreement with	29
of the infected	29
for the presence	29
and identification of	29
regulatory t cells	29
of pigs infected	29
and in vitro	29
because of the	29
of the nsp	29
of the study	29
be used to	29
the prrsv n	29
supernatants were collected	29
in the respiratory	29
in which the	29
the endoplasmic reticulum	29
syndrome virus vaccine	29
higher levels of	29
region of the	29
were maintained in	29
genbank accession no	28
characterization of swine	28
subgenomic mrna synthesis	28
analysis was performed	28
the synthesis of	28
similar to that	28
of north american	28
none of the	28
demonstrated that prrsv	28
proteins in the	28
were used as	28
the authors declare	28
were used for	28
appears to be	28
prrsv nsp and	28
viral replication and	28
a and orf	28
were expressed as	28
production in the	28
wills et al	28
there is a	28
against prrsv infection	28
of highly pathogenic	28
cells of the	28
inhibition of prrsv	28
is important for	28
halbur et al	28
approved by the	28
all pigs were	28
isolates of prrsv	28
and orf b	28
the orf gene	28
present in the	28
was confirmed by	28
was associated with	28
replication and transcription	28
and activation of	28
data indicate that	28
suggesting that the	28
syndrome virus strain	28
cells treated with	28
crystal structure of	28
the gp protein	27
d kv adj	27
genetic diversity of	27
did not affect	27
for the development	27
were grown in	27
in the porcine	27
ability to induce	27
at days post	27
of prrsv rna	27
respiratory and reproductive	27
cells were grown	27
essential for virus	27
but not in	27
are shown in	27
live porcine reproductive	27
development of the	27
is consistent with	27
to detect the	27
results indicate that	27
swine disease in	27
t cells in	27
in the development	27
syndrome virus nonstructural	27
immune response of	27
were obtained from	27
hek t cells	27
protein in the	27
the identification of	27
in porcine alveolar	27
swine industry worldwide	27
three independent experiments	27
and sg mrna	27
hepatitis b virus	27
strain of prrsv	27
suggest that the	27
were collected and	27
than that of	27
c kv adj	27
suggests that the	27
was isolated from	26
were determined by	26
t cells and	26
prrsv infection is	26
were purchased from	26
washed twice with	26
antiviral activity against	26
at the end	26
the sdd motif	26
production of proinflammatory	26
experiments were performed	26
of the cell	26
cd and cd	26
with the indicated	26
between the two	26
the lungs and	26
was significantly higher	26
genomic rna and	26
can be used	26
be involved in	26
remains to be	26
opriessnig et al	26
characterization of a	26
of the same	26
in terms of	26
isolation of lelystad	26
in the field	26
is mediated by	26
an increase in	26
well as the	26
the context of	26
the virus to	26
k akt pathway	26
strand rna viruses	26
in infected pigs	26
reeth et al	26
the presence or	26
i ifn signaling	26
prrsv in the	26
expression of prrsv	26
in the thymus	26
neutralizing antibody response	26
play a role	26
inoculated with prrsv	26
is a member	25
was replaced with	25
fang et al	25
b and c	25
type i interferons	25
wootton et al	25
were performed using	25
the plasma membrane	25
to determine if	25
for virus replication	25
was performed by	25
is critical for	25
is related to	25
and characterization of	25
and european isolates	25
after challenge with	25
in vivo and	25
part of the	25
min at room	25
a key role	25
to those of	25
used for the	25
the impact of	25
vesicular stomatitis virus	25
the regulation of	25
with fetal bovine	25
the control of	25
the structure of	25
rossow et al	25
north american prrsv	25
viral rna in	25
obtained from the	25
the release of	25
h n p	25
specificity of the	25
and porcine circovirus	25
belonging to the	25
compared to pigs	25
after prrsv infection	25
is an important	25
the generation of	25
viral loads in	25
and that the	25
cells inoculated with	25
similar to the	25
virus and porcine	25
were collected at	25
n protein was	24
in both the	24
due to a	24
respiratory syndrome in	24
was shown to	24
regions of the	24
supported by the	24
den boon et	24
the result of	24
and incubated with	24
respiratory syncytial virus	24
prrsv and the	24
the study of	24
were washed with	24
their ability to	24
to test the	24
between prrsv and	24
were positive for	24
care and use	24
t cells were	24
been reported to	24
van reeth et	24
of swine to	24
neutralizing antibodies in	24
syndrome virus porcine	24
at h after	24
the amino acid	24
there is no	24
by interfering with	24
the prrs virus	24
in situ hybridization	24
persistently infected with	24
by centrifugation at	24
n influenza virus	24
herpes simplex virus	24
the structural proteins	24
the interaction of	24
zhou et al	24
cells grown in	24
the in vitro	24
there was a	24
referred to as	24
of infected pigs	24
in the prrsv	24
the same time	24
early stage of	24
to be the	24
pathogenesis of porcine	24
porcine nidovirus infection	24
is known to	24
the pathogenicity of	24
used to detect	24
activity of prrsv	24
boon et al	24
associated with a	24
was performed as	24
a previous study	24
immune response by	24
intranasal delivery of	24
of the family	23
also known as	23
could not be	23
it is not	23
porcine respiratory and	23
responses to porcine	23
depending on the	23
stranded rna virus	23
was performed in	23
in the early	23
and lymph nodes	23
a result of	23
response in pigs	23
the genome of	23
suggested that the	23
gp of porcine	23
rowland et al	23
have also been	23
as early as	23
pcr products were	23
hepatitis e virus	23
nested set of	23
cells and the	23
plays an important	23
at and dpi	23
were analyzed using	23
for prrsv replication	23
the clearance of	23
a modified live	23
kindly provided by	23
molecular biology of	23
syndrome virus on	23
is dependent on	23
virus replication and	23
in the swine	23
properties of the	23
is caused by	23
t cell responses	23
in serum and	23
in the host	23
inoculated with the	23
encoded by orfs	23
similar to those	23
the serum of	23
the unfolded protein	23
antibodies to the	23
clinical signs and	23
the function of	22
cloned into the	22
mouth disease virus	22
us porcine reproductive	22
i and ii	22
resulted in a	22
sequences of the	22
amino acid residues	22
and adaptive immunity	22
isolate of porcine	22
has been demonstrated	22
van dinten et	22
infected pigs at	22
pigs in the	22
chinese highly pathogenic	22
were washed twice	22
is able to	22
washed with pbs	22
the virus was	22
assay was performed	22
of atypical prrs	22
the degradation of	22
of the north	22
component of the	22
of severe acute	22
dinten et al	22
a series of	22
supplemented with fbs	22
to the virus	22
plasmacytoid dendritic cells	22
conjugated goat anti	22
enhanced replication of	22
to the family	22
virus characterization of	22
mediated immune responses	22
and the cells	22
were negative for	22
han et al	22
in the context	22
members of the	22
infectious bronchitis virus	22
their susceptibility to	22
prrsv replication was	22
bautista et al	22
processing of the	22
outbreaks of atypical	22
thermo fisher scientific	22
and north american	22
induced by the	22
subpopulation of ma	22
the cell surface	22
nucleocapsid protein of	22
lee et al	22
pcr was performed	22
is possible that	22
to the nucleolus	22
were washed three	21
prrsv and prrsv	21
linked immunosorbent assay	21
was performed with	21
lower than that	21
mutations in the	21
well tissue culture	21
performed using the	21
the molecular biology	21
antibody response to	21
alveolar macrophages and	21
the culture medium	21
cells were lysed	21
on swine production	21
and reproductive syndrome	21
by prrsv infection	21
syndrome on swine	21
following infection with	21
has also been	21
induced by prrsv	21
animal care and	21
a critical role	21
were harvested and	21
swine production in	21
in figure a	21
bovine serum albumin	21
a set of	21
been shown that	21
each of the	21
in combination with	21
response to the	21
or infected with	21
of prrsv was	21
unparalleled outbreaks of	21
of the protein	21
on the cell	21
the plates were	21
the degree of	21
performed according to	21
response in the	21
replication in the	21
stranded rna viruses	21
snijder and meulenberg	21
culture supernatants were	21
the site of	21
identification of a	21
lymphoid tissues of	21
orf and orf	21
with ml of	21
was analyzed by	21
of the unique	21
the inhibitory effect	21
with respect to	21
differences in the	21
correlated with the	21
infection with porcine	21
summarized in table	21
at different time	21
clinical signs of	21
syndrome virus comparison	21
the supernatants were	21
the viral rna	21
respiratory syndrome on	21
show that the	21
the supernatant was	21
in the first	21
serum samples were	21
atypical prrs in	21
the analysis of	21
a negative control	21
the atpase activity	21
any of the	21
in a humidified	21
balb c mice	20
key role in	20
and gp proteins	20
dissection of the	20
prrs in china	20
emergence of fatal	20
after infection with	20
syndrome virus strains	20
as determined by	20
genome replication and	20
dub activity of	20
the control pigs	20
the accumulation of	20
north american strains	20
the pigs were	20
the kinetics of	20
syndrome virus nsp	20
type and type	20
morbidity and mortality	20
of maternal antibodies	20
on two continents	20
in lymphoid tissues	20
experimentally infected with	20
the purpose of	20
is the first	20
this study were	20
in accordance with	20
in primary pams	20
contributes to the	20
resulting in the	20
hennings et al	20
homogeneous subpopulation of	20
formation of autophagosomes	20
may be a	20
fatal prrsv variants	20
mhc class i	20
is responsible for	20
and nuclear translocation	20
was purchased from	20
mardassi et al	20
were isolated from	20
to study the	20
of influenza a	20
sequence analysis of	20
prrsv challenged pigs	20
with prrsv strain	20
north america and	20
of fatal prrsv	20
h after the	20
c for s	20
interferon regulatory factor	20
cysteine protease domain	20
in the nsp	20
protection against prrsv	20
in a homogeneous	20
mediated immune response	20
those of the	20
of the nucleocapsid	20
the involvement of	20
as a negative	20
in the number	20
response to infection	20
polymerase chain reaction	20
to investigate whether	20
the humoral immune	20
the unique hallmark	20
tissue culture infectious	20
rpm for min	20
verheije et al	20
the disease in	20
used as an	20
due to its	20
cytokines in the	20
sun et al	20
neutralizing antibodies and	20
considered statistically significant	20
were measured by	20
viruses such as	20
pigs persistently infected	20
the appearance of	20
added to each	20
shown that the	20
prrsv has been	20
a homogeneous subpopulation	20
of the first	20
in the nucleus	20
molecular dissection of	20
were performed in	20
a significant increase	20
viral rna was	20
shown in table	19
of prrsv helicase	19
the virus in	19
presence of the	19
detection of prrsv	19
emergence of a	19
pigs with porcine	19
china and molecular	19
of the lung	19
reported that the	19
the pathogenesis of	19
showed that prrsv	19
inhibitory effect of	19
encoded by orf	19
cd t cell	19
immunity to porcine	19
portion of the	19
in pregnant sows	19
the occurrence of	19
identification of the	19
not required for	19
h at room	19
n protein is	19
the most economically	19
the lipid rafts	19
prrsv strain vr	19
the infection of	19
rna and sg	19
molecular characterization of	19
infection in the	19
in the cell	19
and western blot	19
cellular immune responses	19
protein of prrsv	19
led to the	19
supplemented with fetal	19
a nested set	19
did not have	19
this work was	19
better understanding of	19
the entry of	19
european and american	19
a standard curve	19
and mycoplasma hyopneumoniae	19
no significant differences	19
the type i	19
our understanding of	19
host response to	19
agent of porcine	19
reproduction of the	19
in the order	19
may play a	19
those in the	19
data suggest that	19
an infectious clone	19
are expressed as	19
increased expression of	19
by the addition	19
in another study	19
proteolytic processing of	19
significantly lower in	19
of prrsv on	19
mucosal and systemic	19
n protein and	19
as compared to	19
of cellular cholesterol	19
and molecular dissection	19
the loss of	19
then infected with	19
in pam cultures	19
was performed to	19
the mrna level	19
meier et al	19
stage of infection	19
the viral envelope	19
at different times	19
were significantly lower	19
pam cultures were	19
by the use	19
sequencing of the	19
in the us	19
ldv and eav	19
plasma membrane cholesterol	19
african swine fever	19
and evolution of	19
with regard to	19
of the vaccine	19
study showed that	19
of the lelystad	19
s p ratios	19
is a major	19
are summarized in	19
responses to prrsv	19
and experimental reproduction	19
was used in	19
was measured by	19
authors declare that	19
protective immunity against	19
a multiplicity of	19
the surface of	19
was performed on	19
are essential for	19
the expression levels	19
of infection and	19
on the surface	18
cells were inoculated	18
amino acid sequence	18
included in the	18
in pigs exposed	18
viral life cycle	18
the activity of	18
control of the	18
replication in marc	18
c and d	18
and of the	18
could be used	18
proteins of the	18
significant economic losses	18
in the virus	18
the arterivirus replicase	18
order comprising coronaviridae	18
analysis of porcine	18
by using the	18
antigen presenting cells	18
were cultured in	18
transcript abundance of	18
is independent of	18
the lungs were	18
than those in	18
dea et al	18
levels of prrsv	18
new order comprising	18
in neonatal pigs	18
activation of nf	18
and then infected	18
passive transfer of	18
of prrsv to	18
structural protein of	18
nuclear translocation of	18
syndrome virus vaccines	18
and pcv b	18
divergent evolution on	18
to examine the	18
nature of the	18
with prrsv or	18
were detected by	18
the university of	18
the immunogenicity of	18
with pbs and	18
economic losses to	18
prrsv gp protein	18
in infected cells	18
h at c	18
expression of nsp	18
h and then	18
b cell activation	18
wissink et al	18
the inflammatory response	18
responses in pigs	18
experimental infection of	18
of immune cells	18
immune responses against	18
in cells infected	18
not detected in	18
syndrome virus date	18
the frequencies of	18
levels of ifn	18
the crystal structure	18
the nucleus and	18
the nonstructural protein	18
differential production of	18
major histocompatibility complex	18
in the control	18
followed by a	18
activity against prrsv	18
prior to infection	18
the possibility that	18
activity of nsp	18
was due to	18
prrsv e protein	18
of porcine alveolar	18
did not show	18
transferred to a	18
laguna et al	18
partial protection against	18
in the study	18
in sows and	18
study was to	18
of the ifn	18
cells cultured in	18
pigs porcine reproductive	18
for h with	18
african green monkey	18
failure in sows	18
pasternak et al	18
to identify the	18
swine to porcine	18
the viral loads	18
humoral and cellular	18
of viral replication	18
was found in	18
virus in pigs	18
lead to the	18
mucosal immune system	18
little is known	18
is an enveloped	18
c in a	18
of a live	18
of amino acids	18
comprising coronaviridae and	18
infectious clone of	18
infection of marc	18
analyses were performed	18
a new order	18
attributed to the	18
as indicated by	18
prrsv replication and	18
second lps administration	18
listed in table	18
the m protein	17
innate immune system	17
in the future	17
critical role in	17
to the swine	17
to explore the	17
as a consequence	17
thanawongnuwech et al	17
were seeded in	17
with the immune	17
are needed to	17
the evolution of	17
orfs to of	17
cells were seeded	17
at the c	17
prrsv at an	17
positive for prrsv	17
data indicated that	17
the negative control	17
strand rna virus	17
liu et al	17
evolution on two	17
a consequence of	17
at a multiplicity	17
of a virus	17
of proteins encoded	17
of viral proteins	17
and incubated at	17
immune responses of	17
antibody response in	17
downstream of the	17
with prrsv alone	17
terminal region of	17
mrna levels of	17
susceptibility to porcine	17
of the animals	17
response to swine	17
response against prrsv	17
no effect on	17
the host response	17
inhibition of the	17
the highly pathogenic	17
isolation of swine	17
effect of the	17
homologous and heterologous	17
pigs compared to	17
n protein in	17
positive cells were	17
of pigs to	17
be detected in	17
were challenged with	17
responses in the	17
the prrsv e	17
effect of prrsv	17
the endoribonuclease activity	17
expressing prrsv antigens	17
a review of	17
america and experimental	17
dendritic cells and	17
to infection with	17
expression of cd	17
determined by the	17
rna was isolated	17
as a model	17
we demonstrated that	17
the stability of	17
antibody responses to	17
the challenge of	17
duan et al	17
to each well	17
disease in gnotobiotic	17
was obtained from	17
are listed in	17
in pigs and	17
leading to the	17
among prrsv strains	17
with or without	17
is shown in	17
was significantly reduced	17
evolution of porcine	17
the host cell	17
was not detected	17
of the innate	17
were cloned into	17
protective immune responses	17
decrease in the	17
lung lesion scores	17
with cold pbs	17
were pretreated with	17
coronaviridae and arteriviridae	17
blood samples were	17
tissue culture plates	17
were incubated at	17
and found that	17
prrsv have been	17
virus in swine	17
by the virus	17
was determined using	17
green monkey kidney	17
in eav nsp	17
ostrowski et al	17
a group of	17
vivo and in	17
of monocytes macrophages	17
washed once with	17
an attempt to	17
in comparison with	17
the parental virus	17
the existence of	17
to characterize the	17
the spread of	17
the gdd motif	17
immune response is	17
to control prrsv	17
of rna viruses	17
found that prrsv	17
been observed in	17
is not known	17
reproductive syndrome virus	17
of prrsv are	17
pulmonary alveolar macrophages	17
performed as described	17
were shown to	17
with the control	16
the active site	16
in the hp	16
immune response was	16
viral protein expression	16
modified live vaccines	16
ube d mrna	16
pigs coinfected with	16
described in the	16
infected with vr	16
benfield et al	16
times with pbs	16
pathogenicity of two	16
are associated with	16
characterization of porcine	16
the porcine respiratory	16
by using a	16
were fixed with	16
a better understanding	16
h prior to	16
plates were infected	16
were significantly higher	16
considered to be	16
replication of the	16
of prrsv strain	16
prrsv or pedv	16
carried out in	16
and the virus	16
infected cells at	16
we did not	16
have been observed	16
that they have	16
were collected from	16
activity in vitro	16
at c for	16
be used as	16
japanese encephalitis virus	16
based on a	16
mrna level of	16
during infection of	16
the combination of	16
weesendorp et al	16
the chimeric vlps	16
shown that prrsv	16
with the porcine	16
were harvested at	16
but not the	16
novel structural protein	16
inhibits porcine reproductive	16
in the orf	16
narita et al	16
no significant difference	16
m protein was	16
were added to	16
collected from the	16
and respiratory disease	16
sequence of a	16
the cell lysates	16
in lymphoid organs	16
american strains of	16
of the three	16
alveolar macrophages in	16
the prrsv gp	16
in group pigs	16
evolution of prrsv	16
syndrome virus encoded	16
the lelystad virus	16
appeared to be	16
of a highly	16
inhibition of porcine	16
caused by prrsv	16
an infectious cdna	16
culture infectious dose	16
replication in vitro	16
linked polyubiquitin chains	16
be related to	16
expression of both	16
differentiation and activation	16
were resuspended in	16
but does not	16
challenged with prrsv	16
the extent of	16
s p ratio	16
to inhibit the	16
lymph nodes and	16
in cells treated	16
which is a	16
wu et al	16
losses to the	16
for use in	16
the cytoplasm and	16
of cd and	16
provided by dr	16
and analyzed by	16
performed in triplicate	16
of adaptive immunity	16
of pigs persistently	16
have been described	16
isolated from the	16
virus attachment and	16
of the cells	16
in the uk	16
these results indicate	16
of the lungs	16
has been observed	16
infected or infected	16
that in the	15
its ability to	15
as the mean	15
to the control	15
these results indicated	15
the titers of	15
strains of the	15
to pigs infected	15
of differentiation and	15
interacts with the	15
antibodies to prrsv	15
pigs experimentally infected	15
the effectiveness of	15
during viral infection	15
been identified in	15
during persistent infection	15
at the n	15
declare that they	15
the arteriviridae family	15
number of cells	15
sows and respiratory	15
and h n	15
of lipid rafts	15
marle et al	15
influenza virus and	15
of clinical signs	15
of prrsv strains	15
economic losses in	15
accordance with the	15
of innate immune	15
statistical significance was	15
of the p	15
been reported that	15
in oral fluids	15
protein and the	15
work was supported	15
of e protein	15
total number of	15
infected cells and	15
in pigs vaccinated	15
of virus in	15
supernatants were harvested	15
to ldv and	15
production of infectious	15
use of a	15
test was used	15
reduction in the	15
then incubated with	15
essential for the	15
is composed of	15
knowledge on the	15
is known about	15
randomly assigned to	15
proportion of pigs	15
negative for prrsv	15
pcr amplification of	15
studies on the	15
was assessed by	15
have been used	15
immune responses by	15
for virus infectivity	15
significant increase in	15
may be due	15
the dko pigs	15
different time points	15
viral load in	15
the early stage	15
and may be	15
treated with m	15
of the predicted	15
of cd t	15
were found in	15
virus encoded by	15
is capable of	15
vaccines based on	15
of mg ml	15
the samples were	15
components of the	15
time pcr system	15
related to ldv	15
in comparison to	15
of these cells	15
and nsp are	15
a recent study	15
a large number	15
inhibition of viral	15
modified live porcine	15
of protective immunity	15
prrsv infection has	15
induced by a	15
sybr green i	15
significantly higher in	15
of the other	15
carried out using	15
and control of	15
and pp ab	15
for an additional	15
expression of pro	15
are presented as	15
reported to be	15
respiratory coronavirus in	15
the ectodomain of	15
in vivo studies	15
immunity in the	15
mlv mtb wcl	15
european isolates of	15
structural protein in	15
the same as	15
infection of porcine	15
for more than	15
was amplified from	15
syndrome virus is	15
formation of the	15
host immune responses	15
were then washed	15
lung lesions and	15
the viral life	15
they have no	15
levels of the	15
of the gdd	15
proinflammatory cytokines in	15
contributed to the	15
after incubation for	15
levels in the	15
and lymphoid tissues	15
mouse hepatitis virus	15
with nc b	15
virus from the	15
of porcine respiratory	15
atpase activity of	15
each time point	15
these results suggest	15
n protein of	15
to this end	15
of antibodies to	15
syndrome virus characterization	15
inserted into the	15
the immune responses	15
are shown as	15
virus strains of	15
the cysteine protease	15
with a potent	15
interfering with the	15
virus isolates with	15
secondary bacterial infections	15
the viral replication	15
the virus and	15
serial dilutions of	15
biology of arteriviruses	15
followed by incubation	15
responses to the	15
structural protein coding	15
was detected by	15
the influence of	15
in alveolar macrophages	15
and use committee	15
nuclear localization signal	15
by orfs to	15
the potential to	15
were determined using	15
of the parental	15
were fixed in	15
culture medium was	15
of gp and	15
of nsp and	15
immunodeficiency virus type	15
vaccines have been	15
can be found	15
and cellular immune	15
large number of	15
tissues of pigs	15
a model for	15
results demonstrated that	15
of the major	15
from the er	15
of the mutant	15
need to be	15
van marle et	15
differences between the	15
suggest that prrsv	15
prrsv in marc	15
be attributed to	15
were considered statistically	15
development of an	15
shown to inhibit	14
with porcine circovirus	14
as a control	14
thought to be	14
ability of the	14
the genetic diversity	14
of the experiment	14
a positive control	14
structural proteins are	14
the total number	14
compared to that	14
isolates with that	14
of porcine nidoviruses	14
compared to controls	14
knu cells were	14
to prrsv and	14
vaccine in pigs	14
compared to other	14
in the sera	14
ml of the	14
the transcription of	14
the cytoplasm of	14
in the lipid	14
of nonstructural protein	14
the susceptibility of	14
of the s	14
gene expression profiles	14
of the virion	14
to control prrs	14
mg ml of	14
into the cytoplasm	14
pulmonary intravascular macrophages	14
and infected with	14
with porcine respiratory	14
piglets infected with	14
the largest rna	14
from the same	14
cellular immune response	14
and then incubated	14
of the interferon	14
the construction of	14
incubation for h	14
were not detected	14
was incubated with	14
infection with a	14
antibodies in the	14
plates were incubated	14
had significantly higher	14
in the late	14
faaberg et al	14
stat and stat	14
oral fluid samples	14
compared to those	14
syndrome virus induces	14
with the same	14
the secretion of	14
cruz et al	14
porcine alveolar macrophage	14
under field conditions	14
stages of infection	14
of the upr	14
for the expression	14
science foundation of	14
genotype of porcine	14
american isolates of	14
cd a cd	14
the prrsv nsp	14
of a porcine	14
the culture supernatants	14
medicinal plant extracts	14
highly pathogenic strain	14
and cells were	14
wide range of	14
virus and the	14
the prrsv replicase	14
prrsv field isolates	14
log tcid ml	14
different from the	14
these results are	14
results suggested that	14
in utero infection	14
to those in	14
and stained with	14
porcine high fever	14
the acute phase	14
the nsp nsp	14
induction of ifn	14
influenza virus infection	14
significant differences between	14
is similar to	14
followed by the	14
by quantitative real	14
of the adaptive	14
associated with porcine	14
polyclonal b cell	14
from a prrsv	14
cytotoxic t lymphocyte	14
role in prrsv	14
lager et al	14
positive strand rna	14
the abundance of	14
protein is a	14
antigen was detected	14
of gene expression	14
in the expression	14
the indicated times	14
likely due to	14
to generate a	14
is not required	14
following prrsv infection	14
were carried out	14
tl co hb	14
concentration of mg	14
to the development	14
lung lesions were	14
is based on	14
for min in	14
prrsv m protein	14
different prrsv strains	14
in the induction	14
pp a and	14
an additional h	14
a and pp	14
orf a protein	14
and structural proteins	14
of the control	14
infection and the	14
inactivated prrsv vaccine	14
sequence of the	14
medium was replaced	14
the t cell	14
followed by cycles	14
that the expression	14
determined to be	14
have not been	14
this is the	14
immune system and	14
when compared with	14
to inhibit ifn	14
interaction with the	14
and differentiation of	14
compared to mock	14
cell responses to	14
to prrsv in	14
in this review	14
has been used	14
thai medicinal plant	14
of the humoral	14
in the middle	14
the innate and	14
to elucidate the	14
and interaction with	14
to swine arterivirus	14
in tissue culture	14
the membrane was	14
time points post	14
and transferred to	14
levels of viral	14
lps exposed pigs	14
diaz et al	14
for vaccine development	14
it is well	14
h n virus	14
c for h	14
antibodies against prrsv	14
are able to	14
was also observed	14
dependent on the	14
vaccines against porcine	14
with mn c	14
of this article	14
fresh medium containing	14
ingelvac prrs mlv	14
activity of porcine	13
the specificity of	13
van gorp et	13
important roles in	13
the antibody response	13
in the u	13
protein in porcine	13
most economically important	13
our results showed	13
was no significant	13
domain of porcine	13
for s and	13
was measured using	13
during acute infection	13
serve as a	13
to infection by	13
a crucial role	13
data are presented	13
virus belonging to	13
epidemiology of porcine	13
with bovine serum	13
green fluorescent protein	13
of all ages	13
by the end	13
the prrsv strain	13
were stimulated with	13
of total rna	13
vaccines against prrsv	13
has shown that	13
is not a	13
using trizol reagent	13
to form the	13
prrsv strain wuh	13
of the structural	13
encoded in orf	13
of vn antibodies	13
amino acid changes	13
infection in marc	13
that the virus	13
of humoral immunity	13
at each time	13
of the wt	13
inoculated with acaprrs	13
immune system is	13
would like to	13
disease of swine	13
pigs did not	13
by dendritic cells	13
interaction between prrsv	13
cd and papn	13
of nk cells	13
gene expression and	13
comparative pathogenicity of	13
of type porcine	13
with the recombinant	13
which has been	13
specific immune responses	13
the late s	13
type i and	13
mechanism by which	13
and approved the	13
virus and swine	13
in a co	13
is sufficient to	13
type and mutant	13
tracheobronchial lymph nodes	13
host innate immunity	13
of prrsv have	13
the e gene	13
infection in neonatal	13
european and north	13
correlates of protection	13
to understand the	13
of the second	13
sense rna virus	13
on their susceptibility	13
identification of porcine	13
fixed with paraformaldehyde	13
santa cruz biotechnology	13
the method of	13
microscopic lung lesions	13
duration of infection	13
in vitro interferon	13
high morbidity and	13
largest rna virus	13
of nsp was	13
entry of prrsv	13
were prepared in	13
challenge of prrs	13
was performed according	13
north american isolates	13
a wide range	13
virus replication by	13
against a heterologous	13
were introduced into	13
been found to	13
present in all	13
load in the	13
insights into the	13
of transmissible gastroenteritis	13
and the number	13
an internal control	13
required for the	13
to be determined	13
of p and	13
and treated with	13
in both marc	13
deletion in the	13
and the viral	13
epitopes in the	13
the proteolytic activity	13
secondary lymphoid organs	13
production of ifn	13
are known to	13
molecular biology and	13
washed five times	13
on the replication	13
pigs at dpc	13
and have been	13
there were no	13
probably due to	13
not affect the	13
difference in the	13
dual infections of	13
labarque et al	13
be important for	13
memory b cells	13
cells were further	13
are required for	13
and subjected to	13
significantly higher than	13
resulting in a	13
activation of monocytes	13
of prrs virus	13
dmem supplemented with	13
rna copy numbers	13
in pigs is	13
in the pathogenesis	13
of prrs immunology	13
gene expression in	13
severity of the	13
infected pigs were	13
experimentally infected pigs	13
mutational analysis of	13
biology of prrsv	13
to interact with	13
significant difference in	13
infection and proportion	13
and the expression	13
the nature of	13
infection or vaccination	13
measured using a	13
in our previous	13
in the usa	13
performed using a	13
the viability of	13
and release of	13
of alveolar macrophages	13
of this virus	13
natural science foundation	13
genes of porcine	13
renukaradhya et al	13
increased frequency of	13
virus replication is	13
the online version	13
impact on the	13
whole cell lysate	13
in the fetus	13
of the heterologous	13
replicase orf a	13
in the regulation	13
pigs had significantly	13
the cell membrane	13
human coronavirus e	13
be responsible for	13
is an essential	13
of the mean	13
extracted from the	13
as shown by	13
and centrifuged at	13
prrsv gp and	13
to the viral	13
of the efficacy	13
recombinant vaccinia virus	13
prrsv infection was	13
e protein is	13
the european genotype	13
had the highest	13
cells and pam	13
are representative of	13
of the present	13
inflammatory cytokine production	13
in growing pigs	13
differentially expressed proteins	13
replaced with fresh	13
used to evaluate	13
of influenza virus	13
structure of prrsv	13
dwivedi et al	13
when the cells	13
kb in length	13
in our laboratory	13
the results obtained	13
in the infection	13
that the e	13
the prevalence of	13
antiviral effect of	13
infected pigs and	13
to analyze the	13
was detected with	13
in of the	13
several studies have	13
interfere with the	13
emerging in china	13
stability of the	13
closely related to	13
the arterivirus group	13
in the two	13
gorp et al	13
the cause of	13
strain of the	13
in induction of	13
important for virus	13
nedialkova et al	13
a humidified co	13
in the frequency	13
of nonstructural proteins	13
the lung of	13
in cells expressing	13
and proportion of	13
during the course	13
d and d	13
spread of the	13
efficacy of a	13
which is the	13
a potent adjuvant	13
dpi and dpi	13
of virus and	13
feng et al	13
led to a	13
of prrsv were	13
diseases of swine	13
systemic immune responses	13
tumor necrosis factor	13
performed to determine	13
that has been	13
current knowledge on	13
were mock infected	13
and n protein	13
pigs of all	13
removal of the	13
north american genotype	13
is located in	13
interact with the	13
used to determine	12
divided into two	12
has been described	12
from porcine reproductive	12
cholesterol in the	12
prrsv genomic rna	12
presented as the	12
was essential for	12
treated cells were	12
to porcine respiratory	12
inhibit the replication	12
protection against heterologous	12
in europe and	12
syndrome virus emerging	12
the genomic rna	12
is difficult to	12
the equine arteritis	12
r o o	12
foundation of china	12
the catalytic domain	12
fold dilutions of	12
sialoadhesin and cd	12
detection of the	12
p r e	12
the two groups	12
been demonstrated that	12
guo et al	12
of two us	12
were analyzed for	12
prrsv and pcv	12
the pig lung	12
disease caused by	12
isolate of prrsv	12
multisystemic wasting syndrome	12
in fetal and	12
and type prrsv	12
virus emerging in	12
for the first	12
inhibits prrsv replication	12
it should be	12
the replication cycle	12
to better understand	12
for viral replication	12
and cloned into	12
the dynamics of	12
compared with those	12
with prrsv for	12
north american porcine	12
the severe acute	12
choi et al	12
a virulent strain	12
american porcine reproductive	12
the significance of	12
cdna clone of	12
using the same	12
the modulation of	12
result of the	12
genotypes of prrsv	12
the suppression of	12
structural proteins in	12
and pam cultures	12
was measured at	12
were derived from	12
united states pork	12
of clinical disease	12
response of pigs	12
have been developed	12
showed that nsp	12
cells in a	12
statistical analyses were	12
different respiratory virus	12
autophagosomes induced by	12
reactive oxygen species	12
analysis of variance	12
j o u	12
nonstructural protein of	12
endoribonuclease activity of	12
cell signaling technology	12
protein expression was	12
osorio et al	12
virus in vitro	12
tijms et al	12
not have an	12
of orf and	12
european genotype of	12
indicated time points	12
in pigs naturally	12
p r o	12
of these cytokines	12
a l p	12
syndrome virus through	12
presence of virus	12
of lactate dehydrogenase	12
seybert et al	12
protective immunity to	12
was calculated using	12
steps of the	12
the sequences of	12
alpha response to	12
immunoperoxidase monolayer assay	12
response was observed	12
by western blotting	12
the intensity of	12
of a prrsv	12
are consistent with	12
prrsv infected pigs	12
responses of swine	12
in the blood	12
the culture supernatant	12
for h prior	12
lesions in the	12
to control the	12
the most common	12
the sequence of	12
detection of antibodies	12
the plp dub	12
in mammalian cells	12
crucial role in	12
the final manuscript	12
prrsv is an	12
a lesser extent	12
effect on prrsv	12
virus titers were	12
detected in all	12
endoplasmic reticulum stress	12
required for prrsv	12
treated with the	12
were selected for	12
the virus is	12
porcine nidovirus replication	12
out of the	12
monoclonal antibodies to	12
evaluation of the	12
weeks of age	12
r n a	12
isolates in a	12
vaccination with a	12
approved the final	12
interaction of the	12
arterivirus molecular biology	12
to one of	12
correlations with pathogenicity	12
is a positive	12
pathogenesis and interaction	12
two us porcine	12
compare lanes and	12
were similar to	12
virus gp ectodomain	12
mechanisms by which	12
with the nsp	12
by inhibition of	12
immunity against porcine	12
lung during different	12
of dendritic cell	12
presence of sqe	12
expression of a	12
pig lung during	12
coronavirus in pigs	12
were infected at	12
rna virus genome	12
of respiratory disease	12
and the results	12
emerging and re	12
n a l	12
as measured by	12
in the vector	12
functions as a	12
repertoire development in	12
due to their	12
can also be	12
were associated with	12
were normalized to	12
amplified by pcr	12
of the recombinant	12
the mechanisms of	12
cells were stained	12
and expression of	12
the cell culture	12
be able to	12
inactivated porcine reproductive	12
read and approved	12
to induce a	12
and dendritic cells	12
inhibit prrsv replication	12
l p r	12
ability of prrsv	12
virus titer was	12
sasa quelpaertensis nakai	12
to the porcine	12
collins et al	12
number of cd	12
of the manuscript	12
of orf a	12
d and e	12
on united states	12
killed prrsv vaccine	12
detection of viral	12
the arterivirus nsp	12
oleksiewicz et al	12
the probability of	12
elevating virus and	12
of the respiratory	12
respiratory virus infections	12
may not be	12
u r n	12
the reaction was	12
e and f	12
of the pathogenicity	12
nsp to inhibit	12
differentially expressed genes	12
during different respiratory	12
immunity and porcine	12
protein encoded by	12
i interferon production	12
room temperature for	12
susceptibility to secondary	12
a persistent infection	12
viral genomic rna	12
the need for	12
for prrsv and	12
or swine influenza	12
by cycles of	12
of mycobacterium tuberculosis	12
the virulence of	12
studies are needed	12
viral genome and	12
the n gene	12
to interfere with	12
in the cells	12
o o f	12
equine influenza virus	12
seems to be	12
of the replicase	12
present study was	12
in lung tissues	12
significantly higher levels	12
it would be	12
cell culture media	12
morgan et al	12
critical for the	12
and the cytoplasm	12
vaccinated with prrs	12
than in the	12
syndrome virus rna	12
a role for	12
lin et al	12
detection and differentiation	12
kg body weight	12
biology and pathogenesis	12
states pork producers	12
had no effect	12
forsberg et al	12
immunogenicity of the	12
antigen in the	12
of this protein	12
and resuspended in	12
which may be	12
prrsv for h	12
were amplified by	12
focused on the	12
o u r	12
contrast to the	12
also observed in	12
cdna clones of	12
be found in	12
sodium dodecyl sulfate	12
to prrsv is	12
buffer containing mm	12
authors read and	12
was amplified by	12
with the expression	12
antibody repertoire development	12
findings indicate that	12
significant differences in	12
titers of prrsv	12
may be the	12
type prrsv strain	12
the increase in	12
the phosphorylation of	12
are indicated by	12
to the same	12
virus on united	12
the infectivity of	12
north american strain	12
proteins of prrsv	12
the differences in	12
the proteins were	12
in marc cells	12
were used in	12
to confirm the	12
effect of porcine	12
have no competing	12
medium supplemented with	12
shen et al	12
of pigs were	12
analyzed using the	12
institutional animal care	12
current status and	12
at the p	12
primers used for	12
humidified co incubator	12
both prrsv and	11
in the other	11
protein expression in	11
nucleus and the	11
gross lung lesions	11
h post infection	11
and analyzed for	11
these data indicate	11
the serum samples	11
from this study	11
sites in the	11
development of adaptive	11
lelystad virus and	11
diseases in pigs	11
sg mrna s	11
of the sdd	11
gene expression levels	11
pigs challenged with	11
in viral rna	11
to protect against	11
the exception of	11
and european porcine	11
the plp cis	11
with the exception	11
the design of	11
to secondary bacterial	11
difference between the	11
prrsv appears to	11
the binding of	11
serves as a	11
virus challenged pigs	11
host responses to	11
days after challenge	11
studies showed that	11
the recombinant baculovirus	11
treated with ifn	11
could be observed	11
in this regard	11
exploring the multifunctionality	11
in a single	11
been identified as	11
and swine influenza	11
was identified as	11
severity of clinical	11
prrsv may be	11
in d cells	11
it was reported	11
a reduction in	11
in escherichia coli	11
cd cd t	11
disease of pigs	11
residues of the	11
infected with nc	11
plays a key	11
paraformaldehyde for min	11
influenza a viruses	11
phosphorylation and nuclear	11
results are expressed	11
and the presence	11
challenge with mn	11
macrophages on their	11
development in fetal	11
not be detected	11
of b cells	11
transmissible gastroenteritis coronavirus	11
of an infectious	11
emerged in china	11
swine infl uenza	11
and nonneutralizing epitopes	11
mediated by the	11
for their ability	11
tumour necrosis factor	11
presence of a	11
among prrsv isolates	11
national natural science	11
the results in	11
to the proteins	11
could be detected	11
titers were measured	11
was reported to	11
titers in the	11
nielsen et al	11
of at least	11
in prrsv pathogenesis	11
of nsp is	11
addition to the	11
pigs at different	11
for h in	11
the sites of	11
of the antiviral	11
comparison of porcine	11
h at rt	11
the complement system	11
we hypothesized that	11
at dpi in	11
of the four	11
prrsv isolates and	11
course of infection	11
role of porcine	11
to a lesser	11
a monoclonal antibody	11
us prrsv vr	11
that the presence	11
of genomic rna	11
of the human	11
depletion of cellular	11