quadgram

This is a table of type quadgram and their frequencies. Use it to search & browse the list to learn more about your study carrel.

quadgram	frequency
of influenza a virus	165
n and h n	116
h n and h	115
influenza a virus infection	89
has been shown to	83
in the presence of	74
h n influenza virus	72
memory cd t cells	69
influenza a h n	63
in the absence of	61
middle east respiratory syndrome	59
h n influenza a	57
severe acute respiratory syndrome	54
of influenza a viruses	53
h n influenza viruses	52
have been shown to	51
east respiratory syndrome coronavirus	47
pandemic h n influenza	46
cells were infected with	44
influenza a virus in	44
in vitro and in	43
highly pathogenic avian influenza	40
cd t cells in	40
in the united states	40
the influenza a virus	40
and h n viruses	39
in the context of	39
vitro and in vivo	39
acute respiratory syndrome coronavirus	37
been shown to be	37
to display the preprint	35
is the author funder	35
the preprint in perpetuity	35
license to display the	35
a h n virus	35
for the treatment of	35
medrxiv a license to	35
has granted medrxiv a	35
granted medrxiv a license	35
display the preprint in	35
who has granted medrxiv	35
a license to display	35
in the case of	35
of ha and na	34
acute respiratory distress syndrome	33
specific cd t cells	33
t cells in the	33
of the influenza virus	33
n influenza a virus	33
in the upper respiratory	32
influenza virus infection in	31
of the h n	31
of the influenza a	31
an important role in	31
h n viruses were	31
the upper respiratory tract	31
on the surface of	31
in influenza a virus	31
were found to be	30
of the nlrp inflammasome	30
h n virus was	29
as shown in fig	29
of h n virus	28
to influenza a virus	28
of cd t cells	28
h n virus in	28
on the other hand	27
cells were transfected with	26
against influenza a virus	26
respiratory syncytial virus infection	26
not certified by peer	25
certified by peer review	25
which was not certified	25
was not certified by	25
the lower respiratory tract	25
as well as the	25
of h n influenza	24
cd t cell responses	24
the first report of	24
influenza a and b	24
of pandemic h n	23
in a mouse model	23
h n swine influenza	23
influenza a virus replication	23
during influenza virus infection	23
a cells were infected	23
the ha and na	23
in the pathogenesis of	23
holder for this preprint	22
this preprint this version	22
copyright holder for this	22
the h n virus	22
the copyright holder for	22
preprint this version posted	22
the pandemic h n	22
for this preprint this	22
human airway epithelial cells	22
human immunodeficiency virus type	21
influenza a viruses in	21
h n virus isolates	21
in the lungs of	21
the activation of the	21
no reuse allowed without	20
reuse allowed without permission	20
has been reported to	20
with influenza a virus	20
a mouse model of	20
n influenza a viruses	20
one of the most	19
cd t cell response	19
specific memory cd t	19
cells were treated with	19
able to bind to	19
have been reported to	18
influenza a virus and	18
at a moi of	18
n influenza virus infection	18
influenza a virus hemagglutinin	18
influenza h n virus	18
were detected in swine	18
for influenza virus replication	18
oxidative medicine and cellular	17
reproductive and respiratory syndrome	17
as a result of	17
of the immune system	17
porcine reproductive and respiratory	17
a and b viruses	17
the h n and	17
protein of influenza a	17
min at room temperature	17
influenza a virus from	17
of influenza virus infection	17
h n viruses in	17
play a role in	17
medicine and cellular longevity	17
of h n pdm	17
activation of the nlrp	16
also been shown to	16
of human influenza a	16
in the lower respiratory	16
of middle east respiratory	16
and h n influenza	16
for the development of	16
highly pathogenic h n	16
of respiratory syncytial virus	16
h n avian influenza	16
and respiratory syndrome virus	16
for min at room	16
of the innate immune	16
response to iav infection	16
the innate immune response	16
reassortant h n virus	16
of human immunodeficiency virus	16
memory cd t cell	16
specific cd t cell	15
studies have shown that	15
play an important role	15
the expression of the	15
of avian h n	15
a wide range of	15
had no effect on	15
of influenza viruses in	15
van eijk et al	15
has also been shown	15
n swine influenza virus	15
was found to be	15
iav h n pdm	15
the surface of the	15
has been demonstrated to	15
at the nucleotide level	15
innate immune response to	15
avian influenza h n	15
in a murine model	15
it is possible that	15
h n virus infection	15
plays an important role	15
origin h n influenza	15
type i and type	14
the network data exchange	14
the host immune response	14
on the expression of	14
viruses were detected in	14
these results suggest that	14
was detected in the	14
made available under a	14
in addition to the	14
of swine influenza virus	14
presence or absence of	14
it has been shown	14
to influenza virus infection	14
n influenza viruses in	14
to the activation of	14
proteins a and d	14
surfactant proteins a and	14
type i and iii	14
a murine model of	14
is known to be	14
for influenza a virus	14
in the respiratory tract	14
h n pdm virus	14
of tumor necrosis factor	14
is also able to	14
type i ifn signaling	14
was shown to be	14
ha and na genes	14
of severe acute respiratory	14
a and mdck cells	13
cd t cells are	13
a is the author	13
of avian influenza a	13
under a is the	13
infected with h n	13
h n pdm and	13
is able to bind	13
the innate immune system	13
it is made available	13
license it is made	13
this version posted may	13
h n virus antibodies	13
in contrast to the	13
is made available under	13
is involved in the	13
the nucleotide level and	13
in the treatment of	13
h n and a	13
international license it is	13
cells in the lung	13
swine influenza a virus	13
a virus infection by	13
of h n and	13
for the detection of	13
followed by infection with	13
influenza virus h n	13
influenza viruses in pigs	13
avian h n and	13
available under a is	13
virus was detected in	13
as shown in figure	13
the authors declare no	13
the presence or absence	13
influenza a virus ns	13
is one of the	13
infection with pr virus	13
of influenza virus hemagglutinin	13
of influenza a h	13
the amino acid level	13
has also been reported	12
a critical role in	12
avian influenza a h	12
to the cell surface	12
at the amino acid	12
of swine influenza viruses	12
were infected with iav	12
of surfactant protein a	12
in agreement with the	12
the cd t cell	12
were found infected with	12
innate and adaptive immunity	12
were found positive for	12
response to influenza a	12
on the cell surface	12
the nuclear export of	12
pathogenic avian influenza virus	12
virus infection in mice	12
the highest number of	12
of three independent experiments	12
at an moi of	12
human h n virus	12
immune response to influenza	12
with respiratory syncytial virus	12
the treatment of iav	12
of pandemic influenza a	12
as well as in	12
the initial binding rate	12
infected with pr virus	12
in response to iav	12
of influenza c virus	12
and pandemic h n	12
were transfected with a	12
i and type iii	11
the nlrp inflammasome in	11
transmission of avian influenza	11
cd t cells and	11
the h n influenza	11
morbidity and mortality in	11
likely due to the	11
of influenza virus infections	11
induction of type i	11
of type i ifn	11
in the regulation of	11
this is the first	11
of cd t cell	11
influenza virus m protein	11
in the presence or	11
human bronchial epithelial cells	11
has the potential to	11
in human airway epithelial	11
the development of novel	11
response to viral infection	11
against influenza virus infection	11
of the inflammatory response	11
n viruses were detected	11
in the induction of	11
of memory cd t	11
cells infected with iav	11
characterization of h n	11
n influenza virus in	11
the protective role of	11
in the development of	11
influenza virus infection of	11
cd t cell effector	11
inflammatory cytokines and chemokines	11
of type i interferon	11
the antiviral activity of	11
by infection with pr	11
and lower respiratory tract	11
has been shown that	11
been shown to inhibit	11
resident memory cd t	11
increase in the ifitm	11
by influenza a virus	11
have been associated with	11
viruses in pigs in	11
the iav control group	11
models of acute pneumonia	11
been reported to be	11
on the viral surface	11
versus the iav control	11
innate and adaptive immune	11
the mechanism by which	11
the authors declare that	10
and h n virus	10
transmission of influenza a	10
the release of viral	10
the role of the	10
the h n pandemic	10
at week and week	10
identification and characterization of	10
influenza virus ns protein	10
no conflict of interest	10
in the antigenic sites	10
upper respiratory tract of	10
as compared to the	10
and influenza a virus	10
and h n subtypes	10
a virus ns protein	10
be involved in the	10
these results indicate that	10
induced by pr virus	10
no vac no cha	10
for the generation of	10
upper and lower respiratory	10
a virus infection in	10
mouse models of acute	10
of an h n	10
suggesting that pa may	10
in comparison with pr	10
cells in response to	10
inhibition of iav infection	10
at different time points	10
the h n pdm	10
by the addition of	10
of mice infected with	10
n virus isolates were	10
during influenza a virus	10
ha and na proteins	10
pandemic h n virus	10
of influenza virus in	10
in the activation of	10
swine in the united	10
i and iii ifns	10
pi k akt pathway	10
been shown to have	10
restricts influenza a virus	10
sequences obtained from pigs	10
in the response to	10
in line with this	10
a strong nuclear np	10
innate immunity to influenza	10
for the production of	10
in iav infected cells	10
human h n influenza	10
for the first time	10
have been identified in	10
were infected with a	10
west nile virus infection	10
was used as a	10
by the presence of	10
is thought to be	9
cells were incubated with	9
of the immune response	9
with adnrf followed by	9
respiratory syndrome coronavirus infection	9
influenza virus in a	9
of surfactant protein d	9
required for influenza virus	9
n avian influenza virus	9
claims in published maps	9
the immune response to	9
and other respiratory viruses	9
of influenza b virus	9
influenza virus infections in	9
of the ha protein	9
t cell response to	9
published maps and institutional	9
following respiratory virus infection	9
the presence of serum	9
in the immune system	9
n viruses were identified	9
ind entry of iav	9
systematic review and meta	9
during respiratory syncytial virus	9
nature remains neutral with	9
strains of h n	9
and activator of transcription	9
in published maps and	9
avian and human iavs	9
signal transducer and activator	9
has been implicated in	9
with pr virus alone	9
of the pandemic h	9
ha and na subtypes	9
is consistent with the	9
influenza a virus infections	9
in the lung airways	9
cells were seeded in	9
with regard to jurisdictional	9
transducer and activator of	9
and the h n	9
were found to have	9
to the h n	9
it is likely that	9
response to cov infection	9
springer nature remains neutral	9
have been reported in	9
at the site of	9
jurisdictional claims in published	9
h n and pandemic	9
reported in swine in	9
influenza a virus m	9
the context of viral	9
a role in the	9
a cells infected with	9
similar to that of	9
it has been reported	9
was performed using the	9
to influenza a h	9
of the viral genome	9
in influenza virus infection	9
remains neutral with regard	9
in accordance with the	9
neutral with regard to	9
of type i ifns	9
respiratory viral infections in	9
like and atii cells	9
in the present study	9
and respiratory syncytial virus	9
both in vitro and	9
at the same time	9
to jurisdictional claims in	9
the presence of the	9
mdck cells were infected	9
maps and institutional affiliations	9
lower respiratory tract of	9
obtained from pigs sampled	9
of surfactant proteins a	9
the course of infection	9
the lungs of mice	9
respiratory syncytial virus and	9
this version posted april	9
a wide variety of	9
n influenza viruses from	9
a single amino acid	9
h and h challenge	9
regard to jurisdictional claims	9
response to sars infection	9
been shown to bind	9
the presence of fcs	9
a broad spectrum of	9
influenza viruses in swine	8
also able to bind	8
the inhibitory effect of	8
generation of recombinant influenza	8
role of nrf in	8
through the activation of	8
note springer nature remains	8
that they have no	8
an influenza a virus	8
the viral polymerase complex	8
the specificity of the	8
of the virus to	8
and cd t cells	8
influenza virus isolated from	8
has been reported that	8
iav infected a cells	8
like h n and	8
with h n pdm	8
human influenza a viruses	8
strong nuclear np signal	8
effects of curcumin derivatives	8
virus was isolated from	8
resistance to influenza a	8
origin h n virus	8
the treatment of influenza	8
authors declare no conflict	8
the end of the	8
has been associated with	8
to the induction of	8
in this study were	8
nasal swabs and balf	8
for the evaluation of	8
isolation and characterization of	8
and the cells were	8
influenza a virus entry	8
closely related to the	8
on influenza a virus	8
production of type i	8
cd t cells is	8
play a critical role	8
the ha na balance	8
was obtained from the	8
mortality associated with influenza	8
pfu cell pr virus	8
of wdnhbe cells with	8
of influenza a and	8
h n or h	8
was added to the	8
of avian and human	8
the magnitude of the	8
our understanding of the	8
against h n and	8
and type iii ifn	8
n virus was reported	8
for min at uc	8
and characterization of the	8
iav infection in a	8
treated with or without	8
used in this study	8
t cell effector functions	8
pandemic influenza a h	8
responses to influenza a	8
a type ii transmembrane	8
swine influenza virus in	8
declare no conflict of	8
of hepatitis c virus	8
authors declare that they	8
of cytokines and chemokines	8
to swine transmission of	8
a virus in swine	8
pig id at week	8
in lung epithelial cells	8
our data suggest that	8
of iav infection in	8
to be able to	8
respiratory virus infection in	8
and h challenge strains	8
pulmonary surfactant protein a	8
the emergence of a	8
both h n and	8
was also shown to	8
the pathogenesis of iav	8
the activation of nf	8
the case of sars	8
the pathogenesis of influenza	8
n or h n	8
cytokines and chemokines in	8
of swine influenza a	8
pathogenic avian influenza h	8
rift valley fever virus	8
a large number of	8
comparison with pr virus	8
of the role of	8
influenza viruses from pigs	8
in cd t cell	8
from apparently healthy swine	8
complete genome sequence of	8
type i ifn production	8
surfactant protein d in	8
associated with severe influenza	8
formation of fusion pores	8
n pdm and poly	8
at the cell surface	8
prior to infection with	8
structure of the haemagglutinin	8
shown to increase the	8
to the plasma membrane	8
and its role in	8
of the cd t	8
of highly pathogenic avian	8
the generation of recombinant	8
the plasma membrane of	8
declare that they have	8
the upper and lower	8
ns protein of influenza	8
respiratory tract of humans	8
influenza a virus nucleoprotein	8
like h n viruses	8
with h n influenza	8
at later time points	8
in the supernatants of	8
the formation of fusion	8
van asbeck et al	8
blunt the cytokine storm	8
cells were fixed with	8
influenza a viruses from	8
avian h n influenza	8
to be involved in	8
infections in hospitalized children	8
been shown to increase	8
of a number of	8
iav infection in the	8
of avian influenza virus	8
to be associated with	7
replication and gene transcription	7
pandemic influenza a virus	7
we were able to	7
the unfolded protein response	7
a better understanding of	7
was shown to decrease	7
animal care and use	7
to the presence of	7
is likely due to	7
shown to bind to	7
against respiratory syncytial virus	7
was shown to increase	7
it has also been	7
we found that the	7
the length of the	7
h n iav infection	7
swine h n virus	7
at the plasma membrane	7
work was supported by	7
inhibition of influenza virus	7
influenza virus replication and	7
a virus infection and	7
infected with a pr	7
to be required for	7
as shown in table	7
cells were inoculated with	7
from a pig in	7
fusion of the viral	7
influenza virus infection a	7
sibu and kapit hospitals	7
cells and alveolar macrophages	7
has been proposed to	7
and ali bx zhou	7
was determined by real	7
fusion in virus entry	7
as well as by	7
with influenza a viruses	7
it was demonstrated that	7
cells in the presence	7
were reported in swine	7
peripheral blood mononuclear cells	7
type i interferon signaling	7
lower respiratory tract infection	7
of type i and	7
was used to assess	7
physiologic levels of gm	7
the induction of macropinocytosis	7
the airway epithelium in	7
care and use committee	7
mbl is able to	7
nl and hl nl	7
several studies have shown	7
structure and function of	7
and genetic characteristics of	7
was reported to be	7
avian influenza a virus	7
induces apoptosis in human	7
transmission of h n	7
frontiers in immunology www	7
swine h n influenza	7
at the time of	7
the spatial organization of	7
and characterization of a	7
entry by blocking the	7
immunity to influenza a	7
viruses were identified in	7
infection with adnrf followed	7
receptor binding and membrane	7
of h n iav	7
was detected in swine	7
is responsible for the	7
ac and muc b	7
that cd t cells	7
of pig id at	7
virus infection in a	7
approximately log decrease at	7
of type i interferons	7
analysis was performed using	7
activates the nlrp inflammasome	7
response to h n	7
cellular pi k akt	7
growth factor receptor tyrosine	7
were approved by the	7
influenza a virus the	7
of iav in swine	7
respiratory syncytial virus in	7
inflammation and ali bx	7
in mice infected with	7
between iav infection and	7
cells were fixed and	7
this work was supported	7
it is clear that	7
antigenic and genetic characteristics	7
the host cell surface	7
of influenza virus at	7
the presence of a	7
the type i interferon	7
is well known that	7
se of three independent	7
van iwaarden et al	7
immunity to influenza virus	7
and membrane fusion in	7
the results showed that	7
characterization of a novel	7
to that of the	7
important role in the	7
the integrity of the	7
of h n avian	7
cells in the lungs	7
the viruses and their	7
were infected with pr	7
in the inhibition of	7
hl nl and hl	7
was also observed in	7
for disease control and	7
membrane fusion in virus	7
threat to public health	7
were treated with curcumin	7
in the endoplasmic reticulum	7
association between iav infection	7
detection of influenza a	7
bhb was shown to	7
in the lung and	7
of pi k akt	7
a systematic review and	7
avian h n virus	7
the role of nrf	7
induced inflammation and ali	7
does not appear to	7
the human cathelicidin ll	7
been implicated in the	7
to h n pdm	7
pathogenic avian influenza a	7
cells of the immune	7
diversity of swine influenza	7
antiviral effects of curcumin	7
structure of the influenza	7
of a novel influenza	7
alveolar type ii cells	7
the wiv treatment groups	7
of avian influenza viruses	7
in the culture supernatants	7
genome replication and gene	7
membrane glycoprotein of influenza	7
the ns protein of	7
on the host cell	7
was supported by the	7
type i ifn in	7
the role of host	7
for treatment of influenza	7
was a gift from	7
a broad range of	7
in cells infected with	7
on the role of	7
influenza and other respiratory	7
the periphery of the	7
with or without pa	7
are expressed as the	7
the carbohydrate recognition domain	7
the site of infection	7
the middle east respiratory	7
is also involved in	7
factor receptor tyrosine kinases	7
lower respiratory tract infections	7
it is well known	7
the antiviral potential of	7
of the airway epithelium	7
data are expressed as	7
iav strains in swine	7
glycoprotein of influenza virus	7
in the current study	7
and one h n	7
the host immune system	7
washed three times with	7
h n influenza in	7
both h and h	7
inhibition of np expression	7
muc ac and muc	7
our results show that	7
divergent at the nucleotide	7
cells in the absence	7
hepatitis c virus infection	7
to blunt the cytokine	7
in patients with severe	7
and h n iav	7
virus hemagglutinin and neuraminidase	7
of viral respiratory infections	7
vivo and in vitro	7
disease control and prevention	7
of swine influenza in	7
binding and membrane fusion	7
hek cells were transfected	7
of seasonal and pandemic	7
the infectious diseases ward	7
viruses and their replication	7
detected by western blotting	6
and na genes of	6
are shown in figure	6
of acute respiratory distress	6
for the activation of	6
these data suggest that	6
the apical surface of	6
of severe influenza in	6
n highly pathogenic avian	6
authors declare no competing	6
pdm virus isolates were	6
of the viral polymerase	6
an avian h n	6
g cell cycle arrest	6
has been linked to	6
can lead to the	6
and h n in	6
for efficient influenza virus	6
decrease at lg ml	6
viruses have been reported	6
inhibition of influenza a	6
severe influenza virus infection	6
into the mitochondrial matrix	6
host factors crucial for	6
used to determine the	6
from pig id at	6
identifies human host factors	6
added to each well	6
promoter is associated with	6
is dependent on the	6
the extent to which	6
influenza viruses isolated from	6
highly pathogenic strains of	6
to play a role	6
the first evidence of	6
type i interferon production	6
cd t cells to	6
expression was detected by	6
in the airway epithelium	6
of acute lung injury	6
at weeks of age	6
in the course of	6
cd t cells also	6
with the plasma membrane	6
blocking the formation of	6
mediated activation of the	6
on the infectious diseases	6
seroprevalence of h n	6
were negative for the	6
an increased ifitm signal	6
iav h n infection	6
infection with h n	6
the h n viruses	6
associated with influenza a	6
has been proven to	6
pathology of influenza virus	6
of influenza virus replication	6
has been found to	6
no significant difference in	6
and the expression of	6
efficient influenza virus propagation	6
the increased expression of	6
type i interferon and	6
isolated from pigs in	6
lung surfactant protein d	6
influenza a virus through	6
human influenza a h	6
protein expression was detected	6
novel reassortant h n	6
would like to thank	6
outbreak of influenza a	6
prevalence of iav in	6
the nature of the	6
ifitm restricts the morbidity	6
injury induced by pr	6
new world bats harbor	6
apoptosis in human airway	6
mediate cellular resistance to	6
the nlrp inflammasome and	6
may contribute to the	6
leads to the activation	6
and mortality associated with	6
the survival rate of	6
no effect on the	6
rnai screen identifies human	6
the ifitm proteins mediate	6
as well as to	6
factors crucial for influenza	6
human alveolar epithelial cells	6
by type i ifn	6
for h at room	6
strains of influenza a	6
human respiratory syncytial virus	6
the plasma membrane and	6
the s r mutation	6
expressed as the mean	6
wide rnai screen identifies	6
induction of innate immune	6
susceptible to iav infection	6
the production of infectious	6
was detected by western	6
infected with influenza a	6
diverse influenza a viruses	6
treatment of iav infection	6
cd t cells following	6
induced acute lung injury	6
network data exchange sars	6
induced lung injury and	6
in an elementary school	6
an overview of the	6
virus entry by blocking	6
we did not observe	6
were included in the	6
detected in swine sera	6
and the united states	6
the protective effect of	6
h n highly pathogenic	6
consensomes and hct intersection	6
of fusion pores following	6
the acute respiratory distress	6
the influenza virus hemagglutinin	6
shown to inhibit the	6
pi k akt signaling	6
sitosterol on the expression	6
in response to viral	6
evolution and ecology of	6
hek t cells were	6
were significantly dysregulated by	6
it is believed that	6
expression was determined by	6
national institutes of health	6
and were found to	6
the expression of rig	6
test was used to	6
of influenza virus is	6
in a and mdck	6
of the respiratory tract	6
the possible role of	6
virus was reported in	6
reassortment between avian and	6
to the production of	6
have no competing interests	6
iav infection in mice	6
entry into host cells	6
ecology of influenza a	6
was performed using a	6
fusion pores following virus	6
by focus formation assay	6
of human and animal	6
infected a cells were	6
to the expression of	6
in absence of oc	6
chronic obstructive pulmonary disease	6
id at week and	6
an increasing number of	6
virus isolated from pigs	6
human host factors crucial	6
comparative histopathology of mouse	6
the induction of type	6
were identified in the	6
log decrease at lg	6
receptor specificity of the	6
swine influenza a viruses	6
cd t cells that	6
cd and cd t	6
the total number of	6
the antigenic sites of	6
acute lung injury in	6
the severity of the	6
hemagglutinin and neuraminidase activities	6
linear ubiquitin chain assembly	6
pandemic a h n	6
can also bind to	6
carbohydrate recognition domain of	6
in hospitalized children in	6
screen identifies human host	6
ifitm restricts influenza a	6
and human h n	6
and h n avian	6
influenza other respir viruses	6
risk of severe influenza	6
n swine influenza viruses	6
the expression of proinflammatory	6
sialic acids on the	6
influenza a virus blocks	6
virus titers in the	6
of human influenza viruses	6
cells infected with pandemic	6
of the plasma membrane	6
h n and human	6
h n dk variant	6
the morbidity and mortality	6
and severe acute respiratory	6
infection of mice with	6
were identified in swine	6
human influenza viruses to	6
declare no competing interests	6
of swine h n	6
novel influenza a virus	6
induced by iav infection	6
van de wetering et	6
in the lung during	6
relationship and antiviral effects	6
seasonal and pandemic influenza	6
cellular resistance to influenza	6
a virus in mice	6
the cells were lysed	6
may be involved in	6
virus at a moi	6
the molecular basis of	6
viral titers in the	6
a virus entry by	6
ifitm proteins mediate cellular	6
read and approved the	6
the antiviral effects of	6
detected in swine in	6
the nlrp inflammasome by	6
the pandemic potential of	6
histopathology of mouse models	6
morbidity and mortality associated	6
in the direction of	6
is important for the	6
the last two decades	6
be explained by the	6
n virus in swine	6
inhibitor of apoptosis protein	6
the pathology of influenza	6
susceptibility to influenza a	6
bind to sialic acid	6
the expression of il	6
as part of the	6
washed and incubated with	6
an increase in the	6
of innate immune responses	6
rna and dna viruses	6
the adaptive immune response	6
will be required to	6
incubated for min at	6
harbor diverse influenza a	6
as well as their	6
restricts the morbidity and	6
in the peripheral blood	6
the ifitm signal intensity	6
as well as a	6
hepatitis c virus p	6
of mouse models of	6
we were unable to	6
have also been reported	6
to the lack of	6
by blocking the formation	6
in response to the	6
and antiviral effects of	6
by both h n	6
type ii transmembrane protein	6
can be induced by	6
atii cells and am	6
of defective interfering particles	6
these studies indicate that	6
in figshare file f	6
crucial for influenza virus	6
is located in the	6
host immune response to	6
human transmission of avian	6
de wetering et al	6
h n viruses reported	6
reassortant h n viruses	6
is associated with severe	6
pathogenic potential of interferon	6
h at room temperature	6
cd t cells from	6
in combination with the	6
from three independent experiments	6
proteins mediate cellular resistance	6
h n influenza pandemic	6
and the number of	6
of porcine reproductive and	6
antibodies were detected in	6
treated with curcumin derivatives	6
vesicular stomatitis virus infection	6
cells were maintained in	6
a cells were transfected	6
h n virus and	6
cells are critical for	6
in response to influenza	6
no vac cha group	6
influenza viruses have been	6
eukaryotic translation initiation factor	6
could be due to	6
d is also able	6
effect of h n	6
and is able to	6
pr virus at a	6
in swine in the	6
origin iav strains in	6
a virus replication in	6
cd t cells play	6
and adaptive immune responses	6
was added to each	6
this is consistent with	6
pathogenic avian influenza viruses	6
reassortant h n influenza	6
of iav h n	6
pb e k mutation	6
they have no competing	6
are required for the	6
prevention and control of	6
influenza virus infection and	6
the fact that the	6
swabs and balf samples	6
in line with the	6
at the end of	6
and approved the final	6
was approved by the	6
protects mice from lethal	6
of influenza virus a	6
a h n viruses	6
may be able to	6
play important roles in	6
n virus antibodies in	6
can also be used	6
human to swine transmission	6
a h n influenza	6
and ecology of influenza	6
world bats harbor diverse	6
bats harbor diverse influenza	6
in acute influenza infection	6
we would like to	6
ha and na on	6
the heterologous aut com	6
h n infection in	6
to the initial binding	6
response to influenza virus	6
the fusion of the	6
the human respiratory tract	6
of viral infection and	6
novel avian influenza a	6
of recombinant influenza viruses	6
of the viral and	6
natural science foundation of	6
authors read and approved	6
divergent at the amino	6
virus in swine in	6
a key role in	6
no vac cha pigs	6
the emergence of new	6
be due to the	6
the uptake of fdx	6
transport of influenza a	6
shown in figure a	6
a subset of the	6
infected a cells at	6
with the help of	6
samples were collected from	6
it is known that	6
in the human population	6
the h n dk	6
of respiratory viral infections	5
of reactive oxygen species	5
with the exception of	5
defense against influenza a	5
an increased risk of	5
the mitochondrial inner membrane	5
at the indicated time	5
of the haemagglutinin membrane	5
the phosphorylation of stat	5
n and human h	5
h and h influenza	5
target for antiviral therapy	5
cells in the airways	5
to be caused by	5
and hepatitis c virus	5
human and avian influenza	5
followed by pr virus	5
was used as the	5
apoptosis in influenza virus	5
genetic diversity of swine	5
pkr and eif a	5
replication of the iav	5
h n virus which	5
immune responses to influenza	5
consistent with previous reports	5
iav strains in the	5
of the m protein	5
linkages abundantly present on	5
not included in the	5
determined by western blotting	5
id at weeks and	5
in a range of	5
c motif chemokine ligand	5
the cytoplasmic tail of	5
respiratory tract infection in	5
use of laboratory animals	5
acute lower respiratory tract	5
amino acid change in	5
the emergence of novel	5
the activation of pi	5
influenza a virus h	5
airway epithelial cells and	5
of the iav infection	5
of pulmonary surfactant protein	5
the results of a	5
respiratory syndrome coronavirus spike	5
the type i ifn	5
type a influenza virus	5
on the susceptibility of	5
cells infected with pr	5
in swine sera samples	5
infection in a cells	5
inhibits influenza a virus	5
viruses from pigs in	5
of a cells with	5
the potential use of	5
is essential for the	5
the haemagglutinin membrane glycoprotein	5
similarly as described previously	5
an alternative entry pathway	5
targeting ha of iavs	5
human influenza a virus	5
distinct lineage of influenza	5
intracellular m ion channel	5
a cells transfected with	5
to protect mice from	5
used to assess statistical	5
shown in figure b	5
are responsible for the	5
recent studies have shown	5
the pb e k	5
nuclear export of viral	5
curcumin derivatives and neuraminidase	5
in a model of	5
the effective replication of	5
avian influenza virus h	5
with fetal bovine serum	5
by neutrophils and macrophages	5
is considered to be	5
study was supported by	5
either rsv or iav	5
in the promoter region	5
and acute lung injury	5
activates inflammasomes via its	5
influenza a puerto rico	5
infected as well as	5
avian h n iavs	5
is associated with high	5
of influenza h n	5
influenza virus at a	5
ubiquitin chain assembly complex	5
in the guinea pig	5
mouse model of influenza	5
swine influenza virus from	5
influenza a virus surveillance	5
viral load and hypercytokinemia	5
at a density of	5
antiviral activity of dips	5
associated with severe iav	5
type i interferons in	5
type i interferon induction	5
been demonstrated to be	5
like h n virus	5
in the extranuclear space	5
with h n and	5
the mechanism of ifitm	5
into the host cell	5
by the lack of	5
in the formation of	5
after treatment with pa	5
associated with susceptibility to	5
influenza a virus influenza	5
compared to a cells	5
to acute respiratory distress	5
transmission of iav was	5
or in pbs containing	5
the replication of multiple	5
time pcr and normalised	5
different time points after	5
t cells were transfected	5
and avian influenza viruses	5
severe influenza in humans	5
h n pdm infection	5
virus infections in swine	5
of the adaptive immune	5
at h and h	5
over the course of	5
report of h n	5
of iav and ibv	5
with risk of severe	5
pathogenesis of avian influenza	5
of the epidemiology of	5
the primary site of	5
n influenza a epidemic	5
viral infection in mice	5
associated with high viral	5
respiratory syndrome coronavirus envelope	5
were washed and incubated	5
between curcumin derivatives and	5
and evolution of influenza	5
to an increase in	5
context of viral infection	5
t cells to the	5
antiviral activity of ifitm	5
between avian and swine	5
mice infected with a	5
the hemagglutinin of influenza	5
haemagglutinin membrane glycoprotein of	5
in swine at agricultural	5
human lung epithelial cells	5
activation of dendritic cells	5
immune response to iav	5
at least one pathogen	5
influenza viruses in mice	5
wdnhbe cells infected with	5
the absence of standard	5
interaction between iav and	5
in the bronchoalveolar lavage	5
a viruses isolated from	5
or absence of the	5
resident memory t cells	5
pr virus for h	5
a virus in wild	5
pdm and h n	5
induced by influenza a	5
expression of proinflammatory cytokines	5
the bronchoalveolar lavage fluid	5
in addition to its	5
and the formation of	5
in health and disease	5
in the sars consensome	5
the cells were then	5
swine influenza viruses in	5
infection influenza a virus	5
cd domain of ifitm	5
biology of influenza viruses	5
with high viral load	5
a cells were treated	5
genome sequence of a	5
virus infection in vitro	5
origin h n viruses	5
viruses were reported in	5
virus and dengue virus	5
characterization of influenza virus	5
in the last two	5
by h n infection	5
are involved in the	5
alveolar epithelial cell injury	5
as a receptor for	5
of an influenza a	5
an important determinant of	5
pigs sampled between week	5
was not observed in	5
due to the presence	5
phosphorylation of stat tyr	5
system for production of	5
virus in wild birds	5
and hrv were the	5
t cell responses in	5
as well as of	5
of natural killer cells	5
strain of h n	5
a respiratory disease outbreak	5
that memory cd t	5
the ha protein of	5
epithelial cells of the	5
effective replication of the	5
by memory cd t	5
used for the detection	5
in the lung following	5
innate immune responses to	5
evolutionary genomics of the	5
generation of influenza a	5
carried out as described	5
is the first report	5
virus m ion channel	5
infection with influenza a	5
due to the lack	5
in the viral genome	5
n virus was isolated	5
a virus from bats	5
the inhibition of iav	5
an h n virus	5
of the ketogenic diet	5
virus infection of mice	5
of innate immune response	5
to the emergence of	5
the context of the	5
as we previously described	5
between week and week	5
and function of the	5
are in agreement with	5
iav in swine in	5
the antiviral activities of	5
be one of the	5
also involved in the	5
lineage of influenza a	5
infection of human cells	5
the cells were treated	5
can be used for	5
of acute respiratory viral	5
a virus h n	5
to cd t cells	5
alveolar epithelial cells and	5
number of iav positive	5
sampled between week and	5
in mouse models of	5
for middle east respiratory	5
of programmed cell death	5
a h n pdm	5
infection with influenza virus	5
nuclear export of vrnp	5
is also known to	5
protein induces apoptosis in	5
of seasonal influenza a	5
a reassortant h n	5
to the influenza virus	5
the case of iav	5
a past exposure of	5
high levels of gm	5
in the mbl gene	5
vac no cha pigs	5
absence of standard virus	5
influenza a viruses isolated	5
akt and erk mapk	5
infection of pigs with	5
between bat and conventional	5
inflammasomes via its intracellular	5
as a positive control	5
between cov infection and	5
and na on the	5
the development of live	5
a number of studies	5
amino acid sequences of	5
and a decrease in	5
the response to cov	5
to a lesser extent	5
in embryonated chicken eggs	5
influenza virus infection by	5
and swine influenza viruses	5
influenza a virus transmission	5
it is not clear	5
all authors read and	5
were negative for iav	5
following respiratory virus infections	5
antigenic structure of the	5
single nucleotide polymorphisms in	5
ha of the h	5
influenza viruses in the	5
the ability of the	5
the influenza virus nucleoprotein	5
of iav in the	5
pandemic h n and	5
for the induction of	5
the creative commons attribution	5
ifitm promoter is associated	5
adnrf followed by pr	5
in this study we	5
virus infection results in	5
activation of pi k	5
human nasal epithelial cells	5
the supernatants of a	5
the influenza virus genome	5
its intracellular m ion	5
in control and ifitm	5
in pbs containing fcs	5
and use of laboratory	5
iav infection has been	5
in the control group	5
origins and evolutionary genomics	5
to test this hypothesis	5
supernatants of a cells	5
in respiratory virus infections	5
cationic host defence peptides	5
surfactant protein a binds	5
was reported from a	5
is essential for efficient	5
influence of curcumin derivatives	5
t cells play a	5
binding of surfactant protein	5
fatal outcome of human	5
the protective effects of	5
cell pr virus for	5
pandemic influenza a viruses	5
to induce apoptosis in	5
a swine farm in	5
human host factors required	5
the promoter region of	5
influenza virus replication by	5
role of ap m	5
a significant increase in	5
outcome of human influenza	5
increase the expression of	5
a cells at different	5
iav replication in vitro	5
with a pr at	5
n virus antibodies were	5
in the blood of	5
mice were infected with	5
k akt and erk	5
are more likely to	5
was observed in the	5
the absence of iav	5
cells such as macrophages	5
presence of fcs and	5
is not required for	5
in bronchoalveolar lavage fluid	5
by any of the	5
pi k akt and	5
for influenza a viruses	5
in the lung tissues	5
an essential role in	5
characterization of the human	5
and vesicular stomatitis virus	5
patients with h n	5
lung epithelial a cells	5
avian influenza virus in	5
care and use of	5
infection was shown to	5
the ic activity of	5
the lectin complement pathway	5
as a ratio to	5
of iav positive swine	5
was found in the	5
pneumonia symptoms in iav	5
characterization of influenza a	5
of the virus and	5
in the human lung	5
an innate immune response	5
was isolated from the	5
with avian and human	5
host factors required for	5
herpes simplex virus type	5
and evolutionary genomics of	5
the size of the	5
genetic characterization of h	5
involved in iav replication	5
ns protein induces apoptosis	5
at the ifitm promoter	5
a pivotal role in	5
high viral load and	5
trail and sfas ligand	5
between human and swine	5
it is worth noting	5
nlrp inflammasome activation in	5
t cells are critical	5
it was shown that	5
in vivo and in	5
syndrome coronavirus envelope protein	5
two tailed paired students	5
of the viral infection	5
virus infection dynamics in	5
as an alternative entry	5
is closely related to	5
also been associated with	5
shown to decrease the	5
virus activates inflammasomes via	5
protein ion channel activity	5
to assess statistical significance	5
could be used to	5
the inhibition of rig	5
is worth noting that	5
the ifitm promoter is	5
via its intracellular m	5
in cell culture and	5
is required for efficient	5
also been reported to	5
the potential to be	5
pig id at weeks	5
during the course of	5
the expression of ifitm	5
a and d in	5
are one of the	5
macrophages and dendritic cells	5
the viral life cycle	5
of growth factor receptor	5
a novel interaction between	5
cells were pretreated with	5
been shown to induce	5
might be due to	5
moi of pfu cell	5
g g cell cycle	5
performed as previously described	5
on the pathogenesis of	5
approved the final manuscript	5
restrict the replication of	5
pigs from no vac	5
against h n influenza	5
a key regulator of	5
a negative regulator of	5
susceptible to infection with	5
cd t cells can	5
tailed paired students t	5
any of the h	5
in response to sars	5
inducing the expression of	5
roles in the response	5
the severe acute respiratory	5
factors required for influenza	5
in the influenza virus	5
are recruited to the	5
for h prior to	5
through the inhibition of	5
for the emergence of	5
as a model for	5
the pi k akt	5
at weeks and were	5
this study was supported	5
in host defense against	5
the hemagglutinin and neuraminidase	5
production of defective interfering	5
and erk mapk signaling	5
airway epithelium in vitro	5
influenza virus a pr	5
distributed under the terms	5
induction of proinflammatory cytokines	5
under the age of	5
primary human airway epithelial	5
the first months of	5
pcr and normalised to	5
infected with pandemic iav	5
of the nrf pathway	5
involved in the anti	5
virus isolates were reported	5
against influenza a viruses	5
for each of the	5
viruses isolated from pigs	5
such as iav and	5
than years of age	5
that the presence of	5
to increase the expression	5
associated with risk of	5
vaccines for use in	5
the biology of influenza	5
our data showed that	5
protection against h n	5
including inhibition of the	5
of influenza virus and	5
from pigs sampled between	5
in dmem supplemented with	5
were washed twice with	5
the inhibition of the	5
has been suggested to	5
was reported in swine	5
are included in the	5
of the creative commons	5
in response to h	5
cells were grown in	5
of h n swine	5
activity relationship and antiviral	5
with severe influenza in	5
from march to may	5
avian influenza virus a	5
cells at different time	5
by cd t cells	5
was carried out as	5
viral and endosomal membranes	5
in swine in china	5
influenza a virus infected	5
the viral replication cycle	5
in the production of	5
in iav infected mice	5
based swine farm in	5
the inhibitory effects of	5
of human influenza virus	5
in order to determine	5
have been reported from	5
to inhibit replication of	5
glycoprotein of influenza c	5
a novel influenza a	5
h n pdm on	5
of influenza virus ns	5
recent study showed that	5
influenza viruses circulating in	5
have also been shown	5
b on iav infection	5
antibodies against h n	5
the ifitm expression was	5
of the virus with	5
the effect of iav	5
with pr virus at	5
data are presented as	5
a gift from dr	5
virus at a resolution	5
was performed as previously	5
organization of ha and	5
type i ifn response	5
ha of h n	5
on the plasma membrane	5
strain of influenza a	5
a distinct lineage of	5
of na on the	5
pulmonary surfactant protein d	5
immunity against iav infection	5
of the host cell	5
genomics of the swine	5
erk mapk signaling pathways	5
the cd domain of	5
antigenic drift and shift	5
viruses using reverse genetics	5
isolates were retrieved from	5
results suggest that the	5
in presence of oc	5
of proinflammatory cytokines in	5
avian and swine influenza	5
due to the low	5
induced by highly pathogenic	5
influenza virus activates inflammasomes	5
and h n pdm	5
cells were then infected	5
in cells treated with	5
have not been identified	5
as an opsonin for	4
a is also able	4
mortality from iav infection	4
to be closely related	4
ifitm proteins restrict viral	4
in the viral membrane	4
type ii cells and	4
supplemented with fetal bovine	4
h n virus were	4
avian and swine iav	4
impact of cov infection	4
cells lacking a strong	4
to middle east respiratory	4
of ifitm is required	4
a virus infections in	4
involved in activation of	4
reduced lung viral titers	4
immune response and the	4
confluent mdck cells were	4
from pigs sampled at	4
to be critical in	4
of the use of	4
sections were incubated with	4
reassortant h n swine	4
enhanced type i ifn	4
in a human lung	4
the concerted action of	4
the uninfected a cells	4
number of infected cells	4
for iav h n	4
under the terms and	4
ifitm clustering at vesicular	4
role of cd t	4
ifn trail signaling axis	4
factors associated with influenza	4
gene segments from the	4
role in innate immunity	4
a specific inhibitor of	4
severity and prognosis of	4
pdm virus was reported	4
it is conceivable that	4
influenza a penetrates host	4
also contribute to the	4
the indicated concentration of	4
update on the susceptibility	4
the location of the	4
of viral m protein	4
a virus replication by	4
small airway epithelial cells	4
intranasal administration of pa	4
a more recent study	4
in the levels of	4
the avian influenza virus	4
followed by cycles of	4
in severe influenza virus	4
in a manner that	4
were positive for the	4
from a swine in	4
epithelial to mesenchymal transition	4
pulmonary hypersensitivity induced by	4
they can also be	4
the absence or presence	4
first report of h	4
research has shown that	4
to infection with iav	4
pigs in the laiv	4
infected with respiratory syncytial	4
a crucial role in	4
swine was documented during	4
group infected with iav	4
syncytial virus infection in	4
binding at the ifitm	4
the binding of influenza	4
of novel treatment strategies	4
animal and public health	4
in hospitalized children with	4
in mice critical role	4
influenza a virus inhibits	4
the basis of the	4
sow and nursery sites	4
and critical care medicine	4
in response to infection	4
of high levels of	4
in the mitochondrial matrix	4
the emergence of the	4
a swine h n	4
isolates were reported in	4
of hepatitis b virus	4
inhibits the activation of	4
were upregulated by pr	4
plays a key role	4
role of the nlrp	4
avian and human type	4
to bind to the	4
and conditions of the	4
and genome uncoating are	4
procedures were approved by	4
medium was replaced with	4
of double or triple	4
been associated with the	4
of the human genome	4
infection of influenza a	4
to h n virus	4
in this study was	4
of acute lower respiratory	4
opsonin for influenza a	4
the level of the	4
the studies conducted in	4
terms and conditions of	4
np and hsp in	4
had the highest sequence	4
after a respiratory disease	4
of p ipk from	4
the results of the	4
for the effective replication	4
in this article are	4
in the ifitm signal	4
of the viral envelope	4
vaccines need to be	4
influenza a virus endocytic	4
were found seropositive for	4
treatment of influenza viral	4
were shown to bind	4
and swine influenza a	4
immune responses such as	4
for use in the	4
initial virus binding rate	4
in the nuclear export	4
of influenza b viruses	4
ifitms restrict the replication	4
the group infected with	4
can be infected with	4
a virus endocytic routes	4
the ifn trail signaling	4
c is associated with	4
viruses such as iav	4
in addition to iav	4
neuraminidase of influenza virus	4
for apoptosis induction on	4
the course of iav	4
antiviral effector ifitm disrupts	4
the laiv treatment groups	4
adnrf followed by infection	4
swine influenza virus isolated	4
susceptibility of human influenza	4
p ipk from hsp	4
the case of the	4
were infected with h	4
influenza virus infection is	4
the development of influenza	4
t cell responses against	4
the immune response against	4
with the severity of	4
infection in vitro and	4
to the effects of	4
periphery of the lobes	4
on the basis of	4
iav h n and	4
in the globular head	4
rsv or iav infection	4
contribution of na to	4
in both ams and	4
consent was obtained from	4
with h n virus	4
the h n subtype	4
in both mice and	4
and epidemiological characteristics of	4
tumor necrosis factor receptor	4
virus from eight plasmids	4
infection of a cells	4
impact of influenza on	4
proteins were upregulated by	4
fusion and genome uncoating	4
acute respiratory infections in	4
on the presence of	4
to its inhibition of	4
the lack of a	4
disruption of ctcf binding	4
metabolic state of the	4
shown to induce the	4
d can also bind	4
identity to a swine	4
the avian iav strains	4
in the uninfected a	4
and sfas ligand levels	4
of cleaved sialic acid	4
of the influenza viruses	4
to the development of	4
the presence of iav	4
characterization of an h	4
the sow and nursery	4
of cov infection on	4
mice infected with respiratory	4
contributes to severe pathophysiology	4
with a novel avian	4
with type i ifn	4
be caused by the	4
in mice treated with	4
been shown to possess	4
highest number of iav	4
is strongly dependent on	4
science foundation of china	4
acid change in the	4
polymorphisms in the mbl	4
which is activated by	4
reassortant iav strains in	4
reproduction in any medium	4
respiratory tract infections in	4
the structural and functional	4
cell infection with adnrf	4
been shown to stimulate	4
express high levels of	4
it is important to	4
the pathogenesis of avian	4
before being stimulated with	4
influenza virus hemagglutinin to	4
analyzed the effect of	4
nlrp inflammasome by iav	4
generation and characterization of	4
firefly luciferase activity was	4
cells and atii cells	4
virus assembly and budding	4
of equine influenza virus	4
the impact of cov	4
acid sequences of the	4
led to a significant	4
and luciferase activity was	4
of airborne influenza virus	4
the level of ifitm	4
in the viral replication	4
the hepatitis c virus	4
clustering at vesicular structures	4
in an attempt to	4
the adaptive immune system	4
national natural science foundation	4
had no role in	4
the pigs in the	4
of patients with severe	4
which is essential for	4
in the ifitm expression	4
to recognise and bind	4
h influenza virus isolates	4
a binds to the	4
of the a cells	4
bat influenza a viruses	4
against injury induced by	4
stimulated by viroporin ion	4
cd memory t cells	4
time point of examination	4
assay for rapid screening	4
canine kidney cell line	4
weight loss and mortality	4
risk allele associated with	4
influenza virus hemagglutinin and	4
viruses with pandemic potential	4
nuclear export of the	4
five active curcumin derivatives	4
similar results were obtained	4
mouse model of infection	4
or in the presence	4
acids in cell culture	4
a recent study showed	4
at the concentrations of	4
in the spp knowledgebase	4
its ability to inhibit	4
of influenza virus by	4
pigs in laiv none	4
to block viral entry	4
with curcumin derivatives at	4
for at least one	4
influenza h n viruses	4
human infection with a	4
understanding of the epidemiology	4
the hexosamine biosynthesis pathway	4
the cells were transfected	4
and antigenic diversity of	4
cell responses against influenza	4
a risk factor for	4
in one out of	4
influenza virus activity in	4
american journal of respiratory	4
alveolar type ii epithelial	4
infection with a novel	4
like swine h n	4
autophagosome fusion with lysosomes	4
be used as a	4
inhibits influenza virus replication	4
killed approximately two million	4
a virus m ion	4
in alveolar epithelial cells	4
from lethal h n	4
the expression level of	4
plays a role in	4
cd t cells provide	4
the antiviral effector ifitm	4
protein of iav is	4
influenza a virus neuraminidase	4
cellular networks involved in	4
the highly pathogenic h	4
the m segment of	4
porcine respiratory disease complex	4
protein a binds to	4
epithelial cells infected with	4
evidence of avian influenza	4
a virus blocks autophagosome	4
by grants from the	4
uncoating are required for	4
need to be reformulated	4
infection has also been	4
in the defense against	4
way anova followed by	4
supernatants were collected and	4
are known to be	4
responses against influenza virus	4
no increase in the	4
the first time that	4
influenza a virus by	4
the transcriptional response to	4
involved in the activation	4
course of iav infection	4
in the generation of	4
an interacting partner of	4
obtained using the geneoverlap	4
m ion channel activity	4
replication of middle east	4
in this context are	4
upper respiratory tracts of	4
can bind to the	4
is assumed for z	4
of the ha and	4
demonstrated that overexpression of	4
primary antibody and alexa	4
associated with an increased	4
the collagen region of	4
of the presence of	4
interferon alphabeta in acute	4
pigs in the wiv	4
collected from children years	4
infection prevention and control	4
may be used to	4
of the lung and	4
dependent on the lipid	4
and alveolar epithelial cells	4
was not able to	4
cells during infection with	4
nucleoprotein of influenza a	4
epithelial cells and macrophages	4
restrict viral membrane hemifusion	4
cleavage of sialic acid	4
in pregnant women is	4
and european h n	4
alphabeta in acute influenza	4
such as the h	4
proteins restrict viral membrane	4
infect a wide range	4
the vrna packaging signals	4
h and h subtypes	4
effects of type i	4
ha and na are	4
the release of inflammatory	4
viruses circulating in humans	4
is supported by a	4
expressed as a ratio	4
iav strains of h	4
lung epithelial apoptosis in	4
all the ha sequences	4
first report of a	4
in immunity to iav	4
proinflammatory cytokine production in	4
a high seroprevalence of	4
nrf sirna followed by	4
influenza a virus nuclear	4
hemagglutinin of influenza a	4
fig a and fig	4
detection of influenza viruses	4
treatment of iav infections	4
was used to determine	4
in swine populations of	4
the expression of np	4
spatial organization of ha	4
statistically significant risk factor	4
facilitate the emergence of	4
linked inhibitor of apoptosis	4
high levels of cd	4
and h n are	4
release of viral particles	4
with disease severity in	4
may be due to	4
similar trends to apical	4
a a a a	4
entry of influenza a	4
alexa conjugated secondary antibody	4
host factors involved in	4
influenza viruses target different	4
from those in the	4
evidence for a role	4
of viral infections in	4
found that pr virus	4
the terms and conditions	4
up to h p	4
may lead to a	4
effect on the expression	4
associated death domain protein	4
and p mapk signaling	4
as an interacting partner	4
known to interact with	4
in the iav group	4
outbreak of swine influenza	4
influenza viruses during viral	4
with pr virus decreased	4
the nrf pathway in	4
mice were randomly divided	4
influenza research and surveillance	4
the potential of a	4
the airway epithelium was	4
in the human airway	4
of invasive pulmonary aspergillosis	4
of seasonal influenza vaccines	4
that the h n	4
it is estimated that	4
macropinocytosis as an alternative	4
respiratory syncytial virus subtype	4
over a h period	4
d can bind to	4
influenza type b virus	4
forms of cell death	4
was determined by tcid	4
increases susceptibility to secondary	4
institutional animal care and	4
determine the effects of	4
proportion of the population	4
in the central seven	4
influenza virus due to	4
a high mutation rate	4
the h n strain	4
as a target for	4
were collected from children	4
sirna followed by infection	4
examined the effect of	4
as a function of	4
the pandemic influenza virus	4
ctcf binding at the	4
the neuraminidase of influenza	4
host mucus by cleaving	4
rna polymerase i promoter	4
of ctcf binding at	4
the master regulator of	4
detrimental to the host	4
of influenza virus rna	4
a weak ifitm signal	4
pigs in southern china	4
sequence identity to a	4
better understanding of the	4
the globular head of	4
along the length of	4
sialic acids with neuraminidase	4
virus in swine at	4
ifitm disrupts intracellular cholesterol	4
of the pandemic influenza	4
were randomly divided into	4
diet was shown to	4
blocks autophagosome fusion with	4
an h n influenza	4
the severity of pandemic	4
reported in chinese swine	4
it is hypothesized that	4
in influenza virus pneumonia	4
avian influenza virus infection	4
was isolated from a	4
at uc for min	4
a highly pathogenic h	4
the detection of iav	4
the cells were harvested	4
inflammasome by iav virulence	4
influenza a virus date	4
virus isolates were retrieved	4
as described in the	4
ketogenic diet activates protective	4
the determination of the	4
penetrates host mucus by	4
t cell responses to	4
may be responsible for	4
ifitm is required for	4
activation in response to	4
a virus genomic rna	4
detection and isolation of	4
journal of respiratory and	4
of nlrp inflammasome activation	4
influenza viruses to neuraminidase	4
between avian and human	4
is released from the	4
in these primary cells	4
influenza vaccine virus selection	4
the interaction between respiratory	4
to influenza a infection	4
like cells and atii	4
is a determinant of	4
of the host immune	4
of iav in pigs	4
with h n iav	4
the innate and adaptive	4
apparently healthy swine in	4
of the antiviral protein	4
studies have also shown	4
younger than years of	4
these studies suggest that	4
mediated inhibition of iav	4
or h n subtypes	4
strongly dependent on the	4
that this effect is	4
the amplitude of the	4
using the geneoverlap analysis	4
in upper respiratory tract	4
the antiviral properties of	4
of cd t lymphocytes	4
at one of the	4
of curcumin derivatives on	4
at h post transfection	4
in different cell types	4
in each of the	4
the seasonal influenza virus	4
at least two different	4
of the ns protein	4
to the site of	4
of influenza and other	4
the distribution of the	4
host immune response is	4
is proportional to the	4
involved in the influenza	4
dimensional structure of the	4
limited understanding of the	4
which appeared to have	4
have been demonstrated to	4
protection against influenza virus	4
pathogenesis of influenza a	4
emergence of influenza a	4
in type i interferon	4
h n virus having	4
a major role in	4
time points after infection	4
the capacity of the	4
with iav before being	4
which is known to	4
fcs and mm dynasore	4
global update on the	4
a decrease in the	4
influenza virus life cycle	4
in immune response to	4
compared with the control	4
decrease the expression of	4
avian and human influenza	4
pa may be able	4
with pandemic influenza a	4
conditions of the creative	4
animal and human health	4
a and b virus	4
a cells were pretreated	4
and genetic characterization of	4
sialic acid on the	4
highest sequence identity to	4
macrophages and neutrophils in	4
detected in swine samples	4
and porcine reproductive and	4
h n and avian	4
determined by tcid assay	4
n and pandemic h	4
cells infected with influenza	4
cd t cell subsets	4
the cleavage of sialic	4
related to its inhibition	4
respiratory and critical care	4
in pigs in the	4
in influenza virus infected	4
ha and na in	4
viruses the role of	4
rna viruses such as	4
of the hemagglutinin and	4
influenza virus avian influenza	4
have shown that the	4
influenza viruses from swine	4
iav replication through the	4
inhibits the function of	4
a vital role in	4
in the first round	4
a virus surveillance in	4
type i ifn induction	4
n influenza virus by	4
were shown to be	4
identified in this study	4
in cells transfected with	4
mediated disruption of ctcf	4
avian and human iav	4
article is an open	4
sampled at week and	4
did not result in	4
research is needed to	4
positive for at least	4
lethal influenza virus challenge	4
pig id sampled at	4
shown to be a	4
virus endocytic routes reveals	4
pigs sampled at week	4
surfactant protein d and	4
and pandemic influenza a	4
african green monkey kidney	4
the replication cycle of	4
was found to have	4
the activity of na	4
and four h n	4
four h n viruses	4
viruses to neuraminidase inhibitors	4
respiratory disease outbreak in	4
both the upper and	4
for the pathogenesis of	4
is an open access	4
specific intercellular adhesion molecule	4
homeostasis to block viral	4
and na subtypes of	4
of inflammatory cytokines and	4
the increase in the	4
cells with a strong	4
n virus was detected	4
in the influenza a	4
and influenza susceptibility and	4
virus isolated from a	4
there is an urgent	4
the no vac no	4
identification of a novel	4
the source of the	4
absence or presence of	4
acts as a receptor	4
and to a lesser	4
cells in the upper	4
with pandemic h n	4
be related to its	4
of avian and mammalian	4
the antigenic structure of	4
percentage of necrotic cells	4
to iav h n	4
system for generation of	4
glycosylation of the hemagglutinin	4
the influenza virus disease	4
m ion channel blockers	4
respiratory syndrome coronavirus and	4
is associated with risk	4
and human iav binding	4
in tissue culture cells	4
results are expressed as	4
lectin pathway of complement	4
in swine at exhibitions	4
intracellular cholesterol homeostasis to	4
studies are needed to	4
in the human respiratory	4
in mediating viral clearance	4
h n pdm subtypes	4
is required for the	4
found infected with h	4
these data demonstrate that	4
pr mtsin and wsn	4
potential of interferon alphabeta	4
human upper respiratory tract	4
a penetrates host mucus	4
the role of macrophage	4
influenza a virus with	4
virus from pigs in	4
disrupts intracellular cholesterol homeostasis	4
increased ifitm signal intensity	4
ifitm restriction of viral	4
by the influenza a	4
hct intersection networks in	4
cellular signaling pathway nodes	4
the metabolic state of	4
and activation of the	4
for min followed by	4
involved in regulation of	4
of mbl in the	4
ifitm signal intensity in	4
cells transfected with vrna	4
samples were negative for	4
used for the treatment	4
actin filaments of the	4
dissection of the influenza	4
analysis package in r	4
genes in wdnhbe cells	4
localization of ifitm and	4
our results showed that	4
iav has been shown	4
the release of iav	4
the impact of the	4
the activity of the	4
in mice challenged with	4
syncytial virus and influenza	4
the care and use	4
on apparently vesicular structures	4
f contributes to severe	4
the sialic acid binding	4
of the h hemagglutinin	4
been found to be	4
of the cytokine storm	4
we will discuss the	4
infected with iav at	4
results showed that pa	4
isolated from a swine	4
gene of influenza a	4
viral membrane fusion and	4
into the mechanisms of	4
lacking a strong nuclear	4
system development and function	4
there was no difference	4
approaches for the development	4
the cytokine storm in	4
is a type ii	4
and type iii interferons	4
virus and influenza a	4
fold by h n	4
upon influenza virus infection	4
the ratios of the	4
a mouse model for	4
consistent with our previous	4
cytokine and chemokine secretion	4
production of infectious dips	4
detected in swine nasal	4
influenza virus infected cells	4
the iav m protein	4
rsv and hrv were	4
the first time the	4
experiments were performed in	4
nlg site at position	4
with severe iav infection	4
of the common cold	4
known to be an	4
initial binding rate by	4
detection of influenza virus	4
protective effects of the	4
of the proinflammatory cytokines	4
iav infection has also	4
was observed during the	4
of apoptotic cells in	4
matrix protein of influenza	4
reported to be required	4
innate immune response and	4
needed to determine the	4
was determined by using	4
lethal h n infection	4
report of iav in	4
is similar to that	4
defective interfering influenza virus	4
with iav h n	4
of lung surfactant protein	4
mucus by cleaving sialic	4
replication of influenza a	4
of influenza virus neuraminidase	4
total rna was isolated	4
the vast majority of	4
prior to iav infection	4
dengue and zika viruses	4
to injury induced by	4
cases of h n	4
of interferon alphabeta in	4
the top of the	4
with an h n	4
to a number of	4
virus infection by preventing	4
is likely to be	4
activation of the interferon	4
pdm viruses were reported	4
protective role of nrf	4
according to previously described	4
we analyzed the effect	4
genome uncoating are required	4
routes reveals macropinocytosis as	4
viruses during viral entry	4
associated with the first	4
mice infected with iav	4
the inoculum was removed	4
of the epidemiology and	4
around individual influenza viruses	4
a type i ifn	4
cov infection on human	4
sitosterol on the ifn	4
of vdid dequenching in	4
the origin of the	4
cov e protein is	4
and acute respiratory distress	4